Pattern of expression of Toll like receptor (TLR)-3 and -4 genes in drug-naïve and antipsychotic treated patients diagnosed with schizophrenia.


Journal

Psychiatry research
ISSN: 1872-7123
Titre abrégé: Psychiatry Res
Pays: Ireland
ID NLM: 7911385

Informations de publication

Date de publication:
03 2020
Historique:
received: 18 09 2019
revised: 25 11 2019
accepted: 02 12 2019
pubmed: 16 12 2019
medline: 30 9 2020
entrez: 16 12 2019
Statut: ppublish

Résumé

Toll like receptors (TLRs), a class of conserved immune molecules are crucially involved in initiating innate immune response to infection. TLR activation and subsequent inflammation are linked to pathogenesis of many brain disorders. Preliminary studies indicate a possible role of TLR-driven immuno-inflammatory responses in schizophrenia. However, gene expression data of TLRs in drug-naïve as well as antipsychotic treated patients diagnosed with schizophrenia are albeit limited. In this study, expression profile of TLR3 and TLR4 genes in peripheral blood mononuclear cells (PBMCs) was compared between drug-naïve patients diagnosed with schizophrenia (N = 31) and healthy controls (N = 30). In addition, the pattern of expression of TLR3 and TLR4 genes were also examined after three months of antipsychotic medication in patients. Compared to healthy controls, gene expression levels of only TLR4 (F = 3.87, p = 0.05, η

Identifiants

pubmed: 31837816
pii: S0165-1781(19)31957-2
doi: 10.1016/j.psychres.2019.112727
pii:
doi:

Substances chimiques

Antipsychotic Agents 0
TLR3 protein, human 0
TLR4 protein, human 0
Toll-Like Receptor 3 0
Toll-Like Receptor 4 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

112727

Informations de copyright

Copyright © 2019 Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare no conflict of interest.

Auteurs

Renu Balaji (R)

Department of Human Genetics, National Institute of Mental Health and Neurosciences, Hosur Road, Bangalore 560029, Karnataka, India.

Manjula Subbanna (M)

Department of Human Genetics, National Institute of Mental Health and Neurosciences, Hosur Road, Bangalore 560029, Karnataka, India; Translational Psychiatry Laboratory, Neurobiology Research Centre, National Institute of Mental Health and Neurosciences, Bangalore, Karnataka, India.

Venkataram Shivakumar (V)

Translational Psychiatry Laboratory, Neurobiology Research Centre, National Institute of Mental Health and Neurosciences, Bangalore, Karnataka, India; Department of Psychiatry, National Institute of Mental Health and Neurosciences, Bangalore, Karnataka, India.

Fazal Abdul (F)

Department of Human Genetics, National Institute of Mental Health and Neurosciences, Hosur Road, Bangalore 560029, Karnataka, India.

Ganesan Venkatasubramanian (G)

Translational Psychiatry Laboratory, Neurobiology Research Centre, National Institute of Mental Health and Neurosciences, Bangalore, Karnataka, India; Department of Psychiatry, National Institute of Mental Health and Neurosciences, Bangalore, Karnataka, India.

Monojit Debnath (M)

Department of Human Genetics, National Institute of Mental Health and Neurosciences, Hosur Road, Bangalore 560029, Karnataka, India. Electronic address: monojit-d@nimhans.ac.in.

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Classifications MeSH