Heterogeneity of Mild to Moderate Persistent Asthma in Children: Confirmation by Latent Class Analysis and Association with 1-Year Outcomes.

Aeroallergen sensitization Asthma control Asthma exacerbation Asthma in children Asthma outcomes Latent class analysis Lung function Phenotype Type 2 inflammation

Journal

The journal of allergy and clinical immunology. In practice
ISSN: 2213-2201
Titre abrégé: J Allergy Clin Immunol Pract
Pays: United States
ID NLM: 101597220

Informations de publication

Date de publication:
09 2020
Historique:
received: 13 02 2020
accepted: 17 02 2020
pubmed: 12 3 2020
medline: 15 5 2021
entrez: 12 3 2020
Statut: ppublish

Résumé

Compared with adults, phenotypic characterization of children with asthma is still limited and it remains difficult to predict which children with asthma are at highest risk for poor outcomes. To identify latent classes in a large population of treatment-adherent children with mild to moderate asthma enrolled in clinical trials and determine whether latent class assignment predicts future lung function abnormalities and exacerbation rate. Latent class analysis was performed on 2593 children with mild to moderate asthma aged 5 18 years, with 19 variables encompassing demographic characteristics, medical history, symptoms, lung function, allergic sensitization, and type 2 inflammation. Outcomes included lung function and the annualized exacerbation rate at 12 months of follow-up. Five latent classes were identified with differing demographic features, asthma control, sensitization, type 2 inflammatory markers, and lung function. Exacerbation rates were 1.30 ± 0.12 for class 1 (multiple sensitization with partially reversible airflow limitation), 0.90 ± 0.05 for class 2 (multiple sensitization with reversible airflow limitation), 0.87 ± 0.08 for class 3 (lesser sensitization with reversible airflow limitation), 0.87 ± 0.05 for class 4 (multiple sensitization with normal lung function), and 0.71 ± 0.06 for class 5 (lesser sensitization with normal lung function). Lung function abnormalities persisted in class 1 at 12 months. Children with mild to moderate asthma are a heterogeneous group. Allergic sensitization and lung function may be particularly useful in identifying children at the greatest risk for future exacerbation. Additional studies are needed to determine whether latent classes correspond to meaningful phenotypes for the purpose of personalized treatment.

Sections du résumé

BACKGROUND
Compared with adults, phenotypic characterization of children with asthma is still limited and it remains difficult to predict which children with asthma are at highest risk for poor outcomes.
OBJECTIVE
To identify latent classes in a large population of treatment-adherent children with mild to moderate asthma enrolled in clinical trials and determine whether latent class assignment predicts future lung function abnormalities and exacerbation rate.
METHODS
Latent class analysis was performed on 2593 children with mild to moderate asthma aged 5 18 years, with 19 variables encompassing demographic characteristics, medical history, symptoms, lung function, allergic sensitization, and type 2 inflammation. Outcomes included lung function and the annualized exacerbation rate at 12 months of follow-up.
RESULTS
Five latent classes were identified with differing demographic features, asthma control, sensitization, type 2 inflammatory markers, and lung function. Exacerbation rates were 1.30 ± 0.12 for class 1 (multiple sensitization with partially reversible airflow limitation), 0.90 ± 0.05 for class 2 (multiple sensitization with reversible airflow limitation), 0.87 ± 0.08 for class 3 (lesser sensitization with reversible airflow limitation), 0.87 ± 0.05 for class 4 (multiple sensitization with normal lung function), and 0.71 ± 0.06 for class 5 (lesser sensitization with normal lung function). Lung function abnormalities persisted in class 1 at 12 months.
CONCLUSIONS
Children with mild to moderate asthma are a heterogeneous group. Allergic sensitization and lung function may be particularly useful in identifying children at the greatest risk for future exacerbation. Additional studies are needed to determine whether latent classes correspond to meaningful phenotypes for the purpose of personalized treatment.

Identifiants

pubmed: 32156610
pii: S2213-2198(20)30231-2
doi: 10.1016/j.jaip.2020.02.032
pmc: PMC7483393
mid: NIHMS1578474
pii:
doi:

Substances chimiques

Biomarkers 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2617-2627.e4

Subventions

Organisme : NIEHS NIH HHS
ID : P30 ES006694
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR001422
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR002378
Pays : United States

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

Copyright © 2020 American Academy of Allergy, Asthma & Immunology. Published by Elsevier Inc. All rights reserved.

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Auteurs

Anne M Fitzpatrick (AM)

Department of Pediatrics, Emory University and Children's Healthcare of Atlanta, Atlanta, Ga. Electronic address: anne.fitzpatrick@emory.edu.

Leonard B Bacharier (LB)

Department of Pediatrics, Washington University and St Louis Children's Hospital, St Louis, Mo.

Daniel J Jackson (DJ)

Departments of Pediatrics and Medicine, University of Wisconsin School of Medicine and Public Health, Madison, Wis.

Stanley J Szefler (SJ)

Children's Hospital Colorado and Department of Pediatrics, University of Colorado, Aurora, Colo.

Avraham Beigelman (A)

Department of Pediatrics, Washington University and St Louis Children's Hospital, St Louis, Mo.

Michael Cabana (M)

Department of Pediatrics, Albert Einstein College of Medicine and Montefiore Health System, Bronx, New York, NY.

Ronina Covar (R)

Department of Pediatrics, National Jewish Health, Denver, Colo.

Theresa Guilbert (T)

Department of Pediatrics, University of Cincinnati and Cincinnati Children's Hospital and Medical Center, Cincinnati, Ohio.

Fernando Holguin (F)

Department of Medicine, University of Colorado, Denver, Colo.

Robert F Lemanske (RF)

Departments of Pediatrics and Medicine, University of Wisconsin School of Medicine and Public Health, Madison, Wis.

Fernando D Martinez (FD)

Department of Pediatrics, The University of Arizona, Tucson, Ariz.

Wayne Morgan (W)

Department of Pediatrics, The University of Arizona, Tucson, Ariz.

Wanda Phipatanakul (W)

Division of Allergy and Immunology, Boston Children's Hospital and Harvard Medical School Department of Pediatrics, Boston, Mass.

Jacqueline A Pongracic (JA)

Ann and Robert H. Lurie Children's Hospital of Chicago, Chicago, Ill.

Hengameh H Raissy (HH)

Department of Pediatrics, University of New Mexico, Albuquerque, NM.

Robert S Zeiger (RS)

Kaiser Permanente, Southern California Region and Department of Pediatrics, University of California San Diego, San Diego, Calif.

David T Mauger (DT)

Department of Public Health Sciences, Penn State University, Hershey, Pa.

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