Risk factors for congenital heart disease: The Baby Hearts Study, a population-based case-control study.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2020
Historique:
received: 18 06 2019
accepted: 02 01 2020
entrez: 25 2 2020
pubmed: 25 2 2020
medline: 23 4 2020
Statut: epublish

Résumé

We investigated the role of maternal environmental factors in the aetiology of congenital heart disease (CHD). A population-based case-control study (242 CHD cases, 966 controls) was conducted using an iPad questionnaire for mother with linkage to maternity and first trimester prescription records. Risk of CHD was associated with low maternal education (OR adjusted for confounders 1.59; 95% confidence interval [CI], 1.02-2.49), pregestational diabetes (OR 4.04; 95% CI 1.00-16.28), self-reported maternal clotting disorders (adjOR 8.55, 95%CI 1.51-48.44), prescriptions for the anticlotting medication enoxaparin (adjOR 3.22, 95%CI 1.01-10.22) and self-reported vaginal infections (adjOR 1.69, 95%CI 1.01-2.80). There was no strong support for the hypothesis that periconceptional folic acid supplements have a protective effect, but there was a protective effect of frequent consumption of folate rich fruits (adjOR 0.64, 95%CI 0.47-0.89). Compared to the most common pre-pregnancy dietary pattern, CHD risk was associated with a poor diet low in fruit and vegetables (adjOR 1.56, 95%CI 1.05-2.34). Mothers of cases reported more pregnancy related stress (adjOR 1.69; 95% CI 1.22-2.34) and multiple stressors (adjOR 1.94, 95%CI 0.83-4.53). We found no supportive evidence for CHD risk being associated with obesity, smoking, depression or antidepressant use in this population. Our findings add to the previous evidence base to show potential for public health approaches to help prevent CHD in future by modifying environmental factors. Independent confirmation should be sought regarding elevated CHD risk associated with maternal blood clotting disorders and their treatment, since we are the first to report this.

Identifiants

pubmed: 32092068
doi: 10.1371/journal.pone.0227908
pii: PONE-D-19-17236
pmc: PMC7039413
doi:

Substances chimiques

Folic Acid 935E97BOY8

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0227908

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Helen Dolk (H)

Institute of Nursing and Health Research, Ulster University, Newtownabbey, Northern Ireland, United Kingdom.

Nichola McCullough (N)

Institute of Nursing and Health Research, Ulster University, Newtownabbey, Northern Ireland, United Kingdom.

Sinead Callaghan (S)

Department of Cardiology, The Royal Belfast Hospital for Sick Children, Belfast, Northern Ireland, United Kingdom.

Frank Casey (F)

Department of Cardiology, The Royal Belfast Hospital for Sick Children, Belfast, Northern Ireland, United Kingdom.

Brian Craig (B)

Department of Cardiology, The Royal Belfast Hospital for Sick Children, Belfast, Northern Ireland, United Kingdom.

Joanne Given (J)

Institute of Nursing and Health Research, Ulster University, Newtownabbey, Northern Ireland, United Kingdom.

Maria Loane (M)

Institute of Nursing and Health Research, Ulster University, Newtownabbey, Northern Ireland, United Kingdom.

Briege M Lagan (BM)

Institute of Nursing and Health Research, Ulster University, Newtownabbey, Northern Ireland, United Kingdom.

Brendan Bunting (B)

School of Psychology, Ulster University, Coleraine, Northern Ireland, United Kingdom.

Breidge Boyle (B)

Institute of Nursing and Health Research, Ulster University, Newtownabbey, Northern Ireland, United Kingdom.

Tabib Dabir (T)

Department of Genetic Medicine, Belfast City Hospital, Belfast, Northern Ireland, United Kingdom.

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