The metabolic disorders associated with chronic consumption of soft and energy drinks in rats.


Journal

Acta biochimica Polonica
ISSN: 1734-154X
Titre abrégé: Acta Biochim Pol
Pays: Poland
ID NLM: 14520300R

Informations de publication

Date de publication:
19 Mar 2020
Historique:
received: 20 11 2019
accepted: 27 02 2020
pubmed: 20 3 2020
medline: 6 11 2020
entrez: 20 3 2020
Statut: ppublish

Résumé

Energy Drinks (EDs) and Soft Drinks (SDs) are widely consumed among adolescents and young adults. These drinks contain variable amounts of caffeine which is a central nervous system stimulator; in addition to sugar, taurine, vitamins and herbal extracts. Several adverse effects have been reported for the excessive consumption of caffeine and sugar. This work aimed at providing a comparison between the effect of chronic consumption of both drinks on metabolism biochemically as well as at the histopathological level. Adult albino rats were randomly divided into three groups and treated for 4 weeks. Animals received water (control, group 1), 12.5 ml/kg/day of either Pepsi® (SD, group 2) or Power Horse® (ED, group 3). All animals had free access to water and standard animal chow. ED and SD groups showed a significant weight gain compared to control. ED animals showed a significant increase in serum urea, hyperlipidemia and hyperglycemia in comparison to control and SD groups. Serum uric acid significantly increased in ED and SD groups. ED group showed congestion and inflammation in their renal tissues in addition to splenomegaly and increased phagocyte infiltration. The high caffeine-sugar content in ED exerts a more significant influence on the metabolic pathways than SDs. Both increase the incidence of cardiovascular diseases and tissue inflammation due to their effect on lipid profile and blood glucose. The other ingredients in EDs may play a role in the observed metabolic disturbances. Chronic use of EDs should be especially discouraged to avoid these negative effects.

Sections du résumé

BACKGROUND BACKGROUND
Energy Drinks (EDs) and Soft Drinks (SDs) are widely consumed among adolescents and young adults. These drinks contain variable amounts of caffeine which is a central nervous system stimulator; in addition to sugar, taurine, vitamins and herbal extracts. Several adverse effects have been reported for the excessive consumption of caffeine and sugar.
AIM OBJECTIVE
This work aimed at providing a comparison between the effect of chronic consumption of both drinks on metabolism biochemically as well as at the histopathological level.
METHODS METHODS
Adult albino rats were randomly divided into three groups and treated for 4 weeks. Animals received water (control, group 1), 12.5 ml/kg/day of either Pepsi® (SD, group 2) or Power Horse® (ED, group 3). All animals had free access to water and standard animal chow.
RESULTS RESULTS
ED and SD groups showed a significant weight gain compared to control. ED animals showed a significant increase in serum urea, hyperlipidemia and hyperglycemia in comparison to control and SD groups. Serum uric acid significantly increased in ED and SD groups. ED group showed congestion and inflammation in their renal tissues in addition to splenomegaly and increased phagocyte infiltration.
CONCLUSION CONCLUSIONS
The high caffeine-sugar content in ED exerts a more significant influence on the metabolic pathways than SDs. Both increase the incidence of cardiovascular diseases and tissue inflammation due to their effect on lipid profile and blood glucose. The other ingredients in EDs may play a role in the observed metabolic disturbances. Chronic use of EDs should be especially discouraged to avoid these negative effects.

Identifiants

pubmed: 32191411
pii: 2914
doi: 10.18388/abp.2020_2914
doi:

Substances chimiques

Blood Glucose 0
Lipids 0
Sugars 0
Caffeine 3G6A5W338E

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

79-84

Auteurs

Heba M Eltahir (HM)

Department of Pharmacology and Toxicology (subdivision of Biochemistry), College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia.

Ghadi Alamri (G)

College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia.

Asmaa Alamri (A)

College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia.

Asmaa Aloufi (A)

College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia.

Maiiada Nazmy (M)

Department of Biochemistry, Faculty of Pharmacy, Minia University, Minia, Egypt.

Hossein Elbadawy (H)

Department of Pharmacology and Toxicology, College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia.

Saleh Bahashwan (S)

Department of Pharmacology and Toxicology, College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia.

Hossein M Elbadawy (HM)

Department of Pharmacology and Toxicology, College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia.

Yaser M Alahmadi (YM)

Departments of Clinical and Hospital Pharmacy, College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia.

Mekky Abouzied (M)

1Department of Pharmacology and Toxicology (subdivision of Biochemistry), College of Pharmacy, Taibah University, Medina, Kingdom of Saudi Arabia; 2Department of Biochemistry, Faculty of Pharmacy, Minia University, Minia, Egypt.

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Classifications MeSH