Discovery of a novel dehydratase of the fatty acid synthase type II critical for ketomycolic acid biosynthesis and virulence of Mycobacterium tuberculosis.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
07 02 2020
Historique:
received: 13 05 2019
accepted: 20 01 2020
entrez: 9 2 2020
pubmed: 9 2 2020
medline: 13 11 2020
Statut: epublish

Résumé

The fatty acid synthase type II (FAS-II) multienzyme system builds the main chain of mycolic acids (MAs), important lipid pathogenicity factors of Mycobacterium tuberculosis (Mtb). Due to their original structure, the identification of the (3 R)-hydroxyacyl-ACP dehydratases, HadAB and HadBC, of Mtb FAS-II complex required in-depth work. Here, we report the discovery of a third dehydratase protein, HadD

Identifiants

pubmed: 32034201
doi: 10.1038/s41598-020-58967-8
pii: 10.1038/s41598-020-58967-8
pmc: PMC7005898
doi:

Substances chimiques

Bacterial Proteins 0
Mycolic Acids 0
Hydro-Lyases EC 4.2.1.-
Enoyl-CoA Hydratase EC 4.2.1.17
Fatty Acid Synthase, Type II EC 6.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2112

Références

WHO. Global tuberculosis report 2019. WHO Press edn, (World Health Organization, 2019).
Daffé, M., Quémard, A. & Marrakchi, H. Mycolic acids: from Chemistry to Biology in Biogenesis of Fatty Acids, Lipids and Membranes (ed Geiger, O.) 181–216, Handbook of Hydrocarbon and Lipid Microbiology series (Springer, Cham, 2019).
Quémard, A. et al. Binding of catalase-peroxidase-activated isoniazid to wild-type and mutant Mycobacterium tuberculosis enoyl-ACP reductases. J. Am. Chem. Soc. 118, 1561–1562 (1996).
doi: 10.1021/ja950998b
North, E. J., Jackson, M. & Lee, R. E. New Approaches to Target the Mycolic Acid Biosynthesis Pathway for the Development of Tuberculosis Therapeutics. Curr. Pharm. Des 20, 4357–4378 (2014).
doi: 10.2174/1381612819666131118203641
Zuber, B. et al. Direct visualization of the outer membrane of mycobacteria and corynebacteria in their native state. J. Bacteriol. 190, 5672–5680 (2008).
doi: 10.1128/JB.01919-07
Quémard, A. New Insights into the Mycolate-Containing Compound Biosynthesis and Transport in Mycobacteria. Trends Microbiol. 24, 725–738, https://doi.org/10.1016/j.tim.2016.04.009 (2016).
doi: 10.1016/j.tim.2016.04.009 pubmed: 27268593
Rock, C. O. & Cronan, J. E. Escherichia coli as a model for the regulation of dissociable (type II) fatty acid biosynthesis. Biochim. Biophys. Acta 1302, 1–16 (1996).
doi: 10.1016/0005-2760(96)00056-2
Bloch, K. Control mechanisms for fatty acid synthesis in Mycobacterium smegmatis. Adv. Enzymol. Relat. Areas Mol. Biol 45, 1–84 (1977).
pubmed: 21523
Schaeffer, M. L. et al. Purification and biochemical characterization of the Mycobacterium tuberculosis beta-ketoacyl-acyl carrier protein synthases KasA and KasB. J. Biol. Chem. 276, 47029–47037 (2001).
doi: 10.1074/jbc.M108903200
Kremer, L. et al. Mycolic acid biosynthesis and enzymic characterization of the beta-ketoacyl-ACP synthase A-condensing enzyme from Mycobacterium tuberculosis. Biochem. J 364, 423–430 (2002).
doi: 10.1042/bj20011628
Marrakchi, H. et al. MabA (FabG1), a Mycobacterium tuberculosis protein involved in the long-chain fatty acid elongation system FAS-II. Microbiology 148, 951–960 (2002).
doi: 10.1099/00221287-148-4-951
Quémard, A. et al. Enzymatic characterization of the target for isoniazid in Mycobacterium tuberculosis. Biochemistry 34, 8235–8241 (1995).
doi: 10.1021/bi00026a004
Sacco, E. et al. The missing piece of the type II fatty acid synthase system from Mycobacterium tuberculosis. Proc. Natl. Acad. Sci. USA 104, 14628–14633, https://doi.org/10.1073/pnas.0704132104 (2007).
doi: 10.1073/pnas.0704132104 pubmed: 17804795
Biswas, R. et al. Crystal structure of dehydratase component HadAB complex of mycobacterial FAS-II pathway. Biochem. Biophys. Res. Commun. 458, 369–374, https://doi.org/10.1016/j.bbrc.2015.01.119 (2015).
doi: 10.1016/j.bbrc.2015.01.119 pubmed: 25656575
Dong, Y. et al. Molecular basis for the inhibition of beta-hydroxyacyl-ACP dehydratase HadAB complex from Mycobacterium tuberculosis by flavonoid inhibitors. Protein Cell 6, 504–517, https://doi.org/10.1007/s13238-015-0181-1 (2015).
doi: 10.1007/s13238-015-0181-1 pubmed: 26081470 pmcid: 4491049
Lefebvre, C. et al. HadD, a novel fatty acid synthase type II protein, is essential for alpha- and epoxy-mycolic acid biosynthesis and mycobacterial fitness. Sci. Rep 8, 6034, https://doi.org/10.1038/s41598-018-24380-5 (2018).
doi: 10.1038/s41598-018-24380-5 pubmed: 29662082 pmcid: 5902629
Kapopoulou, A., Lew, J. M. & Cole, S. T. The MycoBrowser portal: a comprehensive and manually annotated resource for mycobacterial genomes. Tuberculosis 91, 8–13, https://doi.org/10.1016/j.tube.2010.09.006 (2011).
doi: 10.1016/j.tube.2010.09.006 pubmed: 20980200
Barkan, D., Rao, V., Sukenick, G. D. & Glickman, M. S. Redundant function of cmaA2 and mmaA2 in Mycobacterium tuberculosis cis cyclopropanation of oxygenated mycolates. J. Bacteriol. 192, 3661–3668, https://doi.org/10.1128/JB.00312-10 (2010).
doi: 10.1128/JB.00312-10 pubmed: 20472794 pmcid: 2897352
Parish, T. & Stoker, N. G. Use of a flexible cassette method to generate a double unmarked Mycobacterium tuberculosis tlyA plcABC mutant by gene replacement. Microbiology 146(Pt 8), 1969–1975, https://doi.org/10.1099/00221287-146-8-1969 (2000).
doi: 10.1099/00221287-146-8-1969 pubmed: 10931901
Sassetti, C. M., Boyd, D. H. & Rubin, E. J. Genes required for mycobacterial growth defined by high density mutagenesis. Mol. Microbiol 48, 77–84 (2003).
doi: 10.1046/j.1365-2958.2003.03425.x
Griffin, J. E. et al. High-resolution phenotypic profiling defines genes essential for mycobacterial growth and cholesterol catabolism. PLoS Pathog. 7, e1002251, https://doi.org/10.1371/journal.ppat.1002251 (2011).
doi: 10.1371/journal.ppat.1002251 pubmed: 21980284 pmcid: 3182942
Pym, A. S., Brodin, P., Brosch, R., Huerre, M. & Cole, S. T. Loss of RD1 contributed to the attenuation of the live tuberculosis vaccines Mycobacterium bovis BCG and Mycobacterium microti. Mol. Microbiol 46, 709–717 (2002).
doi: 10.1046/j.1365-2958.2002.03237.x
Bottai, D. et al. ESAT-6 secretion-independent impact of ESX-1 genes espF and espG1 on virulence of Mycobacterium tuberculosis. J. Infect. Dis 203, 1155–1164, https://doi.org/10.1093/infdis/jiq089 (2011).
doi: 10.1093/infdis/jiq089 pubmed: 21196469
Hsu, T. et al. The primary mechanism of attenuation of bacillus Calmette-Guerin is a loss of secreted lytic function required for invasion of lung interstitial tissue. Proc. Natl. Acad. Sci. USA 100, 12420–12425, https://doi.org/10.1073/pnas.1635213100 (2003).
doi: 10.1073/pnas.1635213100 pubmed: 14557547
Frigui, W. et al. Control of M. tuberculosis ESAT-6 secretion and specific T cell recognition by PhoP. PLoS Pathog. 4, e33, https://doi.org/10.1371/journal.ppat.0040033 (2008).
doi: 10.1371/journal.ppat.0040033 pubmed: 18282096 pmcid: 2242835
Glickman, M. S., Cahill, S. M. & Jacobs, W. R. Jr. The Mycobacterium tuberculosis cmaA2 gene encodes a mycolic acid trans-cyclopropane synthetase. J. Biol. Chem. 276, 2228–2233, https://doi.org/10.1074/jbc.C000652200 (2001).
doi: 10.1074/jbc.C000652200 pubmed: 11092877
Slama, N. et al. The changes in mycolic acid structures caused by hadC mutation have a dramatic effect on the virulence of Mycobacterium tuberculosis. Mol. Microbiol. 99, 794–807, https://doi.org/10.1111/mmi.13266 (2016).
doi: 10.1111/mmi.13266 pubmed: 26538472
Dubnau, E. et al. Mycobacterium bovis BCG genes involved in the biosynthesis of cyclopropyl keto- and hydroxy-mycolic acids. Mol. Microbiol. 23, 313–322 (1997).
doi: 10.1046/j.1365-2958.1997.2301589.x
Das, M. et al. Expression pattern similarities support the prediction of orthologs retaining common functions after gene duplication events. Plant Physiol 171, 2343–2357, https://doi.org/10.1104/pp.15.01207 (2016).
doi: 10.1104/pp.15.01207 pubmed: 27303025 pmcid: 4972257
Liu, J., Barry, C. E. III, Besra, G. S. & Nikaido, H. Mycolic acid structure determines the fluidity of the mycobacterial cell wall. J. Biol. Chem. 271, 29545–29551 (1996).
doi: 10.1074/jbc.271.47.29545
Yuan, Y., Zhu, Y., Crane, D. D. & Barry, C. E. III The effect of oxygenated mycolic acid composition on cell wall function and macrophage growth in Mycobacterium tuberculosis. Mol. Microbiol. 29, 1449–1458 (1998).
doi: 10.1046/j.1365-2958.1998.01026.x
Ojha, A. K. et al. Growth of Mycobacterium tuberculosis biofilms containing free mycolic acids and harbouring drug-tolerant bacteria. Mol. Microbiol 69, 164–174, https://doi.org/10.1111/j.1365-2958.2008.06274.x (2008).
doi: 10.1111/j.1365-2958.2008.06274.x pubmed: 18466296 pmcid: 2615189
Sambandan, D. et al. Keto-mycolic acid-dependent pellicle formation confers tolerance to drug-sensitive Mycobacterium tuberculosis. Mbio. 4, e00222–00213, https://doi.org/10.1128/mBio.00222-13 (2013).
doi: 10.1128/mBio.00222-13 pubmed: 23653446 pmcid: 3663190
Dubnau, E. et al. Oxygenated mycolic acids are necessary for virulence of Mycobacterium tuberculosis in mice. Mol. Microbiol 36, 630–637 (2000).
doi: 10.1046/j.1365-2958.2000.01882.x
Dao, D. N. et al. Mycolic acid modification by the mmaA4 gene of M. tuberculosis modulates IL-12 production. PLoS Pathog. 4, e1000081, https://doi.org/10.1371/journal.ppat.1000081 (2008).
doi: 10.1371/journal.ppat.1000081 pubmed: 18535659 pmcid: 2390761
Rachman, H. et al. Unique transcriptome signature of Mycobacterium tuberculosis in pulmonary tuberculosis. Infect. Immun. 74, 1233–1242, https://doi.org/10.1128/IAI.74.2.1233-1242.2006 (2006).
doi: 10.1128/IAI.74.2.1233-1242.2006 pubmed: 16428773 pmcid: 1360294
Liu, Y. et al. Delamanid: From discovery to its use for pulmonary multidrug-resistant tuberculosis (MDR-TB). Tuberculosis 111, 20–30, https://doi.org/10.1016/j.tube.2018.04.008 (2018).
doi: 10.1016/j.tube.2018.04.008 pubmed: 30029909
Sievers, F. et al. Fast, scalable generation of high-quality protein multiple sequence alignments using Clustal Omega. Mol. Syst. Biol. 7, 539, https://doi.org/10.1038/msb.2011.75 (2011).
doi: 10.1038/msb.2011.75 pubmed: 21988835 pmcid: 3261699
Altschul, S. F. et al. Gapped BLAST and PSI-BLAST: a new generation of protein database search programs. Nucleic. Acids. Res 25, 3389–3402 (1997).
doi: 10.1093/nar/25.17.3389
Mahenthiralingam, E. et al. Site-directed mutagenesis of the 19-kilodalton lipoprotein antigen reveals no essential role for the protein in the growth and virulence of Mycobacterium intracellulare. Infect. Immun 66, 3626–3634 (1998).
doi: 10.1128/IAI.66.8.3626-3634.1998
Vaubourgeix, J. et al. S-adenosyl-N-decyl-aminoethyl, a potent bisubstrate inhibitor of Mycobacterium tuberculosis mycolic acid methyltransferases. J. Biol. Chem. 284, 19321–19330, https://doi.org/10.1074/jbc.M809599200 (2009).
doi: 10.1074/jbc.M809599200 pubmed: 19439410 pmcid: 2740557

Auteurs

Cyril Lefebvre (C)

Département Tuberculose & Biologie des Infections, Institut de Pharmacologie et de Biologie Structurale, UMR5089, Université de Toulouse, CNRS, UPS, 31077, Toulouse, Cedex 04, France.

Wafa Frigui (W)

Institut Pasteur, Unit for Integrated Mycobacterial Pathogenomics, CNRS UMR3525, Paris, France.

Nawel Slama (N)

Département Tuberculose & Biologie des Infections, Institut de Pharmacologie et de Biologie Structurale, UMR5089, Université de Toulouse, CNRS, UPS, 31077, Toulouse, Cedex 04, France.
Toulouse White Biotechnology (UMS INRA / INSA / CNRS), Ramonville Saint-Agne, France.

Françoise Lauzeral-Vizcaino (F)

Département Tuberculose & Biologie des Infections, Institut de Pharmacologie et de Biologie Structurale, UMR5089, Université de Toulouse, CNRS, UPS, 31077, Toulouse, Cedex 04, France.
Centre de Recherches en Cancérologie de Toulouse, UMR1037 Inserm/UPS, Toulouse, France.

Patricia Constant (P)

Département Tuberculose & Biologie des Infections, Institut de Pharmacologie et de Biologie Structurale, UMR5089, Université de Toulouse, CNRS, UPS, 31077, Toulouse, Cedex 04, France.

Anne Lemassu (A)

Département Tuberculose & Biologie des Infections, Institut de Pharmacologie et de Biologie Structurale, UMR5089, Université de Toulouse, CNRS, UPS, 31077, Toulouse, Cedex 04, France.

Tanya Parish (T)

TB Discovery Research, Infectious Disease Research Institute, Seattle, WA, USA.
Center for Global Infectious Disease Research, Seattle Children's Research Institute, Seattle, WA, USA.

Nathalie Eynard (N)

Département Tuberculose & Biologie des Infections, Institut de Pharmacologie et de Biologie Structurale, UMR5089, Université de Toulouse, CNRS, UPS, 31077, Toulouse, Cedex 04, France.

Mamadou Daffé (M)

Département Tuberculose & Biologie des Infections, Institut de Pharmacologie et de Biologie Structurale, UMR5089, Université de Toulouse, CNRS, UPS, 31077, Toulouse, Cedex 04, France.

Roland Brosch (R)

Institut Pasteur, Unit for Integrated Mycobacterial Pathogenomics, CNRS UMR3525, Paris, France.

Annaïk Quémard (A)

Département Tuberculose & Biologie des Infections, Institut de Pharmacologie et de Biologie Structurale, UMR5089, Université de Toulouse, CNRS, UPS, 31077, Toulouse, Cedex 04, France. annaik.quemard@ipbs.fr.

Articles similaires

Robotic Surgical Procedures Animals Humans Telemedicine Models, Animal

Odour generalisation and detection dog training.

Lyn Caldicott, Thomas W Pike, Helen E Zulch et al.
1.00
Animals Odorants Dogs Generalization, Psychological Smell
Photosynthesis Ribulose-Bisphosphate Carboxylase Carbon Dioxide Molecular Dynamics Simulation Cyanobacteria
Animals TOR Serine-Threonine Kinases Colorectal Neoplasms Colitis Mice

Classifications MeSH