Impact of microbial contamination of the islet product during total pancreatectomy with islet autotransplantation.


Journal

Journal of hepato-biliary-pancreatic sciences
ISSN: 1868-6982
Titre abrégé: J Hepatobiliary Pancreat Sci
Pays: Japan
ID NLM: 101528587

Informations de publication

Date de publication:
Apr 2020
Historique:
received: 02 10 2019
revised: 22 11 2019
accepted: 08 01 2020
pubmed: 17 1 2020
medline: 16 2 2021
entrez: 17 1 2020
Statut: ppublish

Résumé

The combined use of interleukin-1β and tumor necrosis factor-α blockers in the peritransplant period has improved outcomes of total pancreatectomy with islet autotransplantation (TPIAT). However, these drugs may suppress the immune system, resulting in severe infection. We retrospectively investigated the impact of microbial-contaminated islet product on posttransplant complications and metabolic outcomes of TPIAT patients receiving the IL-1β and TNF-blockade treatment at our center. Among 108 TPIAT patients, 37 patients (34%) received contaminated products. Preoperative stent treatment and fibrosis score were independent risk factors for the contamination. There were no significant differences between the contaminated and noncontaminated product groups in posttransplant infectious complication rate, length of hospitalization, or readmission rate. However, islet equivalents (P < .0001) and insulin independence rate (P = .036) at 6 months were significantly lower for patients receiving contaminated product. These results suggest that combined anti-inflammatory drug use is safe and well tolerated in TPIAT patients who receive contaminated islet product and does not increase the rate of infectious complications; however, contaminated islet product is associated with poor metabolic outcomes.

Sections du résumé

BACKGROUND BACKGROUND
The combined use of interleukin-1β and tumor necrosis factor-α blockers in the peritransplant period has improved outcomes of total pancreatectomy with islet autotransplantation (TPIAT). However, these drugs may suppress the immune system, resulting in severe infection.
METHODS METHODS
We retrospectively investigated the impact of microbial-contaminated islet product on posttransplant complications and metabolic outcomes of TPIAT patients receiving the IL-1β and TNF-blockade treatment at our center.
RESULTS RESULTS
Among 108 TPIAT patients, 37 patients (34%) received contaminated products. Preoperative stent treatment and fibrosis score were independent risk factors for the contamination. There were no significant differences between the contaminated and noncontaminated product groups in posttransplant infectious complication rate, length of hospitalization, or readmission rate. However, islet equivalents (P < .0001) and insulin independence rate (P = .036) at 6 months were significantly lower for patients receiving contaminated product.
CONCLUSIONS CONCLUSIONS
These results suggest that combined anti-inflammatory drug use is safe and well tolerated in TPIAT patients who receive contaminated islet product and does not increase the rate of infectious complications; however, contaminated islet product is associated with poor metabolic outcomes.

Identifiants

pubmed: 31944603
doi: 10.1002/jhbp.709
doi:

Substances chimiques

Anti-Bacterial Agents 0
Anti-Inflammatory Agents, Non-Steroidal 0
C-Peptide 0
Glycated Hemoglobin A 0
Hypoglycemic Agents 0
Immunosuppressive Agents 0
Insulin 0
Interleukin 1 Receptor Antagonist Protein 0
Interleukin-1beta 0
Tumor Necrosis Factor-alpha 0
hemoglobin A1c protein, human 0
Etanercept OP401G7OJC

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

211-218

Informations de copyright

© 2020 Japanese Society of Hepato-Biliary-Pancreatic Surgery.

Références

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Auteurs

Kenjiro Kumano (K)

Baylor Scott and White Research Institute, Dallas, TX, USA.

Morihito Takita (M)

Baylor Scott and White Research Institute, Dallas, TX, USA.

Srividya Vasu (S)

Baylor Scott and White Research Institute, Dallas, TX, USA.

Carly Darden (C)

Baylor University, Waco, TX, USA.

Michael Lawrence (M)

Baylor Scott and White Research Institute, Dallas, TX, USA.

Ernest Beecherl (E)

Baylor Simmons Transplant Institute, Dallas, TX, USA.

Amar Gupta (A)

Baylor Simmons Transplant Institute, Dallas, TX, USA.

Nicholas Onaca (N)

Baylor Simmons Transplant Institute, Dallas, TX, USA.

Bashoo Naziruddin (B)

Baylor Simmons Transplant Institute, Dallas, TX, USA.

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