Mean muscle attenuation correlates with severe acute pancreatitis unlike visceral adipose tissue and subcutaneous adipose tissue.


Journal

United European gastroenterology journal
ISSN: 2050-6414
Titre abrégé: United European Gastroenterol J
Pays: England
ID NLM: 101606807

Informations de publication

Date de publication:
12 2019
Historique:
received: 01 05 2019
accepted: 18 09 2019
entrez: 17 12 2019
pubmed: 17 12 2019
medline: 1 7 2020
Statut: ppublish

Résumé

Acute pancreatitis (AP) is a frequent disorder with considerable morbidity and mortality. Obesity has previously been reported to influence disease severity. The aim of this study was to investigate the association of adipose and muscle parameters with the severity grade of AP. In total 454 patients were recruited. The first contrast-enhanced computed tomography of each patient was reviewed for adipose and muscle tissue parameters at L3 level. Associations with disease severity were analysed through logistic regression analysis. The predictive capacity of the parameters was investigated using receiver operating characteristic (ROC) curves. No distinct variation was found between the AP severity groups in either adipose tissue parameters (visceral adipose tissue and subcutaneous adipose tissue) or visceral muscle ratio. However, muscle mass and mean muscle attenuation differed significantly with Our results demonstrate that a low muscle attenuation level is associated with an increased risk of severe AP. Future prospective studies will help identify the underlying mechanisms and characterise the influence of body composition parameters on AP.

Sections du résumé

Background
Acute pancreatitis (AP) is a frequent disorder with considerable morbidity and mortality. Obesity has previously been reported to influence disease severity.
Objective
The aim of this study was to investigate the association of adipose and muscle parameters with the severity grade of AP.
Methods
In total 454 patients were recruited. The first contrast-enhanced computed tomography of each patient was reviewed for adipose and muscle tissue parameters at L3 level. Associations with disease severity were analysed through logistic regression analysis. The predictive capacity of the parameters was investigated using receiver operating characteristic (ROC) curves.
Results
No distinct variation was found between the AP severity groups in either adipose tissue parameters (visceral adipose tissue and subcutaneous adipose tissue) or visceral muscle ratio. However, muscle mass and mean muscle attenuation differed significantly with
Conclusions
Our results demonstrate that a low muscle attenuation level is associated with an increased risk of severe AP. Future prospective studies will help identify the underlying mechanisms and characterise the influence of body composition parameters on AP.

Identifiants

pubmed: 31839956
doi: 10.1177/2050640619882520
pii: 10.1177_2050640619882520
pmc: PMC6893994
doi:

Types de publication

Journal Article Multicenter Study Research Support, N.I.H., Extramural Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

1312-1320

Subventions

Organisme : NIDDK NIH HHS
ID : R01 DK092460
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

© Author(s) 2019.

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Auteurs

Hanna Sternby (H)

Department of Surgery, Institution of Clinical Sciences Malmö, Lund University, Malmö, Sweden.

Mariella Mahle (M)

Department of Internal Medicine I, Martin Luther University, Halle (Saale), Germany.

Nicolas Linder (N)

Department of Diagnostic and Interventional Radiology, University Hospital Leipzig, Leipzig, Germany.
IFB Adiposity Diseases, University of Leipzig, Leipzig, Germany.

Laureen Erichson-Kirst (L)

Department of Internal Medicine, Neurology and Dermatology, Division of Gastroenterology, Leipzig, Germany.

Robert C Verdonk (RC)

Department of Gastroenterology, St. Antonius Ziekenhuis, Nieuwegein, the Netherlands.

Alexandra Dimova (A)

Department of Surgery, University Hospital for Emergency Medicine "Pirogov", Sofia, Bulgaria.

Povilas Ignatavicius (P)

Department of Surgery, Lithuanian University of Health Sciences, Kaunas, Lithuania.

Lucas Ilzarbe (L)

Department of Gastroenterology, Hospital del Mar, Barcelona, Spain.

Peeter Koiva (P)

Department of Gastroenterology, East Tallinn Central Hospital, Tallinn, Estonia.

Anne Penttilä (A)

Department of Surgery, Helsinki University Central Hospital, Helsinki, Finland.

Sara Regnér (S)

Department of Surgery, Institution of Clinical Sciences Malmö, Lund University, Malmö, Sweden.

Thomas L Bollen (TL)

Department of Radiology, St. Antonius Ziekenhuis, Nieuwegein, the Netherlands.

Richard Brill (R)

Department of Radiology, Martin Luther University Halle-Wittenberg, Halle (Saale), Germany.

Franz Stangl (F)

Department of Radiology, Martin Luther University Halle-Wittenberg, Halle (Saale), Germany.

Walter A Wohlgemuth (WA)

Department of Radiology, Martin Luther University Halle-Wittenberg, Halle (Saale), Germany.

Vijay Singh (V)

Department of Medicine, Mayo Clinic, Scottsdale, Arizona, USA.

Harald Busse (H)

Department of Diagnostic and Interventional Radiology, University Hospital Leipzig, Leipzig, Germany.
IFB Adiposity Diseases, University of Leipzig, Leipzig, Germany.

Patrick Michl (P)

Department of Internal Medicine I, Martin Luther University, Halle (Saale), Germany.

Sebastian Beer (S)

Department of Internal Medicine, Neurology and Dermatology, Division of Gastroenterology, Leipzig, Germany.

Jonas Rosendahl (J)

Department of Internal Medicine I, Martin Luther University, Halle (Saale), Germany.

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