Women who received varicella vaccine versus natural infection have different long-term T cell immunity but similar antibody levels.


Journal

Vaccine
ISSN: 1873-2518
Titre abrégé: Vaccine
Pays: Netherlands
ID NLM: 8406899

Informations de publication

Date de publication:
11 02 2020
Historique:
received: 02 07 2019
revised: 24 12 2019
accepted: 31 12 2019
pubmed: 22 1 2020
medline: 9 3 2021
entrez: 22 1 2020
Statut: ppublish

Résumé

Varicella-zoster virus (VZV) infection during pregnancy is associated with serious fetal anomalies. The live-attenuated VZV vaccine was approved in 1995, so many vaccinated women are now of childbearing age. The question of long-term immunity to varicella is critical because breakthrough chickenpox can occur after vaccination. To compare humoral and T cell immunity between women of childbearing age who were immunized by vaccination or chickenpox disease. Non-pregnant females between 18 and 36 years old with a history of VZV immunization (n = 20) or prior chickenpox disease (n = 20) were recruited. IgG antibody titers and T cell responses were measured by flow cytometry-based methods in serum and peripheral blood, respectively. There were no significant differences in median antibody titers between vaccinated and chickenpox groups (p = 0.34). The chickenpox group had significantly higher levels of VZV antigen-specific CD4 T cells (p = 0.004). Natural infection induced higher VZV-specific T cell immune responses than vaccination.

Sections du résumé

BACKGROUND
Varicella-zoster virus (VZV) infection during pregnancy is associated with serious fetal anomalies. The live-attenuated VZV vaccine was approved in 1995, so many vaccinated women are now of childbearing age. The question of long-term immunity to varicella is critical because breakthrough chickenpox can occur after vaccination.
OBJECTIVE
To compare humoral and T cell immunity between women of childbearing age who were immunized by vaccination or chickenpox disease.
STUDY DESIGN
Non-pregnant females between 18 and 36 years old with a history of VZV immunization (n = 20) or prior chickenpox disease (n = 20) were recruited. IgG antibody titers and T cell responses were measured by flow cytometry-based methods in serum and peripheral blood, respectively.
RESULTS
There were no significant differences in median antibody titers between vaccinated and chickenpox groups (p = 0.34). The chickenpox group had significantly higher levels of VZV antigen-specific CD4 T cells (p = 0.004).
CONCLUSION
Natural infection induced higher VZV-specific T cell immune responses than vaccination.

Identifiants

pubmed: 31959424
pii: S0264-410X(20)30003-7
doi: 10.1016/j.vaccine.2019.12.067
pii:
doi:

Substances chimiques

Antibodies, Viral 0
Chickenpox Vaccine 0
Immunoglobulin G 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1581-1585

Subventions

Organisme : NIAID NIH HHS
ID : R24 AI054953
Pays : United States

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Ellen Tourtelot (E)

University of Rochester, Strong Memorial Hospital, Department of Obstetrics and Gynecology, United States. Electronic address: Ellen_tourtelot@urmc.rochester.edu.

Sally Quataert (S)

University of Rochester, Strong Memorial Hospital, Department of Vaccine Biology & Immunology, United States.

J Christopher Glantz (JC)

University of Rochester, Strong Memorial Hospital, Department of Obstetrics and Gynecology, United States.

Lauren Perlis (L)

University of Rochester, Strong Memorial Hospital, Department of Obstetrics and Gynecology, United States.

Gowrishankar Muthukrishnan (G)

University of Rochester, Strong Memorial Hospital, Department of Vaccine Biology & Immunology, United States.

Tim Mosmann (T)

University of Rochester, Strong Memorial Hospital, Department of Vaccine Biology & Immunology, United States.

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Classifications MeSH