Carotid Wallstent Versus Roadsaver Stent and Distal Versus Proximal Protection on Cerebral Microembolization During Carotid Artery Stenting.


Journal

JACC. Cardiovascular interventions
ISSN: 1876-7605
Titre abrégé: JACC Cardiovasc Interv
Pays: United States
ID NLM: 101467004

Informations de publication

Date de publication:
24 02 2020
Historique:
received: 02 07 2019
revised: 14 08 2019
accepted: 04 09 2019
pubmed: 3 2 2020
medline: 29 9 2020
entrez: 3 2 2020
Statut: ppublish

Résumé

The aim of this study was to randomly compare the double-layer Roadsaver stent (RS) (Terumo, Tokyo, Japan) with the single-layer Carotid Wallstent (CW) (Boston Scientific, Santa Clara, California) in association with either distal embolic protection with the FilterWire (FW) device (Boston Scientific) or proximal protection with the Mo.Ma Ultra device (Medtronic, Santa Rosa, California) in patients with lipid-rich carotid plaques. The role of both stent type and brain protection during carotid artery stenting (CAS) remains unsettled. A total of 104 consecutive patients with carotid artery stenosis were randomized to CAS with FW + RS (group 1, n = 27), FW + CW (group 2, n = 25), Mo.Ma + RS (group 3, n = 27), or Mo.Ma + CW (group 4, n = 25). The primary endpoint was the number of microembolic signals (MES) on transcranial Doppler among groups in the following CAS steps: 1 and 2) target vessel access; 3) lesion wiring; 4) pre-dilation; 5) stent crossing; 6) stent deployment; 7) stent dilation; and 8) device retrieval and deflation. No significant differences in baseline characteristics were found among the 4 groups. Compared with the FW device, the Mo.Ma Ultra device significantly reduced mean MES count (p < 0.0001) during lesion crossing, stent crossing, stent deployment, and post-dilation. Compared with the CW, the RS significantly reduced MES count (p = 0.016) in steps 6 to 8, including spontaneous MES (29% of patients). The combination of Mo.Ma + RS performed significantly better than Mo.Ma + CW (p = 0.043). Clinical major adverse cardiac and cerebrovascular events occurred in 3 patients (p = 0.51). After CAS, peak systolic velocity significantly decreased in all patients. In-stent restenosis developed in 1 patient (0.98%) at 6-month follow-up. The RS was an independent predictor of external carotid artery patency over time. In patients with high-risk, lipid-rich plaque undergoing CAS, Mo.Ma + RS led to the lowest microembolic signals count. (Role of the Type of Carotid Stent and Cerebral Protection on Cerebral Microembolization During Carotid Artery Stenting. A Randomized Study Comparing Carotid Wallstent vs Roadsaver® Stent and Distal vs Proximal Protection; NCT02915328).

Sections du résumé

OBJECTIVES
The aim of this study was to randomly compare the double-layer Roadsaver stent (RS) (Terumo, Tokyo, Japan) with the single-layer Carotid Wallstent (CW) (Boston Scientific, Santa Clara, California) in association with either distal embolic protection with the FilterWire (FW) device (Boston Scientific) or proximal protection with the Mo.Ma Ultra device (Medtronic, Santa Rosa, California) in patients with lipid-rich carotid plaques.
BACKGROUND
The role of both stent type and brain protection during carotid artery stenting (CAS) remains unsettled.
METHODS
A total of 104 consecutive patients with carotid artery stenosis were randomized to CAS with FW + RS (group 1, n = 27), FW + CW (group 2, n = 25), Mo.Ma + RS (group 3, n = 27), or Mo.Ma + CW (group 4, n = 25). The primary endpoint was the number of microembolic signals (MES) on transcranial Doppler among groups in the following CAS steps: 1 and 2) target vessel access; 3) lesion wiring; 4) pre-dilation; 5) stent crossing; 6) stent deployment; 7) stent dilation; and 8) device retrieval and deflation.
RESULTS
No significant differences in baseline characteristics were found among the 4 groups. Compared with the FW device, the Mo.Ma Ultra device significantly reduced mean MES count (p < 0.0001) during lesion crossing, stent crossing, stent deployment, and post-dilation. Compared with the CW, the RS significantly reduced MES count (p = 0.016) in steps 6 to 8, including spontaneous MES (29% of patients). The combination of Mo.Ma + RS performed significantly better than Mo.Ma + CW (p = 0.043). Clinical major adverse cardiac and cerebrovascular events occurred in 3 patients (p = 0.51). After CAS, peak systolic velocity significantly decreased in all patients. In-stent restenosis developed in 1 patient (0.98%) at 6-month follow-up. The RS was an independent predictor of external carotid artery patency over time.
CONCLUSIONS
In patients with high-risk, lipid-rich plaque undergoing CAS, Mo.Ma + RS led to the lowest microembolic signals count. (Role of the Type of Carotid Stent and Cerebral Protection on Cerebral Microembolization During Carotid Artery Stenting. A Randomized Study Comparing Carotid Wallstent vs Roadsaver® Stent and Distal vs Proximal Protection; NCT02915328).

Identifiants

pubmed: 32007460
pii: S1936-8798(19)31930-2
doi: 10.1016/j.jcin.2019.09.007
pii:
doi:

Banques de données

ClinicalTrials.gov
['NCT02915328']

Types de publication

Comparative Study Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

403-414

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2020 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

Auteurs

Piero Montorsi (P)

Department of Clinical Sciences and Community Health, University of Milan, Milan, Italy; Centro Cardiologico Monzino, IRCCS, Milan, Italy. Electronic address: piero.montorsi@ccfm.it.

Luigi Caputi (L)

Department of Cerebrovascular Disease, Fondazione IRCCS Istituto Neurologico "C. Besta," Milan, Italy.

Stefano Galli (S)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Paolo M Ravagnani (PM)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Giovanni Teruzzi (G)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Andrea Annoni (A)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Giuseppe Calligaris (G)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Franco Fabbiocchi (F)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Daniela Trabattoni (D)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Stefano de Martini (S)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Luca Grancini (L)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Gianluca Pontone (G)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Daniele Andreini (D)

Department of Clinical Sciences and Community Health, University of Milan, Milan, Italy; Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Sarah Troiano (S)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Davide Restelli (D)

Centro Cardiologico Monzino, IRCCS, Milan, Italy.

Antonio L Bartorelli (AL)

Department of Clinical Sciences and Community Health, University of Milan, Milan, Italy; Centro Cardiologico Monzino, IRCCS, Milan, Italy.

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