Putting the Pieces Together: Completing the Mechanism of Action Jigsaw for Sipuleucel-T.


Journal

Journal of the National Cancer Institute
ISSN: 1460-2105
Titre abrégé: J Natl Cancer Inst
Pays: United States
ID NLM: 7503089

Informations de publication

Date de publication:
01 06 2020
Historique:
received: 04 10 2019
revised: 23 01 2020
accepted: 30 01 2020
pubmed: 8 3 2020
medline: 15 1 2021
entrez: 8 3 2020
Statut: ppublish

Résumé

Sipuleucel-T is an autologous cellular immunotherapy that induces an immune response targeted against prostatic acid phosphatase (PAP) to treat asymptomatic or minimally symptomatic metastatic castration-resistant prostate cancer. In the phase III IMPACT study, sipuleucel-T was associated with a statistically significantly increased overall survival (OS) (median = 4.1 months) vs placebo. Patients with baseline prostate-specific antigen levels in the lowest quartile (≤22.1 ng/mL) exhibited a 13-month improvement in OS with sipuleucel-T. Together, this led sipuleucel-T to be approved and recommended as first-line therapy in various guidelines for treatment of metastatic castration-resistant prostate cancer. This review discusses the varied findings about the mechanisms of action of sipuleucel-T, bringing them together to form a more coherent picture. These pieces include inducing a statistically significant increase in antigen-presenting cell activation; inducing a peripheral immune response specific to the target (PAP) and/or immunizing (PA2024) antigens; stimulating systemic cytotoxic T-lymphocyte activity; and mediating antigen spread (ie, increased antibody responses to secondary proteins in addition to PAP and PA2024). Each of these pieces individually correlates with OS. Sipuleucel-T also traffics T cells to the prostate and is associated with long-term immune memory such that a second course of treatment induces an anamnestic immune response. Prostate cancer does not have a strongly inflamed microenvironment, thus its response to immune checkpoint inhibitors is limited. Because sipuleucel-T is able to traffic T cells to the tumor, it may be an ideal combination partner with immunotherapies including immune checkpoint inhibitors or with radiation therapy.

Identifiants

pubmed: 32145020
pii: 5799082
doi: 10.1093/jnci/djaa021
pmc: PMC7301097
doi:

Substances chimiques

Cancer Vaccines 0
Tissue Extracts 0
sipuleucel-T 8Q622VDR18

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

562-573

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press.

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Auteurs

Ravi A Madan (RA)

Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA.

Emmanuel S Antonarakis (ES)

Johns Hopkins Sidney Kimmel Comprehensive Cancer Center, Baltimore, MD, USA.

Charles G Drake (CG)

Columbia University Herbert Irving Comprehensive Cancer Center, New York, NY, USA.

Lawrence Fong (L)

Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, CA, USA.

Evan Y Yu (EY)

University of Washington and Seattle Cancer Care Alliance, Seattle, WA, USA.

Douglas G McNeel (DG)

University of Wisconsin Carbone Cancer Center, Madison, WI, USA.

Daniel W Lin (DW)

University of Washington and Seattle Cancer Care Alliance, Seattle, WA, USA.

Nancy N Chang (NN)

Dendreon Pharmaceuticals LLC, Seattle, WA, USA.

Nadeem A Sheikh (NA)

Dendreon Pharmaceuticals LLC, Seattle, WA, USA.

James L Gulley (JL)

Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA.

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Classifications MeSH