Characterization of Carotid Plaque Components by Quantitative Susceptibility Mapping.


Journal

AJNR. American journal of neuroradiology
ISSN: 1936-959X
Titre abrégé: AJNR Am J Neuroradiol
Pays: United States
ID NLM: 8003708

Informations de publication

Date de publication:
02 2020
Historique:
received: 15 08 2019
accepted: 14 11 2019
pubmed: 28 12 2019
medline: 30 9 2020
entrez: 28 12 2019
Statut: ppublish

Résumé

Intraplaque hemorrhage in the carotid artery is related to an increased risk of cerebrovascular ischemic events. We aimed to investigate whether quantitative susceptibility mapping can characterize carotid artery plaque components and quantify the severity of intraplaque hemorrhage. For this ex vivo quantitative susceptibility mapping study, 9 carotid endarterectomy specimens were imaged on a 3T MR imaging scanner using a 3D multi-echo gradient-echo sequence and a microscopy coil. The samples were examined histologically using immunostains, including glycophorin A and Prussian blue. The areas of erythrocytes, iron deposits, calcification, and fibrous matrices observed on stained sections were compared with quantitative susceptibility mapping findings and their mean susceptibility values. Intraplaque hemorrhage and iron deposits were observed only in areas hyperintense on quantitative susceptibility mapping; calcifications and fibrous matrices were prevalent in hypointense areas. The mean susceptibility values for necrotic cores with intraplaque hemorrhage but no iron deposits, cores with iron deposits but no intraplaque hemorrhage, cores without either intraplaque hemorrhage or iron deposits, and cores with calcification were 188 ± 51, 129 ± 49, -11 ± 17, and -158 ± 78 parts per billion, respectively. There was a significant difference in the mean susceptibility values among the 4 histologic components ( Our findings suggest that quantitative susceptibility mapping can characterize the composition of carotid plaques and quantify the degree of intraplaque hemorrhage and iron deposits.

Sections du résumé

BACKGROUND AND PURPOSE
Intraplaque hemorrhage in the carotid artery is related to an increased risk of cerebrovascular ischemic events. We aimed to investigate whether quantitative susceptibility mapping can characterize carotid artery plaque components and quantify the severity of intraplaque hemorrhage.
MATERIALS AND METHODS
For this ex vivo quantitative susceptibility mapping study, 9 carotid endarterectomy specimens were imaged on a 3T MR imaging scanner using a 3D multi-echo gradient-echo sequence and a microscopy coil. The samples were examined histologically using immunostains, including glycophorin A and Prussian blue. The areas of erythrocytes, iron deposits, calcification, and fibrous matrices observed on stained sections were compared with quantitative susceptibility mapping findings and their mean susceptibility values.
RESULTS
Intraplaque hemorrhage and iron deposits were observed only in areas hyperintense on quantitative susceptibility mapping; calcifications and fibrous matrices were prevalent in hypointense areas. The mean susceptibility values for necrotic cores with intraplaque hemorrhage but no iron deposits, cores with iron deposits but no intraplaque hemorrhage, cores without either intraplaque hemorrhage or iron deposits, and cores with calcification were 188 ± 51, 129 ± 49, -11 ± 17, and -158 ± 78 parts per billion, respectively. There was a significant difference in the mean susceptibility values among the 4 histologic components (
CONCLUSIONS
Our findings suggest that quantitative susceptibility mapping can characterize the composition of carotid plaques and quantify the degree of intraplaque hemorrhage and iron deposits.

Identifiants

pubmed: 31879331
pii: ajnr.A6374
doi: 10.3174/ajnr.A6374
pmc: PMC7015197
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

310-317

Subventions

Organisme : NINDS NIH HHS
ID : R01 NS105144
Pays : United States

Informations de copyright

© 2020 by American Journal of Neuroradiology.

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Auteurs

M Azuma (M)

From the Departments of Radiology (M.A., Z.A.K., T.H.) minako_azuma@med.miyazaki-u.ac.jp.

K Yokogami (K)

Neurosurgery (K.Y., H.T., Y.A.), Faculty of Medicine, University of Miyazaki, Miyazaki, Japan.

M Enzaki (M)

Radiology Section (M.E.), University of Miyazaki Hospital, Miyazaki, Japan.

Z A Khant (ZA)

From the Departments of Radiology (M.A., Z.A.K., T.H.).

H Takeshima (H)

Neurosurgery (K.Y., H.T., Y.A.), Faculty of Medicine, University of Miyazaki, Miyazaki, Japan.

Y Asada (Y)

Neurosurgery (K.Y., H.T., Y.A.), Faculty of Medicine, University of Miyazaki, Miyazaki, Japan.

Y Wang (Y)

Department of Radiology (Y.W.), Weill Medical College of Cornell University, New York, New York.

T Hirai (T)

From the Departments of Radiology (M.A., Z.A.K., T.H.).

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