Cisplatin and 5-fluorouracil with or without epidermal growth factor receptor inhibition panitumumab for patients with non-resectable, advanced or metastatic oesophageal squamous cell cancer: a prospective, open-label, randomised phase III AIO/EORTC trial (POWER).


Journal

Annals of oncology : official journal of the European Society for Medical Oncology
ISSN: 1569-8041
Titre abrégé: Ann Oncol
Pays: England
ID NLM: 9007735

Informations de publication

Date de publication:
02 2020
Historique:
received: 17 06 2019
revised: 10 10 2019
accepted: 15 10 2019
entrez: 22 1 2020
pubmed: 22 1 2020
medline: 7 1 2021
Statut: ppublish

Résumé

Palliative chemotherapy of advanced oesophageal squamous cell cancer (ESCC) consists of cisplatin/5-fluorouracil (CF) to target epidermal growth factor receptor (EGFR) with panitumumab (P); chemotherapy enhanced overall survival (OS) in advanced colorectal or squamous cell head and neck cancers. With prospective serum and tumour biomarkers, we tested if P added to CF (CFP) improved OS in advanced ESCC. Eligible patients with confirmed ESCC that was not curatively resectable or did not qualify for definitive radiochemotherapy, were randomised 1 : 1 to receive CF [cisplatin (C) 100 mg/m The trial was stopped early based on interim efficacy results triggered by the third safety analysis: median OS (mOS) favoured CF over CFP, regardless of cisplatin dose [hazard ratio (HR) 1.77, 95% confidence interval (CI) 1.06-2.98; P = 0.028]. In the final analysis, mOS was 10.2 versus 9.4 months for CF versus CFP, respectively (HR 1.17, 95% CI 0.79-1.75; P = 0.43). One hundred (70.4%) of 142 patients in the safety population died, 51 (51.0%) with CFP. Most deaths were related to disease progression [44/49 (90%) deaths in CF versus 34/51 (67%) deaths in CFP]; objective responses [27/73 (37.0%)] were identical. The most common serious adverse events were kidney injury [3 (4.3%) versus 7 (9.7%)], general health deterioration [5 (7.1%) versus 5 (6.9%)] and dysphagia [4 (5.7%) versus 4 (5.6%)] in CF versus CFP, respectively. There were three (4.3%) and 17 (23.6%) common terminology criteria for adverse events (CTCAE) grade 5 events in CF versus CFP, respectively. Low soluble (s)EGFR levels were associated with better progression-free survival; sEGFR was induced under CFP. EGFR inhibition added to CF did not improve survival in unselected advanced ESCC patients. The results support further liquid biopsy studies. ClinicalTrials.gov (NCT01627379) and EudraCT (2010-020606-15).

Sections du résumé

BACKGROUND
Palliative chemotherapy of advanced oesophageal squamous cell cancer (ESCC) consists of cisplatin/5-fluorouracil (CF) to target epidermal growth factor receptor (EGFR) with panitumumab (P); chemotherapy enhanced overall survival (OS) in advanced colorectal or squamous cell head and neck cancers. With prospective serum and tumour biomarkers, we tested if P added to CF (CFP) improved OS in advanced ESCC.
PATIENTS AND METHODS
Eligible patients with confirmed ESCC that was not curatively resectable or did not qualify for definitive radiochemotherapy, were randomised 1 : 1 to receive CF [cisplatin (C) 100 mg/m
RESULTS
The trial was stopped early based on interim efficacy results triggered by the third safety analysis: median OS (mOS) favoured CF over CFP, regardless of cisplatin dose [hazard ratio (HR) 1.77, 95% confidence interval (CI) 1.06-2.98; P = 0.028]. In the final analysis, mOS was 10.2 versus 9.4 months for CF versus CFP, respectively (HR 1.17, 95% CI 0.79-1.75; P = 0.43). One hundred (70.4%) of 142 patients in the safety population died, 51 (51.0%) with CFP. Most deaths were related to disease progression [44/49 (90%) deaths in CF versus 34/51 (67%) deaths in CFP]; objective responses [27/73 (37.0%)] were identical. The most common serious adverse events were kidney injury [3 (4.3%) versus 7 (9.7%)], general health deterioration [5 (7.1%) versus 5 (6.9%)] and dysphagia [4 (5.7%) versus 4 (5.6%)] in CF versus CFP, respectively. There were three (4.3%) and 17 (23.6%) common terminology criteria for adverse events (CTCAE) grade 5 events in CF versus CFP, respectively. Low soluble (s)EGFR levels were associated with better progression-free survival; sEGFR was induced under CFP.
CONCLUSION
EGFR inhibition added to CF did not improve survival in unselected advanced ESCC patients. The results support further liquid biopsy studies.
TRIAL REGISTRATION
ClinicalTrials.gov (NCT01627379) and EudraCT (2010-020606-15).

Identifiants

pubmed: 31959339
pii: S0923-7534(19)36074-0
doi: 10.1016/j.annonc.2019.10.018
pii:
doi:

Substances chimiques

Panitumumab 6A901E312A
ErbB Receptors EC 2.7.10.1
Cisplatin Q20Q21Q62J
Fluorouracil U3P01618RT

Banques de données

ClinicalTrials.gov
['NCT01627379']
EudraCT
['2010-020606-15']

Types de publication

Clinical Trial, Phase III Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

228-235

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019 European Society for Medical Oncology. Published by Elsevier Ltd. All rights reserved.

Auteurs

M Moehler (M)

1st Department of Internal Medicine, Johannes Gutenberg-University Mainz, Mainz, Germany. Electronic address: markus.moehler@unimedizin-mainz.de.

A Maderer (A)

1st Department of Internal Medicine, Johannes Gutenberg-University Mainz, Mainz, Germany.

P C Thuss-Patience (PC)

Medical Department, Division of Hematology, Oncology and Tumor Immunology, Charité - University Medicine Berlin, Berlin, Germany.

B Brenner (B)

Institute of Oncology, Davidoff Center, Rabin Medical Center, Petach Tikva and Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

J Meiler (J)

Department of Internal Medicine, University Hospital Essen, Essen, Germany.

T J Ettrich (TJ)

Department of Internal Medicine I, University Hospital Ulm, Ulm, Germany.

R-D Hofheinz (RD)

Medical Department III, University Hospital Mannheim, Mannheim, Germany.

S E Al-Batran (SE)

Institute of Clinical Cancer Research, Hospital North-West, Frankfurt, Germany.

A Vogel (A)

Department of Gastroenterology, Hepatology and Endocrinology, Hannover Medical School, Hannover, Germany.

L Mueller (L)

Oncology Leer-Emden-Papenburg, Leer, Germany.

M P Lutz (MP)

Gastroenterology, Caritas Hospital, Saarbrücken, Germany.

F Lordick (F)

1st Medical Department and University Cancer Center Leipzig, University of Leipzig Medical Center, Leipzig, Germany.

M Alsina (M)

Department of Medical Oncology, Hospital Universitari Vall d'Hebron and Vall d'Hebron Institute of Oncology, Universitat Autònoma de Barcelona, Barcelona, Spain.

K Borchert (K)

Medical Department III, University Hospital Rostock, Rostock, Germany.

R Greil (R)

3rd Medical Department, Cancer Research Institute, Paracelsus Medical University Salzburg, Salzburg, Austria.

W Eisterer (W)

Department of Internal Medicine V, Medical University Innsbruck, Innsbruck, Austria.

A Schad (A)

Institute of Pathology, Johannes Gutenberg-University Mainz, Mainz, Germany.

J Slotta-Huspenina (J)

Institute of Pathology, School of Medicine, Technical University of Munich, Munich, Germany.

E Van Cutsem (E)

University Hospitals Gasthuisberg Leuven and KU Leuven, Leuven, Belgium.

S Lorenzen (S)

Medical Department III, Klinikum rechts der Isar, Technical University of Munich, Munich, Germany.

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Classifications MeSH