Association of Interleukin-1 and Inteleukin-1 Receptor Antagonist Gene Polymorphisms with Multiple Sclerosis in Azeri Population of Iran.


Journal

Endocrine, metabolic & immune disorders drug targets
ISSN: 2212-3873
Titre abrégé: Endocr Metab Immune Disord Drug Targets
Pays: United Arab Emirates
ID NLM: 101269157

Informations de publication

Date de publication:
2020
Historique:
received: 14 10 2019
revised: 14 01 2020
accepted: 14 01 2020
pubmed: 10 3 2020
medline: 16 6 2021
entrez: 10 3 2020
Statut: ppublish

Résumé

Cytokines as important mediators have a critical role in appropriate immune responses, the irregular production of which can lead to Multiple Sclerosis (MS). Proinflammatory cytokine interleukin-1 (IL-1) triggers inflammatory responses. Function and production of the cytokine are influenced by IL-1 coding gene polymorphism and those antagonists gene polymorphism. The aim of the present study was to evaluate the possible correlation between MS and IL-1 related alleles in Azeri population of Iran. Variable number tandem repeats (VNTR) genotypes of 150 MS patients and 220 healthy non-relative controls were determined. In the healthy controls, genotype TT at IL-1A (-889) location was significantly higher than the MS patients (p=0.0001). However, a significant difference was not found between the two groups in genotypic/allelic frequency at IL- 1B+ 3953 location. Evaluation of the IL-1RA gene revealed that genotype 1/2, and genotype 1/3 were significantly higher in the healthy controls and MS patients, respectively. Our findings indicated that the consumption of fast-food in MS patients was significantly higher than controls (p= <0.05). Also, a considerable number of MS patients had inappropriate dieting behaviors such as not eating breakfast (p= 0.0001), and irregular eating habits (p= 0.0001). Polymorphisms of the IL-1B genes and common alleles of IL-1RA were not considered as risk factors for MS disease. However, genotype TT at IL-1A (-889) location and the rare allele of IL-1RA3 can be a potential risk factor for the disease. Furthermore, inappropriate dieting behaviors and consumption of fast-food can increase the risk of MS.

Sections du résumé

BACKGROUND BACKGROUND
Cytokines as important mediators have a critical role in appropriate immune responses, the irregular production of which can lead to Multiple Sclerosis (MS). Proinflammatory cytokine interleukin-1 (IL-1) triggers inflammatory responses. Function and production of the cytokine are influenced by IL-1 coding gene polymorphism and those antagonists gene polymorphism.
OBJECTIVE OBJECTIVE
The aim of the present study was to evaluate the possible correlation between MS and IL-1 related alleles in Azeri population of Iran.
METHODS METHODS
Variable number tandem repeats (VNTR) genotypes of 150 MS patients and 220 healthy non-relative controls were determined.
RESULTS RESULTS
In the healthy controls, genotype TT at IL-1A (-889) location was significantly higher than the MS patients (p=0.0001). However, a significant difference was not found between the two groups in genotypic/allelic frequency at IL- 1B+ 3953 location. Evaluation of the IL-1RA gene revealed that genotype 1/2, and genotype 1/3 were significantly higher in the healthy controls and MS patients, respectively. Our findings indicated that the consumption of fast-food in MS patients was significantly higher than controls (p= <0.05). Also, a considerable number of MS patients had inappropriate dieting behaviors such as not eating breakfast (p= 0.0001), and irregular eating habits (p= 0.0001).
CONCLUSION CONCLUSIONS
Polymorphisms of the IL-1B genes and common alleles of IL-1RA were not considered as risk factors for MS disease. However, genotype TT at IL-1A (-889) location and the rare allele of IL-1RA3 can be a potential risk factor for the disease. Furthermore, inappropriate dieting behaviors and consumption of fast-food can increase the risk of MS.

Identifiants

pubmed: 32148206
pii: EMIDDT-EPUB-105091
doi: 10.2174/1871530320666200309142541
doi:

Substances chimiques

IL1RN protein, human 0
Interleukin 1 Receptor Antagonist Protein 0
Interleukin-1 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1110-1116

Informations de copyright

Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.

Auteurs

Mohammad Asgharzadeh (M)

Biotechnology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.

Zahra A Jigheh (ZA)

3Department of Clinical Biochemistry, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.

Hossein S Kafil (HS)

Drug Applied Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.

Mehdi Farhoudi (M)

Neurosciences Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.

Daryoush S Oskouei (DS)

Neurosciences Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.

Fatemeh Khaki-Khatibi (F)

3Department of Clinical Biochemistry, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.

Manouchehr Fadaee (M)

Department of Immunology, Shiraz University of Medical Sciences, Shiraz, Iran.

Ali Vegari (A)

Department of Medical Physics, Faculty of Medicine, Urmia University of Medical Sciences, Urmia, Iran.

Vahid Asgharzadeh (V)

8Hematology and Oncology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.

Nima Najafi-Ghalelou (N)

Department of Laboratory Sciences, Faculty of Paramedicine, Tabriz University of Medical Sciences, Tabriz, Iran.

Behroz M Poor (BM)

Department of Laboratory Sciences, Faculty of Paramedicine, Tabriz University of Medical Sciences, Tabriz, Iran.

Jalil Rashedi (J)

Department of Laboratory Sciences, Faculty of Paramedicine, Tabriz University of Medical Sciences, Tabriz, Iran.

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Classifications MeSH