Cancer stem cell enrichment is associated with enhancement of nicotinamide N-methyltransferase expression.


Journal

IUBMB life
ISSN: 1521-6551
Titre abrégé: IUBMB Life
Pays: England
ID NLM: 100888706

Informations de publication

Date de publication:
07 2020
Historique:
received: 16 01 2020
revised: 17 02 2020
accepted: 19 02 2020
pubmed: 10 3 2020
medline: 16 11 2021
entrez: 10 3 2020
Statut: ppublish

Résumé

The cancer stem cell theory states that a subset of tumor cells, termed cancer stem cells (CSCs), has the ability to self-renew and differentiate within the tumors. According to this theory, CSCs would be mainly responsible for tumor initiation, progression, resistance to therapy, recurrence, and metastasis. In this study, a culture system was setup to enrich CSCs from bladder cancer (T24), lung cancer (A549), colorectal cancer (CaCo-2), and osteosarcoma (MG63) cell lines, through sphere formation. Magnetic-activated cell sorting was also used to further increase CSC enrichment. Subsequently, molecular characterization of CSC-enriched cell populations and parental cells was carried out, by exploring the expression levels of stem markers and the enzyme nicotinamide N-methyltransferase (NNMT). Results obtained showed a significant upregulation of stem cell markers in CSC-enriched populations, obtained upon sphere formation, compared with parental counterparts. Moreover, NNMT expression levels were markedly increased in samples enriched with CSCs with respect to control cells. Considering the fundamental role played by CSCs in carcinogenesis, reported data strengthen the hypothesis that sustains a pivotal role of NNMT in cancer growth and metastasis. In addition, these findings could represent an important achievement for the development of new and effective anticancer therapies, based on CSC-associated targets.

Identifiants

pubmed: 32150326
doi: 10.1002/iub.2265
doi:

Substances chimiques

NNMT protein, human EC 2.1.1.1
Nicotinamide N-Methyltransferase EC 2.1.1.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1415-1425

Informations de copyright

© 2020 International Union of Biochemistry and Molecular Biology.

Références

Clarke MF, Fuller M. Stem cells and cancer: Two faces of eve. Cell. 2006;124:1111-1115. http://doi.org/10.1016/j.cell.2006.03.011.
Morrison SJ, Kimble J. Asymmetric and symmetric stem-cell divisions in development and cancer. Nature. 2006;441:1068-1074. http://doi.org/10.1038/nature04956.
Liu H, Lv L, Yang K. Chemotherapy targeting cancer stem cells. Am J Cancer Res. 2015;15:880-893.
Qiu H, Fang X, Luo Q, Ouyang G. Cancer stem cells: A potential target for cancer therapy. Cell Mol Life Sci. 2015;72:3411-3424. http://doi.org/10.1007/s00018-015-1920-4.
Dean M, Fojo T, Bates S. Tumour stem cells and drug resistance. Nat Rev Cancer. 2005;5:275-284. http://doi.org/10.1038/nrc1590.
Maccalli C, De Maria R. Cancer stem cells: Perspectives for therapeutic targeting. Cancer Immunol Immunother. 2015;64:91-97. http://doi.org/10.1007/s00262-014-1592-1.
Aksoy S, Szumlanski CL, Weinshilboum RM. Human liver nicotinamide N-methyltransferase. cDNA cloning, expression, and biochemical characterization. J Biol Chem. 1994;269:14835-14840.
Peng Y, Sartini D, Pozzi V, Wilk D, Emanuelli M, Yee VC. Structural basis of substrate recognition in human nicotinamide N-methyltransferase. Biochemistry. 2011;50:7800-7808. http://doi.org/10.1021/bi2007614.
Sartini D, Muzzonigro G, Milanese G, Pierella F, Rossi V, Emanuelli M. Identification of nicotinamide N-methyltransferase as a novel tumor marker for renal clear cell carcinoma. J Urol. 2006;176:2248-2254. http://doi.org/10.1016/j.juro.2006.07.046.
Campagna R, Cecati M, Pozzi V, et al. Involvement of transforming growth factor beta 1 in the transcriptional regulation of nicotinamide N-methyltransferase in clear cell renal cell carcinoma. Cell Mol Biol (Noisy-le-Grand). 2018;64:51-55.
Sartini D, Santarelli A, Rossi V, et al. Nicotinamide N-methyltransferase upregulation inversely correlates with lymph node metastasis in oral squamous cell carcinoma. Mol Med. 2007;13:415-421. http://doi.org/10.2119/2007-00035.Sartini.
Emanuelli M, Santarelli A, Sartini D, Ciavarella D, Rossi V. Nicotinamide N-methyltransferase upregulation correlates with tumour differentiation in oral squamous cell carcinoma. Histol Histopathol. 2010;25:15-20. http://doi.org/10.14670/HH-25.15.
Sartini D, Morganti S, Guidi E, et al. Nicotinamide N-methyltransferase in non-small cell lung cancer: Promising results for targeted anti-cancer therapy. Cell Biochem Biophys. 2013;67:865-873. http://doi.org/10.1007/s12013-013-9574-z.
Sartini D, Muzzonigro G, Milanese G, et al. Upregulation of tissue and urinary nicotinamide N-methyltransferase in bladder cancer: Potential for the development of a urine-based diagnostic test. Cell Biochem Biophys. 2013;65:473-483. http://doi.org/10.1007/s12013-012-9451-1.
Ganzetti G, Sartini D, Campanati A, et al. Nicotinamide N-methyltransferase: Potential involvement in cutaneous malignant melanoma. Melanoma Res. 2018;28:82-88. http://doi.org/10.1097/CMR.0000000000000430.
Mascitti M, Santarelli A, Sartini D, et al. Analysis of nicotinamide N-methyltransferase in oral malignant melanoma and potential prognostic significance. Melanoma Res. 2019;29:151-156. http://doi.org/10.1097/CMR.0000000000000548.
Sartini D, Pozzi V, Renzi E, et al. Analysis of tissue and salivary nicotinamide N-methyltransferase in oral squamous cell carcinoma: Basis for the development of a noninvasive diagnostic test for early-stage disease. Biol Chem. 2012;393:505-511. http://doi.org/10.1515/hsz-2012-0112.
Pozzi V, Di Ruscio G, Sartini D, Campagna R, Seta R, et al. Clinical performance and utility of a NNMT-based urine test for bladder cancer. Int J Biol Markers. 2018;33:94-101. http://doi.org/10.5301/ijbm.5000311.
Pozzi V, Mazzotta M, Lo Muzio L, Sartini D, Santarelli A, et al. Inhibiting proliferation in KB cancer cells by RNA interference-mediated knockdown of nicotinamide N-methyltransferase expression. Int J Immunopathol Pharmacol. 2011;24:69-77. http://doi.org/10.1177/039463201102400109.
Pozzi V, Sartini D, Morganti S, Giuliante R, Di Ruscio G, et al. RNA-mediated gene silencing of nicotinamide N-methyltransferase is associated with decreased tumorigenicity in human oral carcinoma cells. PLoS One. 2013;8:e71272. http://doi.org/10.1371/journal.pone.0071272.
Sartini D, Seta R, Pozzi V, et al. Role of nicotinamide N-methyltransferase in non-small cell lung cancer: in vitro effect of shRNA-mediated gene silencing on tumourigenicity. Biol Chem. 2015;396:225-234. http://doi.org/10.1515/hsz-2014-0231.
Seta R, Mascitti M, Campagna R, et al. Overexpression of nicotinamide N-methyltransferase in HSC-2 OSCC cell line: Effect on apoptosis and cell proliferation. Clin Oral Investig. 2019;23:829-838. http://doi.org/10.1007/s00784-018-2497-8.
Pozzi V, Sartini D, Rocchetti R, et al. Identification and characterization of cancer stem cells from head and neck squamous cell carcinoma cell lines. Cell Physiol Biochem. 2015;36:784-798. http://doi.org/10.1159/000430138.
Livak KJ, Schmittgen TD. Analysis of relative gene expression data using real-time quantitative PCR and the 2(−Delta Delta C[T]) method. Methods. 2001;25:402-408. http://doi.org/10.1006/meth.2001.1262.
Bradford MM. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. Anal Biochem. 1976;72:248-254.
Balducci E, Orsomando G, Polzonetti V, et al. NMN adenylyltransferase from bull testis: Purification and properties. Biochem J. 1995;310:395-400.
He JQ, Vu DM, Hunt G, Chugh A, Bhatnagar A, Bolli R. Human cardiac stem cells isolated from atrial appendages stably express c-kit. PLoS One. 2011;6:e27719. http://doi.org/10.1371/journal.pone.0027719.
Dragu DL, Necula LG, Bleotu C, Diaconu CC, Chivu-Economescu M. Therapies targeting cancer stem cells: Current trends and future challenges. World J Stem Cells. 2015;7:1185-1201. http://doi.org/10.4252/wjsc.v7.i9.1185.
Shiozawa Y, Nie B, Pienta KJ, Morgan TM, Taichman RS. Cancer stem cells and their role in metastasis. Pharmacol Ther. 2013;138:285-293. http://doi.org/10.1016/j.pharmthera.2013.01.014.
Islam F, Gopalan V, Smith RA, Lam AK. Translational potential of cancer stem cells: A review of the detection of cancer stem cells and their roles in cancer recurrence and cancer treatment. Exp Cell Res. 2015;335:135-147. http://doi.org/10.1016/j.yexcr.2015.04.018.
Khan IN, Al-Karim S, Bora RS, Chaudhary AG, Saini KS. Cancer stem cells: A challenging paradigm for designing targeted drug therapies. Drug Discov Today. 2015;20:1205-1216. http://doi.org/10.1016/j.drudis.2015.06.013.
Glumac PM, LeBeau AM. The role of CD133 in cancer: A concise review. Clin Transl Med. 2018;7:18. http://doi.org/10.1186/s40169-018-0198-1.
Saito Y, Kitamura H, Hijikata A, Tomizawa-Murasawa M, Tanaka S, et al. Identification of therapeutic targets for quiescent, chemotherapy-resistant human leukemia stem cells. Sci Transl Med. 2010;2:17ra9. https://doi.org/10.1126/scitranslmed.3000349.
Yoshida GJ, Saya H. Therapeutic strategies targeting cancer stem cells. Cancer Sci. 2016;107:5-11. http://doi.org/10.1111/cas.12817.
D'Andrea FP, Safwat A, Kassem M, Gautier L, Overgaard J, Horsman MR. Cancer stem cell overexpression of nicotinamide N-methyltransferase enhances cellular radiation resistance. Radiother Oncol. 2011;99:373-378. http://doi.org/10.1016/j.radonc.2011.05.086.
Okamoto H, Ishikawa A, Yoshitake Y, et al. Diurnal variations in human urinary excretion of nicotinamide catabolites: Effects of stress on the metabolism of nicotinamide. Am J Clin Nutr. 2003;77:406-410. http://doi.org/10.1093/ajcn/77.2.406.
Zhang J. Are poly(ADP-ribosyl)ation by PARP-1 and deacetylation by Sir2 linked? Bioessays. 2003;25:808-814. http://doi.org/10.1002/bies.10317.
Ulanovskaya OA, Zuhl AM, Cravatt BF. NNMT promotes epigenetic remodeling in cancer by creating a metabolic methylation sink. Nat Chem Biol. 2013;9:300-306. http://doi.org/10.1038/nchembio.1204.
Jung J, Kim LJ, Wang X, Wu Q, Sanvoranart T, et al. Nicotinamide metabolism regulates glioblastoma stem cell maintenance. JCI Insight. 2017;2:90019. http://doi.org/10.1172/jci.insight.90019.

Auteurs

Valentina Pozzi (V)

Department of Clinical Sciences, Polytechnic University of Marche, Ancona, Italy.
New York-Marche Structural Biology Center (NY-MaSBiC), Polytechnic University of Marche, Ancona, Italy.

Eleonora Salvolini (E)

Department of Clinical Sciences, Polytechnic University of Marche, Ancona, Italy.

Guendalina Lucarini (G)

Department of Clinical and Molecular Sciences, University of Marche, Ancona, Italy.

Alessia Salvucci (A)

Department of Clinical Sciences, Polytechnic University of Marche, Ancona, Italy.

Roberto Campagna (R)

Department of Clinical Sciences, Polytechnic University of Marche, Ancona, Italy.

Corrado Rubini (C)

Department of Biomedical Sciences and Public Health, Polytechnic University of Marche, Ancona, Italy.

Davide Sartini (D)

Department of Clinical Sciences, Polytechnic University of Marche, Ancona, Italy.

Monica Emanuelli (M)

Department of Clinical Sciences, Polytechnic University of Marche, Ancona, Italy.
New York-Marche Structural Biology Center (NY-MaSBiC), Polytechnic University of Marche, Ancona, Italy.

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