Mitochondrial Ca
Animals
Autophagy
Calcium
/ metabolism
Calcium Channels
/ genetics
Calcium Signaling
Cell Death
Cell Line
Cytosol
/ metabolism
Diabetes Mellitus, Type 2
/ metabolism
Endoplasmic Reticulum
/ metabolism
Humans
Insulin-Secreting Cells
/ physiology
Membrane Potential, Mitochondrial
Mice
Mitochondria
/ metabolism
Oxidative Stress
Palmitates
/ metabolism
RNA, Small Interfering
/ genetics
cytosolic Ca2+ overload
lipotoxicity
mitochondrial Ca2+ uniporter
oxidative stress
pancreatic β-cell
Journal
Molecules and cells
ISSN: 0219-1032
Titre abrégé: Mol Cells
Pays: Korea (South)
ID NLM: 9610936
Informations de publication
Date de publication:
31 01 2020
31 01 2020
Historique:
received:
27
09
2019
revised:
20
11
2019
accepted:
03
12
2019
pubmed:
14
1
2020
medline:
1
7
2020
entrez:
14
1
2020
Statut:
ppublish
Résumé
Saturated fatty acids contribute to β-cell dysfunction in the onset of type 2 diabetes mellitus. Cellular responses to lipotoxicity include oxidative stress, endoplasmic reticulum (ER) stress, and blockage of autophagy. Palmitate induces ER Ca
Identifiants
pubmed: 31931552
pii: molcells.2019.0223
doi: 10.14348/molcells.2019.0223
pmc: PMC6999716
doi:
Substances chimiques
Calcium Channels
0
Palmitates
0
RNA, Small Interfering
0
mitochondrial calcium uniporter
0
Calcium
SY7Q814VUP
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
66-75Références
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