Sinusoidal obstruction syndrome post-treatment with trastuzumab emtansine (T-DM1) in advanced breast cancer.

Breast cancer Gastric varices Portal hypertension Portal-systemic shunt Sinusoidal obstruction syndrome Trastuzumab emtansine

Journal

International cancer conference journal
ISSN: 2192-3183
Titre abrégé: Int Cancer Conf J
Pays: Singapore
ID NLM: 101734231

Informations de publication

Date de publication:
Jan 2020
Historique:
received: 25 09 2019
accepted: 08 10 2019
entrez: 18 1 2020
pubmed: 18 1 2020
medline: 18 1 2020
Statut: epublish

Résumé

Trastuzumab emtansine (T-DM1) is an antibody-drug conjugate approved for use in the treatment of human epidermal growth factor receptor 2-positive metastatic breast cancer. Here, we present the cases of two patients with metastatic breast cancer who received T-DM1 monotherapy and developed noncirrhotic portal hypertension (NCPH). Patient 1 presented with ruptured gastric varices at 2 years and 5 months after T-DM1 treatment. Patient 2 presented with intrahepatic portal-hepatic venous shunt at 2 years and 6 months and portal-systemic shunt encephalopathy at 4 years and 11 months after T-DM1 treatment. In both the patients, liver biopsies revealed sinusoidal obstruction syndrome (SOS). T-DM1-induced hepatotoxicity can result from SOS. In long-term administration of T-DM1 the unfavorable events associated with chronic liver circulatory disorder due to SOS, such as NCPH, are concerning.

Identifiants

pubmed: 31950012
doi: 10.1007/s13691-019-00392-4
pii: 392
pmc: PMC6942611
doi:

Types de publication

Case Reports

Langues

eng

Pagination

18-23

Informations de copyright

© The Japan Society of Clinical Oncology 2019.

Déclaration de conflit d'intérêts

Conflict of interestThe authors declare that they have no conflict of interest.

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Auteurs

Yasutoshi Fujii (Y)

1Division of Clinical Oncology, Hiroshima Prefectural Hospital, 1-5-54 Ujinakanda, Minami-ku, Hiroshima, 734-8530 Japan.

Mihoko Doi (M)

1Division of Clinical Oncology, Hiroshima Prefectural Hospital, 1-5-54 Ujinakanda, Minami-ku, Hiroshima, 734-8530 Japan.

Naofumi Tsukiyama (N)

1Division of Clinical Oncology, Hiroshima Prefectural Hospital, 1-5-54 Ujinakanda, Minami-ku, Hiroshima, 734-8530 Japan.

Yui Hattori (Y)

2Department of Pathology, Hiroshima Prefectural Hospital, 1-5-54 Ujinakanda, Minami-ku, Hiroshima, 734-8530 Japan.

Kazuki Ohya (K)

3Department of Gastroenterology and Metabolism, Hiroshima University Hospital, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8551 Japan.

Noriyuki Shiroma (N)

4Department of Pathology, Hiroshima University Hospital, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8551 Japan.

Kei Morio (K)

3Department of Gastroenterology and Metabolism, Hiroshima University Hospital, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8551 Japan.

Takehiko Morioka (T)

1Division of Clinical Oncology, Hiroshima Prefectural Hospital, 1-5-54 Ujinakanda, Minami-ku, Hiroshima, 734-8530 Japan.

Hiroshi Aikata (H)

3Department of Gastroenterology and Metabolism, Hiroshima University Hospital, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8551 Japan.

Katsunori Shinozaki (K)

1Division of Clinical Oncology, Hiroshima Prefectural Hospital, 1-5-54 Ujinakanda, Minami-ku, Hiroshima, 734-8530 Japan.

Kazuaki Chayama (K)

3Department of Gastroenterology and Metabolism, Hiroshima University Hospital, 1-2-3 Kasumi, Minami-ku, Hiroshima, 734-8551 Japan.

Classifications MeSH