Third Ventricle Width Assessed by Transcranial Sonography as Predictor of Long-Term Cognitive Impairment.


Journal

Journal of Alzheimer's disease : JAD
ISSN: 1875-8908
Titre abrégé: J Alzheimers Dis
Pays: Netherlands
ID NLM: 9814863

Informations de publication

Date de publication:
2020
Historique:
pubmed: 31 12 2019
medline: 20 11 2020
entrez: 30 12 2019
Statut: ppublish

Résumé

Non-invasive biomarkers of cognitive impairment are needed. We aim to evaluate transcranial sonographic markers as predictors of cognitive impairment in a prospective cohort. To study the changes in the third ventricle diameter and the SN echogenicity between the baseline and the control visit, as well as its association with cognitive performance and the diagnosis of cognitive impairment in a prospective cohort. From the longitudinal population-based Asymptomatic Intracranial Atherosclerosis Study, we selected those subjects that received a complete transcranial sonography (TCS) and extensive cognitive testing, both at baseline and follow-up. We evaluated third ventricle (IIIv) width, echogenicity of substantia nigra (SN), and temporal changes of these parameters. We included 289 participants with a median follow-up time of 7.16 years. Those subjects who developed cognitive decline (n = 23, 7.96%) had a larger IIIv at baseline than those who did not (0.54±0.14 cm versus 0.41±0.15 cm; p = 0.001). A cut-off point of 0.465 cm for the IIIv width was identified as an independent predictor of long-term cognitive impairment after adjustment for age, gender, educational level, and vascular risk score. Change in IIIv diameter after follow-up was not associated with diagnosis of cognitive impairment. The area of SN and the presence of hyperechogenicity of the SN remained stable over time and was not associated with the diagnosis of cognitive impairment. IIIv width assessed by TCS emerged as an independent predictor of long-term cognitive impairment.

Sections du résumé

BACKGROUND
Non-invasive biomarkers of cognitive impairment are needed. We aim to evaluate transcranial sonographic markers as predictors of cognitive impairment in a prospective cohort.
OBJECTIVE
To study the changes in the third ventricle diameter and the SN echogenicity between the baseline and the control visit, as well as its association with cognitive performance and the diagnosis of cognitive impairment in a prospective cohort.
METHODS
From the longitudinal population-based Asymptomatic Intracranial Atherosclerosis Study, we selected those subjects that received a complete transcranial sonography (TCS) and extensive cognitive testing, both at baseline and follow-up. We evaluated third ventricle (IIIv) width, echogenicity of substantia nigra (SN), and temporal changes of these parameters.
RESULTS
We included 289 participants with a median follow-up time of 7.16 years. Those subjects who developed cognitive decline (n = 23, 7.96%) had a larger IIIv at baseline than those who did not (0.54±0.14 cm versus 0.41±0.15 cm; p = 0.001). A cut-off point of 0.465 cm for the IIIv width was identified as an independent predictor of long-term cognitive impairment after adjustment for age, gender, educational level, and vascular risk score. Change in IIIv diameter after follow-up was not associated with diagnosis of cognitive impairment. The area of SN and the presence of hyperechogenicity of the SN remained stable over time and was not associated with the diagnosis of cognitive impairment.
CONCLUSION
IIIv width assessed by TCS emerged as an independent predictor of long-term cognitive impairment.

Identifiants

pubmed: 31884478
pii: JAD190949
doi: 10.3233/JAD-190949
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

741-749

Auteurs

Ane Miren Crespo-Cuevas (AM)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

Elena López-Cancio (E)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.
Department of Neurology, Hospital Universitario Central de Asturias, Oviedo, Spain.

Cynthia Cáceres (C)

Department of Neurosciences, Neuropsychology Unit, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

Anna González (A)

Department of Neurosciences, Neuropsychology Unit, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

Lourdes Ispierto (L)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

María Hernández-Pérez (M)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

María Mataró (M)

Department of Clinical Psychology and Psychobiology, Universitat de Barcelona, Barcelona, Spain.
Institut de Neurociències, University of Barcelona, Barcelona, Spain.
Institut de Recerca Pediàtrica Hospital Sant Joan de Déu, Esplugues de Llobregat, Spain.

Anna Planas (A)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

Tamara Canento (T)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

Lorena Martín (L)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

Juan Francisco Arenillas (JF)

Neurology Service, Hospital Clínico Universitario de Valladolid, Valladolid, Spain.

Ramiro Alvarez (R)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

Dolores Vilas (D)

Department of Neurosciences, Hospital Universitari Germans Trias i Pujol, Badalona, Barcelona, Spain.

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