The anti-viral dynamin family member MxB participates in mitochondrial integrity.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
26 02 2020
Historique:
received: 26 10 2018
accepted: 23 01 2020
entrez: 28 2 2020
pubmed: 28 2 2020
medline: 1 5 2020
Statut: epublish

Résumé

The membrane deforming dynamin family members MxA and MxB are large GTPases that convey resistance to a variety of infectious viruses. During viral infection, Mx proteins are known to show markedly increased expression via an interferon-responsive promoter to associate with nuclear pores. In this study we report that MxB is an inner mitochondrial membrane GTPase that plays an important role in the morphology and function of this organelle. Expression of mutant MxB or siRNA knockdown of MxB leads to fragmented mitochondria with disrupted inner membranes that are unable to maintain a proton gradient, while expelling their nucleoid-based genome into the cytoplasm. These findings implicate a dynamin family member in mitochondrial-based changes frequently observed during an interferon-based, anti-viral response.

Identifiants

pubmed: 32102993
doi: 10.1038/s41467-020-14727-w
pii: 10.1038/s41467-020-14727-w
pmc: PMC7044337
doi:

Substances chimiques

DNA, Mitochondrial 0
MX1 protein, human 0
MX2 protein, human 0
Myxovirus Resistance Proteins 0
RNA, Small Interfering 0
Dynamins EC 3.6.5.5

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

1048

Subventions

Organisme : NIDDK NIH HHS
ID : P30 DK084567
Pays : United States
Organisme : NIAAA NIH HHS
ID : R01 AA020735
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK044650
Pays : United States

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Auteurs

Hong Cao (H)

Department of Biochemistry and Molecular Biology, Mayo Clinic, 200 1st Street SW, Rochester, MN, 55905, USA.
Center for Basic Research in Digestive Diseases, Division of Gastroenterology & Hepatology, Mayo Clinic, 200 1st Street SW, Rochester, MN, 55905, USA.

E W Krueger (EW)

Center for Basic Research in Digestive Diseases, Division of Gastroenterology & Hepatology, Mayo Clinic, 200 1st Street SW, Rochester, MN, 55905, USA.

Jing Chen (J)

Center for Basic Research in Digestive Diseases, Division of Gastroenterology & Hepatology, Mayo Clinic, 200 1st Street SW, Rochester, MN, 55905, USA.

Kristina Drizyte-Miller (K)

Biochemistry and Molecular Biology Program, Mayo Clinic Graduate School of Biomedical Sciences, Mayo Clinic, 200 1st Street SW, Rochester, MN, 55905, USA.

Mary E Schulz (ME)

Center for Basic Research in Digestive Diseases, Division of Gastroenterology & Hepatology, Mayo Clinic, 200 1st Street SW, Rochester, MN, 55905, USA.

Mark A McNiven (MA)

Department of Biochemistry and Molecular Biology, Mayo Clinic, 200 1st Street SW, Rochester, MN, 55905, USA. mcniven.mark@mayo.edu.
Center for Basic Research in Digestive Diseases, Division of Gastroenterology & Hepatology, Mayo Clinic, 200 1st Street SW, Rochester, MN, 55905, USA. mcniven.mark@mayo.edu.

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