Model-driven neuromodulation of the right posterior region promotes encoding of long-term memories.


Journal

Brain stimulation
ISSN: 1876-4754
Titre abrégé: Brain Stimul
Pays: United States
ID NLM: 101465726

Informations de publication

Date de publication:
Historique:
received: 27 06 2019
revised: 21 11 2019
accepted: 16 12 2019
pubmed: 29 12 2019
medline: 25 8 2020
entrez: 29 12 2019
Statut: ppublish

Résumé

Long-term recognition memory depends both on initial encoding and on subsequent recognition processes. In this study we aimed at improving long-term memory by modulating posterior parietal brain activity during the encoding process. If this area is causally involved in memory encoding, its facilitation should lead to behavioral improvement. Based on the dual-process memory framework, we also expected that the neuromodulation would dissociate subsequent familiarity-based and recollection-based recognition. We investigated the role of the posterior parietal brain oscillations in facial memory formation in three separate experiments using electroencephalography (EEG), functional magnetic resonance imaging (fMRI), and model-driven, multi-electrode transcranial alternating current stimulation (tACS). Using fMRI and EEG, we confirmed that the right posterior parietal cortex is an essential node that promotes the encoding of long-term memories. We found that single-trial low theta power in this region predicts subsequent long-term recognition. On this basis, we fine-tuned the spatial and frequency settings of tACS during memory encoding. Model-driven tACS over the right posterior brain area augmented subsequent long-term recognition memory and particularly the familiarity of the observed stimuli. The recollection process, and short-term task performance as control remained unchanged. Control stimulation over the left hemisphere had no behavioral effect. We conclude that the right posterior brain area is crucial in long-term memory encoding.

Sections du résumé

BACKGROUND
Long-term recognition memory depends both on initial encoding and on subsequent recognition processes.
OBJECTIVE
In this study we aimed at improving long-term memory by modulating posterior parietal brain activity during the encoding process. If this area is causally involved in memory encoding, its facilitation should lead to behavioral improvement. Based on the dual-process memory framework, we also expected that the neuromodulation would dissociate subsequent familiarity-based and recollection-based recognition.
METHODS
We investigated the role of the posterior parietal brain oscillations in facial memory formation in three separate experiments using electroencephalography (EEG), functional magnetic resonance imaging (fMRI), and model-driven, multi-electrode transcranial alternating current stimulation (tACS).
RESULTS
Using fMRI and EEG, we confirmed that the right posterior parietal cortex is an essential node that promotes the encoding of long-term memories. We found that single-trial low theta power in this region predicts subsequent long-term recognition. On this basis, we fine-tuned the spatial and frequency settings of tACS during memory encoding. Model-driven tACS over the right posterior brain area augmented subsequent long-term recognition memory and particularly the familiarity of the observed stimuli. The recollection process, and short-term task performance as control remained unchanged. Control stimulation over the left hemisphere had no behavioral effect.
CONCLUSION
We conclude that the right posterior brain area is crucial in long-term memory encoding.

Identifiants

pubmed: 31882373
pii: S1935-861X(19)30490-5
doi: 10.1016/j.brs.2019.12.019
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

474-483

Informations de copyright

Copyright © 2019 The Author(s). Published by Elsevier Inc. All rights reserved.

Auteurs

Ivan Alekseichuk (I)

Department of Clinical Neurophysiology, University Medical Center Göttingen, Georg-August University of Göttingen, 37075, Göttingen, Germany; Department of Biomedical Engineering, University of Minnesota, Minneapolis, MN, 55455, USA. Electronic address: ialeksei@umn.edu.

Zsolt Turi (Z)

Department of Clinical Neurophysiology, University Medical Center Göttingen, Georg-August University of Göttingen, 37075, Göttingen, Germany.

Sibel Veit (S)

Department of Clinical Neurophysiology, University Medical Center Göttingen, Georg-August University of Göttingen, 37075, Göttingen, Germany.

Walter Paulus (W)

Department of Clinical Neurophysiology, University Medical Center Göttingen, Georg-August University of Göttingen, 37075, Göttingen, Germany.

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