The splice c.1815G>A variant in KIAA0586 results in a phenotype bridging short-rib-polydactyly and oral-facial-digital syndrome: A case report and literature review.


Journal

Medicine
ISSN: 1536-5964
Titre abrégé: Medicine (Baltimore)
Pays: United States
ID NLM: 2985248R

Informations de publication

Date de publication:
Feb 2020
Historique:
entrez: 22 2 2020
pubmed: 23 2 2020
medline: 10 3 2020
Statut: ppublish

Résumé

KIAA0586 variants have been associated to short-rib thoracic dysplasia, an autosomal recessive skeletal ciliopathy characterized by a narrow thorax, short limbs, and radiological skeletal abnormalities. Patients 1 and 2 were two Roma Gypsy siblings presenting thoracic dysplasia and a combination of oral cavity anomalies. A custom NGS gene panel, including genes associated to skeletal ciliopathies, identified the homozygous KIAA0586 splicing variant c.1815G>A (p.Gln605Gln) in both siblings, confirming the clinical diagnosis of short-rib-polydactyly. Patients were transferred to neonatal intensive care unit and received life-support treatment. Patients 1 and 2 died after few hours and 1 month of birth, respectively, because of respiratory failure related with the disease. We report two patients affected by short-rib polydactyly syndrome and overlapping phenotype with oral-facial-digital syndrome associated with the c.1815G>A variant in KIAA0586, suggesting a quite peculiar genotype-phenotype correlation.

Identifiants

pubmed: 32080096
doi: 10.1097/MD.0000000000019169
pii: 00005792-202002210-00026
pmc: PMC7034684
doi:

Substances chimiques

Cell Cycle Proteins 0
KIAA0586 protein, human 0

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e19169

Références

Ojeda Naharros I, Cristian FB, Zang J, et al. The ciliopathy protein TALPID3/KIAA0586 acts upstream of Rab8 activation in zebrafish photoreceptor outer segment formation and maintenance. Sci Rep 2018;8:2211.
Bachmann-Gagescu R, Phelps I, Dempsey J, et al. KIAA0586 is mutated in Joubert Syndrome. Human Mutat 2015;36:831–5.
Alby C, Piquand K, Huber C, et al. Mutations in KIAA0586 cause lethal ciliopathies ranging from a hydrolethalus phenotype to short-rib polydactyly syndrome. Am J Hum Genet 2015;97:311–8.
Richards S, Aziz N, Bale S, et al. Standards and guidelines for the interpretation of sequence variants: a joint consensus recommendation of the American College of Medical Genetics and Genomics and the Association for Molecular Pathology. Genet Med 2015;17:405–24.
Yin Y, Bangs F, Paton IR, et al. The Talpid3 gene (KIAA0586) encodes a centrosomal protein that is essential for primary cilia formation. Development 2009;136:655–64.
Bangs F, Antonio N, Thongnuek P, et al. Generation of mice with functional inactivation of talpid3, a gene first identified in chicken. Development 2011;138:3261–72.
Ben J, Elworthy S, Ng AS, et al. Targeted mutation of the talpid3 gene in zebrafish reveals its conserved requirement for ciliogenesis and Hedgehog signalling across the vertebrates. Development 2011;138:4969–78.
Bachmann-Gagescu R, Dempsey JC, Phelps IG, et al. Joubert syndrome: a model for untangling recessive disorders with extreme genetic heterogeneity. J Med Genet 2015;52:514–22.
Huber C, Cormier-Daire V. Ciliary disorder of the skeleton. Am J Med Genet C Semin Med Genet 2012;160C:165–74.
Schmidts M, Vodopiutz J, Christou-Savina S. Mutations in the gene encoding IFT dynein complex component WDR34 cause Jeune asphyxiating thoracic dystrophy. Am J Hum Genet 2013;93:932–44.
Stephen LA, Tawamie H, Davis GM, et al. TALPID3 controls centrosome and cell polarity and the human ortholog KIAA0586 is mutated in Joubert syndrome (JBTS23). Elife 2015;4:e08077.
Braun DA, Hildebrandt F. Ciliopathies. Cold Spring Harbor perspectives in biology 2017;9:a028191.
Maria BL, Quisling RG, Rosainz LC, et al. Molar tooth sign in Joubert syndrome: clinical, radiologic, and pathologic significance. J Child Neurol 1999;14:368–76.
Brancati F, Dallapiccola B, Valente EM. Joubert Syndrome and related disorders. Orphanet J Rare Dis 2010;8:20.
Malicdan MC, Vilboux T, Stephen J, et al. Mutations in human homologue of chicken talpid3 gene (KIAA0586) cause a hybrid ciliopathy with overlapping features of Jeune and Joubert syndromes. J Med Genet 2015;52:830–9.
Roosing S, Hofree M, Kim S, et al. Functional genome-wide siRNA screen identifies KIAA0586 as mutated in Joubert syndrome. Elife 2015;4: doi: 10.7554/eLife.06602.
doi: 10.7554/eLife.06602
Vilboux T, Doherty DA, Glass IA, et al. Molecular genetic findings and clinical correlations in 100 patients with Joubert syndrome and related disorders prospectively evaluated at a single center. Genet Med 2017;19:875–82.

Auteurs

Dario Cocciadiferro (D)

Laboratory of Medical Genetics.

Emanuele Agolini (E)

Laboratory of Medical Genetics.

Maria Cristina Digilio (MC)

Medical Genetics, Department of Pediatrics, Ospedale Pediatrico Bambino Gesù, Rome.

Lorenzo Sinibaldi (L)

Medical Genetics, Department of Pediatrics, Ospedale Pediatrico Bambino Gesù, Rome.

Marco Castori (M)

Division of Medical Genetics, Fondazione IRCCS-Casa Sollievo della Sofferenza, San Giovanni Rotondo (Foggia).

Evelina Silvestri (E)

Division of Pathology, Unit of Fetal and Neonatal Pathology, San Camillo-Forlanini Hospital.

Andrea Dotta (A)

Department of Medical and Surgical Neonatology, Bambino Gesù Children's Hospital.

Bruno Dallapiccola (B)

Department of Medical Genetics, Bambino Gesù Children's Hospital, IRCCS, Rome, Italy.

Antonio Novelli (A)

Laboratory of Medical Genetics.

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