Colistin-induced acquired Bartter-like syndrome: an unusual cause of meltdown.
drugs: infectious diseases
fluid electrolyte and acid-base disturbances
infections
Journal
BMJ case reports
ISSN: 1757-790X
Titre abrégé: BMJ Case Rep
Pays: England
ID NLM: 101526291
Informations de publication
Date de publication:
05 Feb 2020
05 Feb 2020
Historique:
entrez:
8
2
2020
pubmed:
8
2
2020
medline:
8
10
2020
Statut:
epublish
Résumé
Colistin-induced nephrotoxicity is commonly associated with elevation of serum creatinine level or a reduction of urine output. Uncommonly, tubulopathy associated with colistin has been reported. Here we present a unique case of a 46-year-old man who developed polyuria, hypokalaemia, hypocalcaemia, hypomagnesemia and metabolic alkalosis after 3 days of therapy with intravenous colistimethate sodium. After ruling out other causes, a diagnosis of colistin-induced acquired Bartter syndrome was made. The patient required daily aggressive intravenous repletion of fluids and electrolytes. However, polyuria and metabolic abnormalities abated only after drug discontinuation.
Identifiants
pubmed: 32029515
pii: 13/2/e232630
doi: 10.1136/bcr-2019-232630
pmc: PMC7021182
pii:
doi:
Substances chimiques
colistinmethanesulfonic acid
DL2R53P963
Colistin
Z67X93HJG1
Types de publication
Case Reports
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Informations de copyright
© BMJ Publishing Group Limited 2020. No commercial re-use. See rights and permissions. Published by BMJ.
Déclaration de conflit d'intérêts
Competing interests: None declared.
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