Maternal plasma lipid levels across pregnancy and the risks of small-for-gestational age and low birth weight: a cohort study from rural Gambia.


Journal

BMC pregnancy and childbirth
ISSN: 1471-2393
Titre abrégé: BMC Pregnancy Childbirth
Pays: England
ID NLM: 100967799

Informations de publication

Date de publication:
12 Mar 2020
Historique:
received: 19 08 2019
accepted: 24 02 2020
entrez: 14 3 2020
pubmed: 14 3 2020
medline: 29 12 2020
Statut: epublish

Résumé

Sub-optimal maternal lipid levels during pregnancy may be implicated in the pathophysiological mechanisms leading to low birth weight (LBW) and small-for-gestational-age (SGA). We aimed to determine whether maternal lipid levels across pregnancy were associated with birth weight and the risks of LBW and SGA in rural Gambia. This secondary analysis of the ENID trial involved 573 pregnant women with term deliveries. Plasma levels of total cholesterol (TC), high-density lipoprotein cholesterol (HDL-c), low-density lipoprotein cholesterol (LDL-c), and triglycerides (TG) were analyzed at enrolment (mean (SD) = 13.9 (3.3) weeks gestation), 20 and 30 weeks gestation as continuous variables and percentile groups. Regression models with adjustment for confounders were used to examine associations between gestational lipid levels and birth weight and the risks of LBW (birth weight < 2500 g) and SGA (<10th percentile INTERGROWTH-21ST for birth weight). There were 7.9% LBW and 32.5% SGA infants. At enrolment, every unit increase in HDL-c was associated with a 2.7% (P = 0.011) reduction in relative risk of LBW. At 20 weeks gestation, every unit increase in TC levels was associated with a 1.3% reduction in relative risk of LBW (P = 0.002). Low (<10th percentile) HDL-c at enrolment or at 20 weeks gestation was associated with a 2.6 (P = 0.007) and 3.0 (P = 0.003) times greater risk of LBW, respectively, compared with referent (10th─90th) HDL-c. High (>90th percentile) LDL-c at 30 weeks gestation was associated with a 55% lower risk of SGA compared with referent LDL-c (P = 0.017). Increased levels of TC (β = 1.3, P = 0.027) at 20 weeks gestation and of TC (β = 1.2, P = 0.006) and LDL-c (β = 1.5, P = 0.002) at 30 weeks gestation were all associated with higher birth weight. In rural Gambia, lipid levels during pregnancy were associated with infant birth weight and the risks of LBW and SGA. Associations varied by lipid class and changed across pregnancy, indicating an adaptive process by which maternal lipids may influence fetal growth and birth outcomes. This trial was registered as ISRCTN49285450 on: 12/11/2009.

Sections du résumé

BACKGROUND BACKGROUND
Sub-optimal maternal lipid levels during pregnancy may be implicated in the pathophysiological mechanisms leading to low birth weight (LBW) and small-for-gestational-age (SGA). We aimed to determine whether maternal lipid levels across pregnancy were associated with birth weight and the risks of LBW and SGA in rural Gambia.
METHODS METHODS
This secondary analysis of the ENID trial involved 573 pregnant women with term deliveries. Plasma levels of total cholesterol (TC), high-density lipoprotein cholesterol (HDL-c), low-density lipoprotein cholesterol (LDL-c), and triglycerides (TG) were analyzed at enrolment (mean (SD) = 13.9 (3.3) weeks gestation), 20 and 30 weeks gestation as continuous variables and percentile groups. Regression models with adjustment for confounders were used to examine associations between gestational lipid levels and birth weight and the risks of LBW (birth weight < 2500 g) and SGA (<10th percentile INTERGROWTH-21ST for birth weight).
RESULTS RESULTS
There were 7.9% LBW and 32.5% SGA infants. At enrolment, every unit increase in HDL-c was associated with a 2.7% (P = 0.011) reduction in relative risk of LBW. At 20 weeks gestation, every unit increase in TC levels was associated with a 1.3% reduction in relative risk of LBW (P = 0.002). Low (<10th percentile) HDL-c at enrolment or at 20 weeks gestation was associated with a 2.6 (P = 0.007) and 3.0 (P = 0.003) times greater risk of LBW, respectively, compared with referent (10th─90th) HDL-c. High (>90th percentile) LDL-c at 30 weeks gestation was associated with a 55% lower risk of SGA compared with referent LDL-c (P = 0.017). Increased levels of TC (β = 1.3, P = 0.027) at 20 weeks gestation and of TC (β = 1.2, P = 0.006) and LDL-c (β = 1.5, P = 0.002) at 30 weeks gestation were all associated with higher birth weight.
CONCLUSIONS CONCLUSIONS
In rural Gambia, lipid levels during pregnancy were associated with infant birth weight and the risks of LBW and SGA. Associations varied by lipid class and changed across pregnancy, indicating an adaptive process by which maternal lipids may influence fetal growth and birth outcomes.
TRIAL REGISTRATION BACKGROUND
This trial was registered as ISRCTN49285450 on: 12/11/2009.

Identifiants

pubmed: 32164563
doi: 10.1186/s12884-020-2834-1
pii: 10.1186/s12884-020-2834-1
pmc: PMC7068879
doi:

Substances chimiques

Cholesterol, HDL 0
Cholesterol, LDL 0
Lipids 0
Triglycerides 0

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

153

Subventions

Organisme : Medical Research Council
ID : MC_U123292701
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/P012019/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC-A760-5QX00
Pays : United Kingdom
Organisme : Bill and Melinda Gates Foundation
ID : OPP1110668

Références

Am J Physiol Endocrinol Metab. 2011 Aug;301(2):E416-25
pubmed: 21586694
Obesity (Silver Spring). 2011 Jul;19(7):1476-81
pubmed: 21394096
Int J Epidemiol. 2011 Jun;40(3):647-61
pubmed: 21324938
J Perinatol. 2017 Jun;37(6):629-635
pubmed: 28333159
FASEB J. 2018 Feb;32(2):717-727
pubmed: 28982731
J Lipid Res. 1998 Mar;39(3):518-30
pubmed: 9548585
Trop Med Int Health. 2017 Jan;22(1):52-62
pubmed: 27761979
Acta Obstet Gynecol Scand. 2015 Aug;94(8):852-60
pubmed: 25912426
Lancet Glob Health. 2016 Feb;4(2):e98-e108
pubmed: 26795602
Circulation. 2011 Dec 20;124(25):2839-46
pubmed: 22124377
Am J Obstet Gynecol. 2012 Sep;207(3):202.e19-25
pubmed: 22728028
Cardiol Clin. 2015 May;33(2):209-15
pubmed: 25939294
Am J Obstet Gynecol. 2004 May;190(5):1359-68
pubmed: 15167842
Acta Obstet Gynecol Scand. 2012 Jun;91(6):726-35
pubmed: 22404756
PLoS One. 2019 Feb 8;14(2):e0212116
pubmed: 30735555
J Clin Diagn Res. 2016 Mar;10(3):QC12-6
pubmed: 27134947
Cochrane Database Syst Rev. 2018 Aug 31;8:CD012610
pubmed: 30168868
BMC Pregnancy Childbirth. 2017 Oct 16;17(1):360
pubmed: 29037224
Matern Child Nutr. 2017 Oct;13(4):
pubmed: 28025862
Am J Obstet Gynecol. 2009 Nov;201(5):482.e1-8
pubmed: 19631920
Eur J Endocrinol. 2007 Nov;157(5):605-12
pubmed: 17984240
Gynecol Obstet Invest. 2014;77(1):6-13
pubmed: 24334826
Endocrine. 2002 Oct;19(1):43-55
pubmed: 12583601
Diabetes Care. 2013 Sep;36(9):2706-13
pubmed: 23757425
J Pediatr. 2011 Nov;159(5):736-742.e1
pubmed: 21705016
Bull World Health Organ. 1987;65(5):663-737
pubmed: 3322602
Lancet. 2008 Jan 19;371(9608):261-9
pubmed: 18207020
Am J Clin Nutr. 2017 Jun;105(6):1474-1482
pubmed: 28490512
PLoS One. 2019 May 15;14(5):e0207978
pubmed: 31091240
Circ Res. 2009 Mar 13;104(5):600-8
pubmed: 19168441
J Lipid Res. 2004 May;45(5):948-53
pubmed: 14967821
BMC Pregnancy Childbirth. 2016 Mar 21;16:60
pubmed: 27000102
Int J Epidemiol. 2017 Apr 1;46(2):e13
pubmed: 26559544
J Hypertens. 2004 Nov;22(11):2177-83
pubmed: 15480103
BJOG. 2013 Sep;120 Suppl 2:9-26, v
pubmed: 23678873
Hypertens Pregnancy. 2014 Nov;33(4):508-23
pubmed: 25121342
Diabetes Care. 2008 Sep;31(9):1858-63
pubmed: 18606978
PLoS One. 2017 Feb 17;12(2):e0172533
pubmed: 28212393
Obstet Gynecol. 2011 Feb;117(2 Pt 1):225-32
pubmed: 21252733
Clin Sci (Lond). 2016 Apr 1;130(7):491-7
pubmed: 26888561
BMC Pregnancy Childbirth. 2012 Oct 11;12:107
pubmed: 23057665
Curr Opin Cardiol. 2015 Sep;30(5):536-42
pubmed: 26192490
Am J Hypertens. 2004 Jul;17(7):574-81
pubmed: 15233976
J Lipid Res. 2003 Oct;44(10):1909-18
pubmed: 12897187
N Engl J Med. 2007 Sep 27;357(13):1301-10
pubmed: 17898099
J Pediatr. 2013 Oct;163(4):983-8
pubmed: 23810722
Epidemiol Infect. 2019 Aug 30;147:e258
pubmed: 31469064
BMJ. 2017 Aug 17;358:j3677
pubmed: 28819030
Pediatrics. 2007 Oct;120(4):723-33
pubmed: 17908758
Diabet Med. 2005 Jan;22(1):21-5
pubmed: 15606686

Auteurs

Sandra G Okala (SG)

Department of Women and Children's Health, King's College London, London, SE1 7EH, UK.

Ebrima A Sise (EA)

MRC Unit The Gambia at the London School of Hygiene and Tropical Medicine, Banjul, The Gambia.

Fatou Sosseh (F)

MRC Unit The Gambia at the London School of Hygiene and Tropical Medicine, Banjul, The Gambia.

Andrew M Prentice (AM)

MRC Unit The Gambia at the London School of Hygiene and Tropical Medicine, Banjul, The Gambia.

Laura A Woollett (LA)

Department of Pathology and Laboratory Medicine, University of Cincinnati, Cincinnati, Ohio, USA.

Sophie E Moore (SE)

Department of Women and Children's Health, King's College London, London, SE1 7EH, UK. sophie.moore@kcl.ac.uk.
MRC Unit The Gambia at the London School of Hygiene and Tropical Medicine, Banjul, The Gambia. sophie.moore@kcl.ac.uk.

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Classifications MeSH