Underlying embolic and pathologic differentiation by cerebral microbleeds in cryptogenic stroke.

Aortic complicated lesion Cerebral microbleeds Cryptogenic stroke Embolic stroke of undetermined source Transesophageal echocardiography

Journal

Journal of neurology
ISSN: 1432-1459
Titre abrégé: J Neurol
Pays: Germany
ID NLM: 0423161

Informations de publication

Date de publication:
May 2020
Historique:
received: 14 11 2019
accepted: 25 01 2020
revised: 24 01 2020
pubmed: 6 2 2020
medline: 9 2 2021
entrez: 5 2 2020
Statut: ppublish

Résumé

Cryptogenic stroke encompasses diverse emboligenic mechanisms and pathogeneses. Cerebral microbleeds (CMBs) occur differently among stroke subtypes. The association of CMBs with cryptogenic stroke is essentially unknown. CHALLENGE ESUS/CS (Mechanisms of Embolic Stroke Clarified by Transesophageal Echocardiography for ESUS/CS) is a multicenter registry with comprehensive data including gradient-echo T2*-weighted magnetic resonance imaging of cryptogenic stroke patients who underwent transesophageal echocardiography. Patients' clinical characteristics were compared according to the presence and location of CMBs. A total of 661 patients (68.7 ± 12.7 years; 445 males) were enrolled, and 209 (32%) had CMBs. Age (odds ratio [OR] 1.02, 95% confidence interval [CI] 1.00-1.04, p = 0.020), male sex (OR 1.85, 95% CI 1.18-2.91, p = 0.007), hypertension (OR 1.71, 95% CI 1.03-2.86, p = 0.039), chronic kidney disease (OR 1.64, 95% CI 1.11-2.43, p = 0.013), deep and subcortical white matter hyperintensity (OR 1.82, 95% CI 1.16-2.85, p = 0.009), and periventricular hyperintensity (OR 2.18, 95% CI 1.37-3.46, p = 0.001) were independently associated with the presence of CMBs. Aortic complicated lesions (OR 1.78, 95% CI 1.12-2.84, p = 0.015) were associated with deep and diffuse CMBs, whereas prior anticoagulant therapy (OR 7.88, 95% CI, 1.83-33.9, p = 0.006) was related to lobar CMBs. CMBs were common, and age, male sex, hypertension, chronic kidney disease, and cerebral white matter diseases were related to CMBs in cryptogenic stroke. Aortic complicated lesions were associated with deep and diffuse CMBs, while prior anticoagulant therapy was related to lobar CMBs.

Sections du résumé

BACKGROUND BACKGROUND
Cryptogenic stroke encompasses diverse emboligenic mechanisms and pathogeneses. Cerebral microbleeds (CMBs) occur differently among stroke subtypes. The association of CMBs with cryptogenic stroke is essentially unknown.
METHODS METHODS
CHALLENGE ESUS/CS (Mechanisms of Embolic Stroke Clarified by Transesophageal Echocardiography for ESUS/CS) is a multicenter registry with comprehensive data including gradient-echo T2*-weighted magnetic resonance imaging of cryptogenic stroke patients who underwent transesophageal echocardiography. Patients' clinical characteristics were compared according to the presence and location of CMBs.
RESULTS RESULTS
A total of 661 patients (68.7 ± 12.7 years; 445 males) were enrolled, and 209 (32%) had CMBs. Age (odds ratio [OR] 1.02, 95% confidence interval [CI] 1.00-1.04, p = 0.020), male sex (OR 1.85, 95% CI 1.18-2.91, p = 0.007), hypertension (OR 1.71, 95% CI 1.03-2.86, p = 0.039), chronic kidney disease (OR 1.64, 95% CI 1.11-2.43, p = 0.013), deep and subcortical white matter hyperintensity (OR 1.82, 95% CI 1.16-2.85, p = 0.009), and periventricular hyperintensity (OR 2.18, 95% CI 1.37-3.46, p = 0.001) were independently associated with the presence of CMBs. Aortic complicated lesions (OR 1.78, 95% CI 1.12-2.84, p = 0.015) were associated with deep and diffuse CMBs, whereas prior anticoagulant therapy (OR 7.88, 95% CI, 1.83-33.9, p = 0.006) was related to lobar CMBs.
CONCLUSIONS CONCLUSIONS
CMBs were common, and age, male sex, hypertension, chronic kidney disease, and cerebral white matter diseases were related to CMBs in cryptogenic stroke. Aortic complicated lesions were associated with deep and diffuse CMBs, while prior anticoagulant therapy was related to lobar CMBs.

Identifiants

pubmed: 32016623
doi: 10.1007/s00415-020-09732-4
pii: 10.1007/s00415-020-09732-4
doi:

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1482-1490

Investigateurs

Ayano Suzuki (A)
Kodai Kanemaru (K)
Tadashi Kanamoto (T)
Kenichiro Hira (K)
Yuta Hagiwara (Y)
Takahiko Kikuchi (T)

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Auteurs

Muneaki Kikuno (M)

Department of Cerebrovascular Medicine, National Cerebral and Cardiovascular Center, Osaka, Japan.
Department of Neurology, Tokyo Medical University Hospital, Tokyo, Japan.

Yuji Ueno (Y)

Department of Neurology, Juntendo University Faculty of Medicine, 2-1-1 Hongo, Bunkyo-ku, Tokyo, 113-8421, Japan. yuji-u@juntendo.ac.jp.

Takahiro Shimizu (T)

Department of Neurology, St. Marianna University School of Medicine, Kanagawa, Japan.

Ayako Kuriki (A)

Department of Neurology, Showa University Koto Toyosu Hospital, Tokyo, Japan.

Yohei Tateishi (Y)

Department of Neurology and Strokology, Nagasaki University Hospital, Nagasaki, Japan.

Ryosuke Doijiri (R)

Department of Neurology, Iwate Prefectural Central Hospital, Iwate, Japan.

Yoshiaki Shimada (Y)

Department of Neurology, Juntendo University Urayasu Hospital, Chiba, Japan.

Hidehiro Takekawa (H)

Department of Neurology, Dokkyo Medical University, Tochigi, Japan.

Eriko Yamaguchi (E)

Department of Neurology, Iwate Prefectural Central Hospital, Iwate, Japan.

Masatoshi Koga (M)

Department of Cerebrovascular Medicine, National Cerebral and Cardiovascular Center, Osaka, Japan.

Yuki Kamiya (Y)

Department of Neurology, Showa University Koto Toyosu Hospital, Tokyo, Japan.

Masafumi Ihara (M)

Department of Neurology, National Cerebral and Cardiovascular Center, Osaka, Japan.

Akira Tsujino (A)

Department of Neurology and Strokology, Nagasaki University Hospital, Nagasaki, Japan.

Koichi Hirata (K)

Department of Neurology, Dokkyo Medical University, Tochigi, Japan.

Kazunori Toyoda (K)

Department of Cerebrovascular Medicine, National Cerebral and Cardiovascular Center, Osaka, Japan.

Yasuhiro Hasegawa (Y)

Department of Neurology, St. Marianna University School of Medicine, Kanagawa, Japan.

Hitoshi Aizawa (H)

Department of Neurology, Tokyo Medical University Hospital, Tokyo, Japan.

Nobutaka Hattori (N)

Department of Neurology, Juntendo University Faculty of Medicine, 2-1-1 Hongo, Bunkyo-ku, Tokyo, 113-8421, Japan.

Takao Urabe (T)

Department of Neurology, Juntendo University Urayasu Hospital, Chiba, Japan.

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