Epigenetics in Cardiac Hypertrophy and Heart Failure.
BET, bromodomain
EZH2, Enhancer of zeste homolog 2
HAT, histone acetyltransferase
HDAC, histone deacetylase
HDM, histone demethylase
HF, heart failure
HMT, histone methyltransferase
PRC2, polycomb repressor complex 2
PTMs, post-translational modifications
TAD, topologically associating domains
TMAO, trimethylamine N-oxide
cardiac hypertrophy
epigenetics
heart failure
lnc-RNAs, long ncRNAs
Journal
JACC. Basic to translational science
ISSN: 2452-302X
Titre abrégé: JACC Basic Transl Sci
Pays: United States
ID NLM: 101677259
Informations de publication
Date de publication:
Dec 2019
Dec 2019
Historique:
received:
26
02
2019
revised:
29
05
2019
accepted:
30
05
2019
entrez:
8
1
2020
pubmed:
8
1
2020
medline:
8
1
2020
Statut:
epublish
Résumé
Heart failure (HF) is a complex syndrome affecting millions of people around the world. Over the past decade, the therapeutic potential of targeting epigenetic regulators in HF has been discussed extensively. Recent advances in next-generation sequencing techniques have contributed substantial progress in our understanding of the role of DNA methylation, post-translational modifications of histones, adenosine triphosphate (ATP)-dependent chromatin conformation and remodeling, and non-coding RNAs in HF pathophysiology. In this review, we summarize epigenomic studies on human and animal models in HF.
Identifiants
pubmed: 31909304
doi: 10.1016/j.jacbts.2019.05.011
pii: S2452-302X(19)30180-9
pmc: PMC6938823
doi:
Types de publication
Journal Article
Review
Langues
eng
Pagination
976-993Informations de copyright
© 2019 The Authors.
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