Vaccination With Moderate Coverage Eradicates Oncogenic Human Papillomaviruses If a Gender-Neutral Strategy Is Applied.


Journal

The Journal of infectious diseases
ISSN: 1537-6613
Titre abrégé: J Infect Dis
Pays: United States
ID NLM: 0413675

Informations de publication

Date de publication:
17 08 2020
Historique:
received: 01 11 2019
accepted: 03 03 2020
pubmed: 13 3 2020
medline: 9 3 2021
entrez: 13 3 2020
Statut: ppublish

Résumé

Human papillomavirus (HPV) vaccination of girls with very high (>90%) coverage has the potential to eradicate oncogenic HPVs, but such high coverage is hard to achieve. However, the herd effect (HE) depends both on the HPV type and the vaccination strategy. We randomized 33 Finnish communities into gender-neutral HPV16/18 vaccination, girls-only HPV16/18 vaccination, and hepatitis B virus vaccination arms. In 2007-2010, 11 662 of 20 513 of 40 852 of 39 420 resident boys/girls from 1992 to 1995 birth cohorts consented. In 2010-2014, cervicovaginal samples from vaccinated and unvaccinated girls at age 18.5 years were typed for HPV6/11/16/18/31/33/35/39/45/51/52/56/58/59/66/68. Vaccine efficacy for vaccinated girls, HE for unvaccinated girls, and the protective effectiveness (PE) for all girls were estimated. We extended the community-randomized trial results about vaccination strategy with mathematical modeling to assess HPV eradication. The HE and PE estimates in the 1995 birth cohort for HPV18/31/33 were significant in the gender-neutral arm and 150% and 40% stronger than in the girls-only arm. Concordantly, HPV18/31/33 eradication was already predicted in adolescents/young adults in 20 years with 75% coverage of gender-neutral vaccination. With the 75% coverage, eventual HPV16 eradication was also predicted, but only with the gender-neutral strategy. Gender-neutral vaccination is superior for eradication of oncogenic HPVs.

Sections du résumé

BACKGROUND
Human papillomavirus (HPV) vaccination of girls with very high (>90%) coverage has the potential to eradicate oncogenic HPVs, but such high coverage is hard to achieve. However, the herd effect (HE) depends both on the HPV type and the vaccination strategy.
METHODS
We randomized 33 Finnish communities into gender-neutral HPV16/18 vaccination, girls-only HPV16/18 vaccination, and hepatitis B virus vaccination arms. In 2007-2010, 11 662 of 20 513 of 40 852 of 39 420 resident boys/girls from 1992 to 1995 birth cohorts consented. In 2010-2014, cervicovaginal samples from vaccinated and unvaccinated girls at age 18.5 years were typed for HPV6/11/16/18/31/33/35/39/45/51/52/56/58/59/66/68. Vaccine efficacy for vaccinated girls, HE for unvaccinated girls, and the protective effectiveness (PE) for all girls were estimated. We extended the community-randomized trial results about vaccination strategy with mathematical modeling to assess HPV eradication.
RESULTS
The HE and PE estimates in the 1995 birth cohort for HPV18/31/33 were significant in the gender-neutral arm and 150% and 40% stronger than in the girls-only arm. Concordantly, HPV18/31/33 eradication was already predicted in adolescents/young adults in 20 years with 75% coverage of gender-neutral vaccination. With the 75% coverage, eventual HPV16 eradication was also predicted, but only with the gender-neutral strategy.
CONCLUSIONS
Gender-neutral vaccination is superior for eradication of oncogenic HPVs.

Identifiants

pubmed: 32161969
pii: 5803245
doi: 10.1093/infdis/jiaa099
pmc: PMC7430169
doi:

Substances chimiques

Papillomavirus Vaccines 0

Banques de données

ClinicalTrials.gov
['NCT00534638']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

948-956

Commentaires et corrections

Type : CommentIn

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press for the Infectious Diseases Society of America.

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Auteurs

Simopekka Vänskä (S)

Infectious Disease Control and Vaccinations, Finnish Institute for Health and Welfare, Helsinki, Finland.
Department of Laboratory Medicine, Karolinska Institute, Stockholm, Sweden.

Tapio Luostarinen (T)

Finnish Cancer Registry, Helsinki, Finland.

Iacopo Baussano (I)

International Agency for Research on Cancer, Lyon, France.

Dan Apter (D)

VL-Medi Oy, Helsinki, Finland.

Tiina Eriksson (T)

Tampere University, Tampere, Finland.

Kari Natunen (K)

Tampere University, Tampere, Finland.

Pekka Nieminen (P)

University of Helsinki, Helsinki, Finland.

Jorma Paavonen (J)

University of Helsinki, Helsinki, Finland.

Ville N Pimenoff (VN)

Department of Laboratory Medicine, Karolinska Institute, Stockholm, Sweden.
Tampere University, Tampere, Finland.
Catalan Institute of Oncology , Bellvitge Biomedical Research Institute, Barcelona, Spain.

Eero Pukkala (E)

Tampere University, Tampere, Finland.

Anna Söderlund-Strand (A)

Department of Clinical Microbiology, Skåne University Hospital, Lund, Sweden.

Gary Dubin (G)

Takeda Pharmaceuticals International, Zurich, Switzerland.

Geoff Garnett (G)

Gates Foundation, Seattle, Washington, USA.

Joakim Dillner (J)

Department of Laboratory Medicine, Karolinska Institute, Stockholm, Sweden.

Matti Lehtinen (M)

Department of Laboratory Medicine, Karolinska Institute, Stockholm, Sweden.
Tampere University, Tampere, Finland.
Deutsches Krebsforschungszentrum, Heidelberg, Germany.

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