Use of Perampanel and a Ketogenic Diet in Nonketotic Hyperglycinemia: A Case Report.


Journal

Neuropediatrics
ISSN: 1439-1899
Titre abrégé: Neuropediatrics
Pays: Germany
ID NLM: 8101187

Informations de publication

Date de publication:
12 2020
Historique:
pubmed: 17 3 2020
medline: 21 10 2021
entrez: 17 3 2020
Statut: ppublish

Résumé

Nonketotic hyperglycinemia is a severe form of early onset epileptic encephalopathy caused by disturbances in the glycine cleavage system; the neurological damage is mainly attributed to overstimulation of the The patient presented with a severe form of nonketotic hyperglycinemia and experienced frequent epileptic spasms and focal seizures, which were resistant to vigabatrin, adrenocorticotropic hormone therapy, and combined dextromethorphan and sodium benzoate treatments. By 9 months of age, perampanel reduced epileptic spasms by >50%. At 14 months of age, the ketogenic diet markedly reduced focal seizures and glycine levels in the cerebrospinal fluid. Perampanel reduced fast excitatory neuronal activity, which was induced by an α-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptor, followed by prolonged electrical depolarizations due to an

Sections du résumé

BACKGROUND
Nonketotic hyperglycinemia is a severe form of early onset epileptic encephalopathy caused by disturbances in the glycine cleavage system; the neurological damage is mainly attributed to overstimulation of the
CASE
The patient presented with a severe form of nonketotic hyperglycinemia and experienced frequent epileptic spasms and focal seizures, which were resistant to vigabatrin, adrenocorticotropic hormone therapy, and combined dextromethorphan and sodium benzoate treatments. By 9 months of age, perampanel reduced epileptic spasms by >50%. At 14 months of age, the ketogenic diet markedly reduced focal seizures and glycine levels in the cerebrospinal fluid.
CONCLUSION
Perampanel reduced fast excitatory neuronal activity, which was induced by an α-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptor, followed by prolonged electrical depolarizations due to an

Identifiants

pubmed: 32176929
doi: 10.1055/s-0040-1708536
doi:

Substances chimiques

Anticonvulsants 0
Nitriles 0
Pyridones 0
perampanel H821664NPK

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

417-420

Informations de copyright

Thieme. All rights reserved.

Déclaration de conflit d'intérêts

None.

Auteurs

Atsuro Daida (A)

Division of Neurology, Saitama Children's Medical Center, Saitama-City, Saitama, Japan.
Department of Pediatrics, Tokyo Medical University, Shinjuku-ku, Tokyo, Japan.

Shin-Ichiro Hamano (SI)

Division of Neurology, Saitama Children's Medical Center, Saitama-City, Saitama, Japan.

Satoru Ikemoto (S)

Division of Neurology, Saitama Children's Medical Center, Saitama-City, Saitama, Japan.

Yuko Hirata (Y)

Division of Neurology, Saitama Children's Medical Center, Saitama-City, Saitama, Japan.

Ryuki Matsuura (R)

Division of Neurology, Saitama Children's Medical Center, Saitama-City, Saitama, Japan.

Reiko Koichihara (R)

Division of Neurology, Saitama Children's Medical Center, Saitama-City, Saitama, Japan.

Daiju Oba (D)

Division of Medical Genetics, Saitama Children's Medical Center, Saitama-City, Saitama, Japan.

Hirofumi Ohashi (H)

Division of Medical Genetics, Saitama Children's Medical Center, Saitama-City, Saitama, Japan.

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Classifications MeSH