Size of portally deprived liver lobe after portal vein ligation and additional partial hepatectomy: Result of balancing proliferation and apoptosis.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
17 03 2020
Historique:
received: 15 07 2019
accepted: 06 02 2020
entrez: 19 3 2020
pubmed: 19 3 2020
medline: 18 11 2020
Statut: epublish

Résumé

The liver has the ability to maintain its total size by adjusting the size of the individual liver lobes differently in response to regeneration- and atrophy-stimuli. Portal vein ligation (PVL) drives the ligated lobe to undergo atrophy whereas partial hepatectomy (PHx) drives the total remnant liver to regenerate. We hypothesize that the size of the PVL-lobe is dependent on the balance between the extent of PVL and the extent of PHx inducing a complex interplay between hepatocyte proliferation, apoptosis and autophagy. Lewis-rats were subjected to either 20%PVL + 70%PHx or 70%PVL + 20%PHx. Control groups consisted of 20%PVL and 70%PVL. Liver lobe weight, BrdU-proliferation-index, proliferating-cell-nuclear-antigen-mRNA-expression level, apoptotic density and autophagy-related-proteins were investigated. The PVL-liver lobe adjusted its weight differently, increasing by 40% after 20%PVL + 70%PHx, but decreasing by 25% after 70%PVL + 20%PHx. Additional resection induced a low, but substantial size-dependent hepatocyte proliferation rate (maximal 6.3% and 3.6% vs. 0.3% and significantly suppressed apoptotic density in the deportalized-liver-lobe (3 and 14 cells/mm

Identifiants

pubmed: 32184404
doi: 10.1038/s41598-020-60310-0
pii: 10.1038/s41598-020-60310-0
pmc: PMC7078252
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

4893

Références

Mao, S. A., Glorioso, J. M. & Nyberg, S. L. Liver regeneration. Transl. Res. 163, 352–362, https://doi.org/10.1016/j.trsl.2014.01.005 (2014).
doi: 10.1016/j.trsl.2014.01.005 pubmed: 24495569 pmcid: 3976740
Ueda, J., Chijiiwa, K. & Nakano, K. Cyclin expression in the atrophying and proliferating lobes of the liver after portal vein branch ligation and hepatectomy in rats. J. Surg. Res. 120, 89–96, https://doi.org/10.1016/j.jss.2003.11.020 (2004).
doi: 10.1016/j.jss.2003.11.020 pubmed: 15172194
Wei, W. et al. Intrahepatic Size Regulation in a Surgical Model: Liver Resection-Induced Liver Regeneration Counteracts the Local Atrophy following Simultaneous Portal Vein Ligation. Eur. Surg. Res. 57, 125–137, https://doi.org/10.1159/000446875 (2016).
doi: 10.1159/000446875 pubmed: 27308828
Glick, D., Barth, S. & Macleod, K. F. Autophagy: cellular and molecular mechanisms. J. Pathol. 221, 3–12, https://doi.org/10.1002/path.2697 (2010).
doi: 10.1002/path.2697 pubmed: 20225336 pmcid: 2990190
Bento, C. F. et al. Mammalian Autophagy: How Does It Work? Annu. Rev. Biochem. 85, 685–713, https://doi.org/10.1146/annurev-biochem-060815-014556 (2016).
doi: 10.1146/annurev-biochem-060815-014556 pubmed: 26865532
Kim, K. H. & Lee, M. S. Autophagy–a key player in cellular and body metabolism. Nat. Rev. Endocrinol. 10, 322–337, https://doi.org/10.1038/nrendo.2014.35 (2014).
doi: 10.1038/nrendo.2014.35 pubmed: 24663220
Madrigal-Matute, J. & Cuervo, A. M. Regulation of Liver Metabolism by Autophagy. Gastroenterology 150, 328–339, https://doi.org/10.1053/j.gastro.2015.09.042 (2016).
doi: 10.1053/j.gastro.2015.09.042 pubmed: 26453774
Wei, W. et al. Intrahepatic Size Regulation in a Surgical Model: Liver Resection-Induced Liver Regeneration Counteracts the Local Atrophy following Simultaneous Portal Vein Ligation. Eur. Surg. Res. 57, 125–137, https://doi.org/10.1159/000446875 (2016).
doi: 10.1159/000446875 pubmed: 27308828
Picard, C. et al. Molecular mechanisms of apoptosis in the liver of rats after portal branch ligation with and without retrorsine. Lab. Invest. 84, 618–628, https://doi.org/10.1038/labinvest.3700085 (2004).
doi: 10.1038/labinvest.3700085 pubmed: 15048135
Zhou, Y., Xu, J. C., Jia, Y. F. & Xu, C. S. Role of death receptors in the regulation of hepatocyte proliferation and apoptosis during rat liver regeneration. Genet. Mol. Res. 14, 14066–14075 (2015).
doi: 10.4238/2015.October.29.26
Kohler, U. A. et al. Activated Nrf2 impairs liver regeneration in mice by activation of genes involved in cell-cycle control and apoptosis. Hepatology 60, 670–678, https://doi.org/10.1002/hep.26964 (2014).
doi: 10.1002/hep.26964 pubmed: 24310875
Lin, C. W. et al. Amiodarone as an autophagy promoter reduces liver injury and enhances liver regeneration and survival in mice after partial hepatectomy. Sci. Rep. 5, 15807, https://doi.org/10.1038/srep15807 (2015).
doi: 10.1038/srep15807 pubmed: 26515640 pmcid: 4626804
Toshima, T. et al. Suppression of autophagy during liver regeneration impairs energy charge and hepatocyte senescence in mice. Hepatology 60, 290–300, https://doi.org/10.1002/hep.27140 (2014).
doi: 10.1002/hep.27140 pubmed: 24668739
Takagi, A. et al. Mammalian autophagy is essential for hepatic and renal ketogenesis during starvation. Sci.Rep. 6, 18944 (2016).
doi: 10.1038/srep18944
Iwao, Y. et al. 114304-Liver atrophy after percutaneous transhepatic portal embolization occurs in two histological phases: Hepatocellular atrophy followed by apoptosis. World J. hepatology 9, 1227–1238, https://doi.org/10.4254/wjh.v9.i32.1227 (2017).
doi: 10.4254/wjh.v9.i32.1227
Lalaoui, N., Lindqvist, L. M., Sandow, J. J. & Ekert, P. G. The molecular relationships between apoptosis, autophagy and necroptosis. Semin. Cell Dev. Biol. 39, 63–69, https://doi.org/10.1016/j.semcdb.2015.02.003 (2015).
doi: 10.1016/j.semcdb.2015.02.003 pubmed: 25736836
Islam, M. A., Sooro, M. A. & Zhang, P. Autophagic Regulation of p62 is Critical for Cancer Therapy. Int J Mol Sci 19, https://doi.org/10.3390/ijms19051405 (2018).
doi: 10.3390/ijms19051405
Sha, Z., Schnell, H. M., Ruoff, K. & Goldberg, A. Rapid induction of p62 and GABARAPL1 upon proteasome inhibition promotes survival before autophagy activation. J. Cell Biol. 217, 1757–1776, https://doi.org/10.1083/jcb.201708168 (2018).
doi: 10.1083/jcb.201708168 pubmed: 29535191 pmcid: 5940303
Zhang, L. et al. Redox signaling: Potential arbitrator of autophagy and apoptosis in therapeutic response. Free. Radic. Biol. Med. 89, 452–465, https://doi.org/10.1016/j.freeradbiomed.2015.08.030 (2015).
doi: 10.1016/j.freeradbiomed.2015.08.030 pubmed: 26454086
Marino, G., Niso-Santano, M., Baehrecke, E. H. & Kroemer, G. Self-consumption: the interplay of autophagy and apoptosis. Nat. Rev. Mol. Cell Biol. 15, 81–94, https://doi.org/10.1038/nrm3735 (2014).
doi: 10.1038/nrm3735 pubmed: 24401948 pmcid: 3970201
Chow, S. E., Chen, Y. W., Liang, C. A., Huang, Y. K. & Wang, J. S. Wogonin induces cross-regulation between autophagy and apoptosis via a variety of Akt pathway in human nasopharyngeal carcinoma cells. J. Cell Biochem. 113, 3476–3485, https://doi.org/10.1002/jcb.24224 (2012).
doi: 10.1002/jcb.24224 pubmed: 22689083
Shi, H. B. et al. Autophagy in anti-apoptotic effect of augmenter of liver regeneration in HepG2 cells. World J. Gastroenterol. 21, 5250–5258, https://doi.org/10.3748/wjg.v21.i17.5250 (2015).
doi: 10.3748/wjg.v21.i17.5250 pubmed: 25954098 pmcid: 4419065
Kilkenny, C., Browne, W. J., Cuthill, I. C., Emerson, M. & Altman, D. G. Improving bioscience research reporting: the ARRIVE guidelines for reporting animal research. PLoS. Biol. 8, e1000412 (2010).
doi: 10.1371/journal.pbio.1000412
Madrahimov, N., Dirsch, O., Broelsch, C. & Dahmen, U. Marginal hepatectomy in the rat: from anatomy to surgery. Ann. Surg. 244, 89–98 (2006).
doi: 10.1097/01.sla.0000218093.12408.0f
Wei, W. et al. Establishment of a rat model: Associating liver partition with portal vein ligation for staged hepatectomy. Surg. 159, 1299–1307 (2016).
doi: 10.1016/j.surg.2015.12.005
Eipel, C., Hirschmann, M., Abshagen, K., Menger, M. D. & Vollmar, B. Local interaction of apoptotic hepatocytes and Kupffer cells in a rat model of systemic endotoxemia. Hepatol. Res. 37, 863–871 (2007).
doi: 10.1111/j.1872-034X.2007.00133.x
Liu, A., Fang, H., Dahmen, U. & Dirsch, O. Chronic lithium treatment protects against liver ischemia/reperfusion injury in rats. Liver Transpl. 19, 762–772, https://doi.org/10.1002/lt.23666 (2013).
doi: 10.1002/lt.23666 pubmed: 23696274

Auteurs

Weiwei Wei (W)

Department of General, Visceral and Vascular Surgery, Jena University Hospital, Jena, Germany.

Chuanfeng Hua (C)

Department of General, Visceral and Vascular Surgery, Jena University Hospital, Jena, Germany.

Tianjiao Zhang (T)

Department of Radiotherapy and Radiooncology, Jena University Hospital, Jena, Germany.

Olaf Dirsch (O)

Institute of Pathology, Klinikum Chemnitz gGmbH, Chemnitz, Germany.

Felix Gremse (F)

Department of Experimental Molecular Imaging, RWTH Aachen University, Aachen, Germany.

André Homeyer (A)

Fraunhofer Institute for Medical Image Computing MEVIS, Bremen, Germany.

Utz Settmacher (U)

Department of General, Visceral and Vascular Surgery, Jena University Hospital, Jena, Germany.

Uta Dahmen (U)

Department of General, Visceral and Vascular Surgery, Jena University Hospital, Jena, Germany. Uta.Dahmen@med.uni-jena.de.

Articles similaires

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male
Humans Meals Time Factors Female Adult

Classifications MeSH