NOX2-Dependent Reactive Oxygen Species Regulate Formyl-Peptide Receptor 1-Mediated TrkA Transactivation in SH-SY5Y Cells.
Journal
Oxidative medicine and cellular longevity
ISSN: 1942-0994
Titre abrégé: Oxid Med Cell Longev
Pays: United States
ID NLM: 101479826
Informations de publication
Date de publication:
2019
2019
Historique:
received:
11
07
2019
revised:
08
10
2019
accepted:
18
10
2019
entrez:
25
12
2019
pubmed:
25
12
2019
medline:
6
5
2020
Statut:
epublish
Résumé
Several enzymes are capable of producing reactive oxygen species (ROS), but only NADPH oxidases (NOX) generate ROS as their primary and sole function. In the central nervous system, NOX2 is the major source of ROS, which play important roles in signalling and functions. NOX2 activation requires p47
Identifiants
pubmed: 31871542
doi: 10.1155/2019/2051235
pmc: PMC6913242
doi:
Substances chimiques
NTRK1 protein, human
0
Reactive Oxygen Species
0
Receptors, Formyl Peptide
0
NADPH Oxidase 2
EC 1.6.3.-
Receptor, trkA
EC 2.7.10.1
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
2051235Informations de copyright
Copyright © 2019 Martina Castaldo et al.
Déclaration de conflit d'intérêts
The authors declare that there is no conflict of interest regarding the publication of this paper.
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