Advancing RAS/RASopathy therapies: An NCI-sponsored intramural and extramural collaboration for the study of RASopathies.


Journal

American journal of medical genetics. Part A
ISSN: 1552-4833
Titre abrégé: Am J Med Genet A
Pays: United States
ID NLM: 101235741

Informations de publication

Date de publication:
04 2020
Historique:
received: 17 12 2019
accepted: 22 12 2019
pubmed: 9 1 2020
medline: 4 2 2021
entrez: 9 1 2020
Statut: ppublish

Résumé

RASopathies caused by germline pathogenic variants in genes that encode RAS pathway proteins. These disorders include neurofibromatosis type 1 (NF1), Noonan syndrome (NS), cardiofaciocutaneous syndrome (CFC), and Costello syndrome (CS), and others. RASopathies are characterized by heterogenous manifestations, including congenital heart disease, failure to thrive, and increased risk of cancers. Previous work led by the NCI Pediatric Oncology Branch has altered the natural course of one of the key manifestations of the RASopathy NF1. Through the conduct of a longitudinal cohort study and early phase clinical trials, the MEK inhibitor selumetinib was identified as the first active therapy for the NF1-related peripheral nerve sheath tumors called plexiform neurofibromas (PNs). As a result, selumetinib was granted breakthrough therapy designation by the FDA for the treatment of PN. Other RASopathy manifestations may also benefit from RAS targeted therapies. The overall goal of Advancing RAS/RASopathy Therapies (ART), a new NCI initiative, is to develop effective therapies and prevention strategies for the clinical manifestations of the non-NF1 RASopathies and for tumors characterized by somatic RAS mutations. This report reflects discussions from a February 2019 initiation meeting for this project, which had broad international collaboration from basic and clinical researchers and patient advocates.

Identifiants

pubmed: 31913576
doi: 10.1002/ajmg.a.61485
pmc: PMC7456498
mid: NIHMS1614136
doi:

Substances chimiques

Biomarkers, Tumor 0
ras Proteins EC 3.6.5.2

Types de publication

Journal Article Research Support, N.I.H., Intramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

866-876

Subventions

Organisme : NHLBI NIH HHS
ID : R35 HL135742
Pays : United States
Organisme : Intramural NIH HHS
ID : ZIA CP010144
Pays : United States

Informations de copyright

Published 2020. This article is a U.S. Government work and is in the public domain in the USA.

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Auteurs

Andrea M Gross (AM)

Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland.

Megan Frone (M)

Clinical Genetics Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, Maryland.

Karen W Gripp (KW)

Department of Genetics, Division of Pediatrics, Al duPont Hospital for Children, Wilmington, Delaware.

Bruce D Gelb (BD)

Department of Pediatrics, Mindich Child Health and Development Institute, Icahn School of Medicine at Mount Sinai, New York, New York.
Department of Genetics and Genomic Sciences, Mindich Child Health and Development Institute, Icahn School of Medicine at Mount Sinai, New York, New York.

Lisa Schoyer (L)

RASopathies Network, Altadena, California.

Lisa Schill (L)

RASopathies Network, Altadena, California.

Beth Stronach (B)

RASopathies Network, Altadena, California.

Leslie G Biesecker (LG)

Medical Genomics and Metabolic Genetics Branch, National Human Genome Research Institute, Bethesda, Maryland.

Dominic Esposito (D)

NCI RAS Initiative, Frederick National Laboratory for Cancer Research, Frederick, Maryland.

Edjay Ralph Hernandez (ER)

Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland.

Eric Legius (E)

Laboratory for Neurofibromatosis Research, Department of Human Genetics, KU Leuven University Hospital, Leuven, Belgium.

Mignon L Loh (ML)

Department of Pediatrics, Benioff Children's Hospital and the Helen Diller Family Comprehensive Cancer Center, University of California San Francisco, San Francisco, California.

Staci Martin (S)

Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland.

Deborah K Morrison (DK)

Laboratory of Cell and Developmental Signaling, Center for Cancer Research, National Cancer Institute, Frederick, Maryland.

Katherine A Rauen (KA)

Department of Pediatrics, Division of Genomic Medicine, University of California Davis, Sacramento, California.

Pamela L Wolters (PL)

Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland.

Dina Zand (D)

Center for Drug Evaluation and Research, Food and Drug Administration, Rockville, Maryland.

Frank McCormick (F)

NCI RAS Initiative, Frederick National Laboratory for Cancer Research, Frederick, Maryland.

Sharon A Savage (SA)

Clinical Genetics Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, Maryland.

Douglas R Stewart (DR)

Clinical Genetics Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, Maryland.

Brigitte C Widemann (BC)

Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland.

Marielle E Yohe (ME)

Pediatric Oncology Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland.

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