Cxcr4 distinguishes HSC-derived monocytes from microglia and reveals monocyte immune responses to experimental stroke.


Journal

Nature neuroscience
ISSN: 1546-1726
Titre abrégé: Nat Neurosci
Pays: United States
ID NLM: 9809671

Informations de publication

Date de publication:
03 2020
Historique:
received: 24 09 2018
accepted: 02 01 2020
pubmed: 12 2 2020
medline: 14 4 2020
entrez: 12 2 2020
Statut: ppublish

Résumé

Monocyte-derived and tissue-resident macrophages are ontogenetically distinct components of the innate immune system. Assessment of their respective functions in pathology is complicated by changes to the macrophage phenotype during inflammation. Here we find that Cxcr4-CreER enables permanent genetic labeling of hematopoietic stem cells (HSCs) and distinguishes HSC-derived monocytes from microglia and other tissue-resident macrophages. By combining Cxcr4-CreER-mediated lineage tracing with Cxcr4 inhibition or conditional Cxcr4 ablation in photothrombotic stroke, we find that Cxcr4 promotes initial monocyte infiltration and subsequent territorial restriction of monocyte-derived macrophages to infarct tissue. After transient focal ischemia, Cxcr4 deficiency reduces monocyte infiltration and blunts the expression of pattern recognition and defense response genes in monocyte-derived macrophages. This is associated with an altered microglial response and deteriorated outcomes. Thus, Cxcr4 is essential for an innate-immune-system-mediated defense response after cerebral ischemia. We further propose Cxcr4-CreER as a universal tool to study functions of HSC-derived cells.

Identifiants

pubmed: 32042176
doi: 10.1038/s41593-020-0585-y
pii: 10.1038/s41593-020-0585-y
pmc: PMC7523735
mid: NIHMS1622479
doi:

Substances chimiques

CXCR4 protein, mouse 0
Receptors, CXCR4 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

351-362

Subventions

Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NIAID NIH HHS
ID : R01 AI130345
Pays : United States

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Auteurs

Yves Werner (Y)

Institute of Pharmacology and Toxicology, Jena University Hospital, Jena, Germany.

Elvira Mass (E)

Immunology Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, NY, USA. emass@uni-bonn.de.
Developmental Biology of the Immune System, Life and Medical Sciences Institute, University of Bonn, Bonn, Germany. emass@uni-bonn.de.

Praveen Ashok Kumar (P)

Institute of Pharmacology and Toxicology, Jena University Hospital, Jena, Germany.

Thomas Ulas (T)

Genomics and Immunoregulation, Life and Medical Sciences Institute, University of Bonn, Bonn, Germany.
PRECISE Platform for Single Cell Genomics and Epigenomics, German Center for Neurodegenerative Diseases and University of Bonn, Bonn, Germany.

Kristian Händler (K)

Genomics and Immunoregulation, Life and Medical Sciences Institute, University of Bonn, Bonn, Germany.
PRECISE Platform for Single Cell Genomics and Epigenomics, German Center for Neurodegenerative Diseases and University of Bonn, Bonn, Germany.

Arik Horne (A)

Genomics and Immunoregulation, Life and Medical Sciences Institute, University of Bonn, Bonn, Germany.

Kathrin Klee (K)

Genomics and Immunoregulation, Life and Medical Sciences Institute, University of Bonn, Bonn, Germany.

Amelie Lupp (A)

Institute of Pharmacology and Toxicology, Jena University Hospital, Jena, Germany.

Dagmar Schütz (D)

Institute of Pharmacology and Toxicology, Jena University Hospital, Jena, Germany.

Friederike Saaber (F)

Institute of Pharmacology and Toxicology, Jena University Hospital, Jena, Germany.

Christoph Redecker (C)

Lippe General Hospital, Department of Neurology, Lemgo, Germany.

Joachim L Schultze (JL)

Genomics and Immunoregulation, Life and Medical Sciences Institute, University of Bonn, Bonn, Germany.
PRECISE Platform for Single Cell Genomics and Epigenomics, German Center for Neurodegenerative Diseases and University of Bonn, Bonn, Germany.

Frederic Geissmann (F)

Immunology Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, NY, USA. geissmaf@mskcc.org.

Ralf Stumm (R)

Institute of Pharmacology and Toxicology, Jena University Hospital, Jena, Germany. ralf.stumm@med.uni-jena.de.

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