Cardiovascular Mortality After Type 1 and Type 2 Myocardial Infarction in Young Adults.


Journal

Journal of the American College of Cardiology
ISSN: 1558-3597
Titre abrégé: J Am Coll Cardiol
Pays: United States
ID NLM: 8301365

Informations de publication

Date de publication:
10 03 2020
Historique:
received: 07 10 2019
revised: 15 12 2019
accepted: 17 12 2019
entrez: 7 3 2020
pubmed: 7 3 2020
medline: 15 12 2020
Statut: ppublish

Résumé

Type 2 myocardial infarction (MI) and myocardial injury are associated with increased short-term mortality. However, data regarding long-term mortality are lacking. This study compared long-term mortality among young adults with type 1 MI, type 2 MI, or myocardial injury. Adults age 50 years or younger who presented with troponin >99th percentile or the International Classification of Diseases code for MI over a 17-year period were identified. All cases were adjudicated as type 1 MI, type 2 MI, or myocardial injury based on the Fourth Universal Definition of MI. Cox proportional hazards models were constructed for survival free from all-cause and cardiovascular death. The cohort consisted of 3,829 patients (median age 44 years; 30% women); 55% had type 1 MI, 32% had type 2 MI, and 13% had myocardial injury. Over a median follow-up of 10.2 years, mortality was highest for myocardial injury (45.6%), followed by type 2 MI (34.2%) and type 1 MI (12%) (p < 0.001). In an adjusted model, type 2 MI was associated with higher all-cause (hazard ratio: 1.8; 95% confidence interval: 1.2 to 2.7; p = 0.004) and cardiovascular mortality (hazard ratio: 2.7; 95% confidence interval: 1.4 to 5.1; p = 0.003) compared with type 1 MI. Those with type 2 MI or myocardial injury were younger and had fewer cardiovascular risk factors but had more noncardiovascular comorbidities. They were significantly less likely to be prescribed cardiovascular medications at discharge. Young patients who experience a type 2 MI have higher long-term all-cause and cardiovascular mortality than those who experience type 1 MI, with nearly one-half of patients with myocardial injury and more than one-third of patients with type 2 MI dying within 10 years. These findings emphasize the need to provide more aggressive secondary prevention for patients who experience type 2 MI and myocardial injury.

Sections du résumé

BACKGROUND
Type 2 myocardial infarction (MI) and myocardial injury are associated with increased short-term mortality. However, data regarding long-term mortality are lacking.
OBJECTIVES
This study compared long-term mortality among young adults with type 1 MI, type 2 MI, or myocardial injury.
METHODS
Adults age 50 years or younger who presented with troponin >99th percentile or the International Classification of Diseases code for MI over a 17-year period were identified. All cases were adjudicated as type 1 MI, type 2 MI, or myocardial injury based on the Fourth Universal Definition of MI. Cox proportional hazards models were constructed for survival free from all-cause and cardiovascular death.
RESULTS
The cohort consisted of 3,829 patients (median age 44 years; 30% women); 55% had type 1 MI, 32% had type 2 MI, and 13% had myocardial injury. Over a median follow-up of 10.2 years, mortality was highest for myocardial injury (45.6%), followed by type 2 MI (34.2%) and type 1 MI (12%) (p < 0.001). In an adjusted model, type 2 MI was associated with higher all-cause (hazard ratio: 1.8; 95% confidence interval: 1.2 to 2.7; p = 0.004) and cardiovascular mortality (hazard ratio: 2.7; 95% confidence interval: 1.4 to 5.1; p = 0.003) compared with type 1 MI. Those with type 2 MI or myocardial injury were younger and had fewer cardiovascular risk factors but had more noncardiovascular comorbidities. They were significantly less likely to be prescribed cardiovascular medications at discharge.
CONCLUSIONS
Young patients who experience a type 2 MI have higher long-term all-cause and cardiovascular mortality than those who experience type 1 MI, with nearly one-half of patients with myocardial injury and more than one-third of patients with type 2 MI dying within 10 years. These findings emphasize the need to provide more aggressive secondary prevention for patients who experience type 2 MI and myocardial injury.

Identifiants

pubmed: 32138959
pii: S0735-1097(20)30106-6
doi: 10.1016/j.jacc.2019.12.052
pmc: PMC7382936
mid: NIHMS1607105
pii:
doi:

Types de publication

Comparative Study Journal Article Multicenter Study Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

1003-1013

Subventions

Organisme : NHLBI NIH HHS
ID : T32 HL094301
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR001863
Pays : United States

Commentaires et corrections

Type : CommentIn
Type : CommentIn
Type : CommentIn
Type : CommentIn
Type : CommentIn

Informations de copyright

Copyright © 2020 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

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Auteurs

Avinainder Singh (A)

Department of Medicine, Yale University School of Medicine, New Haven, Connecticut. Electronic address: https://twitter.com/AvinainderSingh.

Ankur Gupta (A)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Ersilia M DeFilippis (EM)

Department of Cardiology, Columbia University Medical Center, New York, New York.

Arman Qamar (A)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

David W Biery (DW)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Zaid Almarzooq (Z)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Bradley Collins (B)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Amber Fatima (A)

Department of Medicine, Tufts Medical Center, Boston, Massachusetts.

Candace Jackson (C)

Department of Medicine, Mayo Clinic, Rochester, Minnesota.

Patrycja Galazka (P)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Mattheus Ramsis (M)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Daniel C Pipilas (DC)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Sanjay Divakaran (S)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Mary Cawley (M)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Jon Hainer (J)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Josh Klein (J)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Petr Jarolim (P)

Department of Pathology and Lab Medicine, Brigham and Women's Hospital, Boston, Massachusetts.

Khurram Nasir (K)

Department of Medicine, Yale University School of Medicine, New Haven, Connecticut.

James L Januzzi (JL)

Cardiovascular Division, Massachusetts General Hospital, Boston, Massachusetts.

Marcelo F Di Carli (MF)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts.

Deepak L Bhatt (DL)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts. Electronic address: https://twitter.com/DLBhattMD.

Ron Blankstein (R)

Cardiovascular Division, Brigham and Women's Hospital, Boston, Massachusetts. Electronic address: rblankstein@bwh.harvard.edu.

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Classifications MeSH