Parietal conditioning enhances motor surround inhibition.


Journal

Brain stimulation
ISSN: 1876-4754
Titre abrégé: Brain Stimul
Pays: United States
ID NLM: 101465726

Informations de publication

Date de publication:
Historique:
received: 16 09 2019
revised: 11 12 2019
accepted: 12 12 2019
pubmed: 28 12 2019
medline: 25 8 2020
entrez: 28 12 2019
Statut: ppublish

Résumé

Motor surround inhibition (mSI) is a phenomenon supportive for executing selective finger movements, wherein synergist muscles are selectively facilitated while surround muscles are inhibited. Previous studies of conditioning inputs to several intracortical and cortico-cortical inhibitory networks did not show an influence on mSI. The inhibitory posterior parietal-motor network, which is crucial for executing fine movements, however, has not been studied. To investigate the role of inhibitory posterior parietal-motor network in mSI. We hypothesized that conditioning this inhibitory network would enhance mSI. 11 healthy adults completed study. mSI was elicited by applying a TMS pulse over the motor cortex coupled with or without a conditioning input to an inhibitory spot in the posterior parietal cortex at 2 or 4 ms interval. Conditioning input to the posterior parietal cortex increased mSI by ∼20% CONCLUSION: The inhibitory posterior parietal-motor network appears to contribute to the genesis of mSI.

Sections du résumé

BACKGROUND
Motor surround inhibition (mSI) is a phenomenon supportive for executing selective finger movements, wherein synergist muscles are selectively facilitated while surround muscles are inhibited. Previous studies of conditioning inputs to several intracortical and cortico-cortical inhibitory networks did not show an influence on mSI. The inhibitory posterior parietal-motor network, which is crucial for executing fine movements, however, has not been studied.
OBJECTIVE/HYPOTHESIS
To investigate the role of inhibitory posterior parietal-motor network in mSI. We hypothesized that conditioning this inhibitory network would enhance mSI.
METHODS
11 healthy adults completed study. mSI was elicited by applying a TMS pulse over the motor cortex coupled with or without a conditioning input to an inhibitory spot in the posterior parietal cortex at 2 or 4 ms interval.
RESULTS
Conditioning input to the posterior parietal cortex increased mSI by ∼20% CONCLUSION: The inhibitory posterior parietal-motor network appears to contribute to the genesis of mSI.

Identifiants

pubmed: 31879086
pii: S1935-861X(19)30482-6
doi: 10.1016/j.brs.2019.12.011
pmc: PMC8214400
mid: NIHMS1708909
pii:
doi:

Types de publication

Journal Article Research Support, N.I.H., Intramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

447-449

Subventions

Organisme : Intramural NIH HHS
ID : Z01 NS002669
Pays : United States

Informations de copyright

Published by Elsevier Inc.

Déclaration de conflit d'intérêts

Declaration of competing interest None of the authors have potential conflicts of interest to be disclosed.

Références

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Auteurs

Nivethida Thirugnanasambandam (N)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA.

Giorgio Leodori (G)

IRCCS NEUROMED, Pozzilli, Italy.

Traian Popa (T)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA.

Panagiotis Kassavetis (P)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA.

Alexandra Mandel (A)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA; The George Washington University, Washington, D.C, USA.

Alexander Shaft (A)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA; University of Nevada School of Medicine, Reno, NV, USA.

Jaron Kee (J)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA; University of New Mexico School of Medicine, Albuquerque, NM, USA.

Sarung Kashyap (S)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA.

Gregg Khodorov (G)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA; The George Washington University, Washington, D.C, USA.

Mark Hallett (M)

Human Motor Control Section, National Institute of Neurological Disorders and Stroke (NINDS), NIH, Bethesda, MD, USA. Electronic address: hallettm@ninds.nih.gov.

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Classifications MeSH