Messenger-RNA Expression of Five Gemcitabine Sensitivity-related Genes Predicting Outcome in Advanced-stage Non-small Cell Lung Cancer.


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
Feb 2020
Historique:
received: 03 12 2019
revised: 14 12 2019
accepted: 16 12 2019
entrez: 5 2 2020
pubmed: 6 2 2020
medline: 20 3 2020
Statut: ppublish

Résumé

Tumoural transcriptional levels of RRM1, RRM2, CDA, dCK and hENT1 genes are potential biomarkers for gemcitabine's efficacy in non-small cell lung cancer (NSCLC). We retrospectively analysed each gene's relative mRNA expression by quantitative, real-time polymerase chain reaction in microdissected, formalin-fixed, paraffin-embedded primary-tumour specimens from 219 chemonaïve patients with advanced-stage NSCLC, treated with gemcitabine-based regimens within clinical trials. The five genes' transcriptional patterns were integrated into an ordinal, five-level gemcitabine-susceptibility classifier (5L-GSC). Treatment efficacy increased progressively across the five susceptibility levels, with the very-high chemosensitivity cases obtaining the most clinical benefit. 5L-GSC emerged as an independent prognosticator for overall response and disease control rates, time to progression and overall survival at p-values of 0.03, 0.004, <0.001 and <0.001, respectively, with results remaining significant after bootstrapping. Penalised, optimally-scaled, categorical-regression modelling of overall response identified 5L-GSC as the most stable predictor. The proposed composite biomarker is promising for customising front-line chemotherapy in NSCLC.

Sections du résumé

BACKGROUND/AIM OBJECTIVE
Tumoural transcriptional levels of RRM1, RRM2, CDA, dCK and hENT1 genes are potential biomarkers for gemcitabine's efficacy in non-small cell lung cancer (NSCLC).
PATIENTS AND METHODS METHODS
We retrospectively analysed each gene's relative mRNA expression by quantitative, real-time polymerase chain reaction in microdissected, formalin-fixed, paraffin-embedded primary-tumour specimens from 219 chemonaïve patients with advanced-stage NSCLC, treated with gemcitabine-based regimens within clinical trials. The five genes' transcriptional patterns were integrated into an ordinal, five-level gemcitabine-susceptibility classifier (5L-GSC).
RESULTS RESULTS
Treatment efficacy increased progressively across the five susceptibility levels, with the very-high chemosensitivity cases obtaining the most clinical benefit. 5L-GSC emerged as an independent prognosticator for overall response and disease control rates, time to progression and overall survival at p-values of 0.03, 0.004, <0.001 and <0.001, respectively, with results remaining significant after bootstrapping. Penalised, optimally-scaled, categorical-regression modelling of overall response identified 5L-GSC as the most stable predictor.
CONCLUSION CONCLUSIONS
The proposed composite biomarker is promising for customising front-line chemotherapy in NSCLC.

Identifiants

pubmed: 32014934
pii: 40/2/901
doi: 10.21873/anticanres.14023
doi:

Substances chimiques

RNA, Messenger 0
Deoxycytidine 0W860991D6
Gemcitabine 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

901-913

Informations de copyright

Copyright© 2020, International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Georgios Ioannidis (G)

Laboratory of Translational Oncology, School of Medicine, University of Crete, Heraklion, Greece.
Oncology Department, Nicosia General Hospital, Nicosia, Cyprus.

Chara Papadaki (C)

Laboratory of Translational Oncology, School of Medicine, University of Crete, Heraklion, Greece.

Eleni Lagoudaki (E)

Laboratory of Pathology, School of Medicine, University of Crete, Heraklion, Greece.

Maria Tzardi (M)

Laboratory of Pathology, School of Medicine, University of Crete, Heraklion, Greece.

Maria Trypaki (M)

Laboratory of Translational Oncology, School of Medicine, University of Crete, Heraklion, Greece.

Efstathios Stathopoulos (E)

School of Medicine, University of Crete, Heraklion, Greece.

Dimitrios Mavroudis (D)

Laboratory of Translational Oncology, School of Medicine, University of Crete, Heraklion, Greece.
Department of Medical Oncology, University General Hospital of Heraklion, Heraklion, Greece.

Vassilios Georgoulias (V)

School of Medicine, University of Crete, Heraklion, Greece.

John Souglakos (J)

Laboratory of Translational Oncology, School of Medicine, University of Crete, Heraklion, Greece oncsec@med.uoc.gr.
Department of Medical Oncology, University General Hospital of Heraklion, Heraklion, Greece.

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Classifications MeSH