Recovery training or over-training? The contribution of TLR10 to monocyte fitness.


Journal

Immunology
ISSN: 1365-2567
Titre abrégé: Immunology
Pays: England
ID NLM: 0374672

Informations de publication

Date de publication:
03 2020
Historique:
entrez: 12 2 2020
pubmed: 12 2 2020
medline: 30 4 2020
Statut: ppublish

Résumé

The concept of trained immunity refers to remodelling of the monocyte and macrophage metabolic and epigenetic landscape, conferring an amplified inflammatory response upon secondary stimulation. This effect is typically modelled in vitro by stimulating monocytes with either Bacillus Calmette Guerin (BCG) or β-Glucan for 24 hr, before subsequent stimulation with LPS or Pam-3-Cys (P3C) as a secondary stimulus 6 days later. Here, we focus on a recent paper which interrogated the role of the anti-inflammatory TLR, TLR10, on trained immunity. Using both an in vitro model of trained immunity, and analysis of BCG vaccinated individuals, the authors interestingly demonstrate that, despite its ability to regulate aspects of innate immunity, TLR10 does not have a significant role in this process.

Identifiants

pubmed: 32043595
doi: 10.1111/imm.13177
pmc: PMC7011651
doi:

Substances chimiques

TLR10 protein, human 0
Toll-Like Receptor 10 0

Types de publication

Editorial Comment

Langues

eng

Sous-ensembles de citation

IM

Pagination

243-244

Commentaires et corrections

Type : CommentOn

Informations de copyright

© 2020 John Wiley & Sons Ltd.

Références

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Auteurs

Lewis Rodgers (L)

Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, UK.

Simon Milling (S)

Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, UK.

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Classifications MeSH