Lack of Fetal Insulin Resistance in Maternal Polycystic Ovary Syndrome.


Journal

Reproductive sciences (Thousand Oaks, Calif.)
ISSN: 1933-7205
Titre abrégé: Reprod Sci
Pays: United States
ID NLM: 101291249

Informations de publication

Date de publication:
06 2020
Historique:
received: 01 08 2019
accepted: 01 11 2019
pubmed: 30 1 2020
medline: 15 12 2020
entrez: 30 1 2020
Statut: ppublish

Résumé

Polycystic ovary syndrome (PCOS) affects 8-10% of women. NIH criteria for diagnosis include chronic anovulation and evidence of clinical or biochemical hyperandrogenism. PCOS is associated with adverse neonatal outcomes. Our hypothesis is that insulin resistance is increased in fetuses born to women with PCOS. This is a prospective cohort of women who delivered at our institution. Subjects with a body mass index < 20 or ≥ 50 kg/m

Identifiants

pubmed: 31994004
doi: 10.1007/s43032-019-00125-1
pii: 10.1007/s43032-019-00125-1
doi:

Substances chimiques

Blood Glucose 0
Insulin 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1253-1258

Auteurs

Ana M Tobiasz (AM)

Department of Obstetrics and Gynecology, University of Tennessee Health Science Center, Memphis, TN, USA. atobiasz@me.com.
Department of Obstetrics and Gynecology, Sanford Obstetrics & Gynecology, University of North Dakota, 414 N Seventh Street, Bismarck, ND, 58501, USA. atobiasz@me.com.

Jose R Duncan (JR)

Department of Obstetrics and Gynecology, University of Tennessee Health Science Center, Memphis, TN, USA.
Department of Obstetrics and Gynecology, University of South Florida, FL, Tampa, USA.

Laura Detti (L)

Department of Obstetrics and Gynecology, University of Tennessee Health Science Center, Memphis, TN, USA.

Giancarlo Mari (G)

Department of Obstetrics and Gynecology, University of Tennessee Health Science Center, Memphis, TN, USA.

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Classifications MeSH