Class II phosphatidylinositol 3-kinase α and β isoforms are required for vascular smooth muscle Rho activation, contraction and blood pressure regulation in mice.


Journal

The journal of physiological sciences : JPS
ISSN: 1880-6562
Titre abrégé: J Physiol Sci
Pays: Japan
ID NLM: 101262417

Informations de publication

Date de publication:
19 Mar 2020
Historique:
received: 22 01 2020
accepted: 10 03 2020
entrez: 21 3 2020
pubmed: 21 3 2020
medline: 20 2 2021
Statut: epublish

Résumé

Class II phosphatidylinositol 3-kinases (PI3K), PI3K-C2α and PI3K-C2β, are involved in cellular processes including endocytosis, cilia formation and autophagy. However, the role of PI3K-C2α and PI3K-C2β at the organismal level is not well understood. We found that double knockout (KO) mice with both smooth muscle-specific KO of PI3K-C2α and global PI3K-C2β KO, but not single KO mice of either PI3K-C2α or PI3K-C2β, exhibited reductions in arterial blood pressure and substantial attenuation of contractile responses of isolated aortic rings. In wild-type vascular smooth muscle cells, double knockdown of PI3K-C2α and PI3K-C2β but not single knockdown of either PI3K markedly inhibited contraction with reduced phosphorylation of 20-kDa myosin light chain and MYPT1 and Rho activation, but without inhibition of the intracellular Ca

Identifiants

pubmed: 32192434
doi: 10.1186/s12576-020-00745-2
pii: 10.1186/s12576-020-00745-2
pmc: PMC7082390
doi:

Substances chimiques

Isoenzymes 0
Class II Phosphatidylinositol 3-Kinases EC 2.7.1.137
rho GTP-Binding Proteins EC 3.6.5.2
Calcium SY7Q814VUP

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

18

Subventions

Organisme : Ministry of Education, Culture, Sports, Science and Technology
ID : 25116711
Organisme : Japan Society for the Promotion of Science
ID : 17K08532
Organisme : Japan Society for the Promotion of Science
ID : 16K18988
Organisme : Japan Society for the Promotion of Science
ID : 16K15409
Organisme : Japan Society for the Promotion of Science
ID : 17K08542
Organisme : Japan Society for the Promotion of Science
ID : 15H04673

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Auteurs

Shahidul Islam (S)

Department of Physiology, Kanazawa University Graduate School of Medical Sciences, 13-1 Takara-machi, Kanazawa, Ishikawa, 920-8640, Japan.

Kazuaki Yoshioka (K)

Department of Physiology, Kanazawa University Graduate School of Medical Sciences, 13-1 Takara-machi, Kanazawa, Ishikawa, 920-8640, Japan.

Sho Aki (S)

Department of Physiology, Kanazawa University Graduate School of Medical Sciences, 13-1 Takara-machi, Kanazawa, Ishikawa, 920-8640, Japan.

Kazuhiro Ishimaru (K)

Department of Physiology, Kanazawa University Graduate School of Medical Sciences, 13-1 Takara-machi, Kanazawa, Ishikawa, 920-8640, Japan.

Hiroki Yamada (H)

Department of Physiology, Kanazawa University Graduate School of Medical Sciences, 13-1 Takara-machi, Kanazawa, Ishikawa, 920-8640, Japan.

Noriko Takuwa (N)

Department of Physiology, Kanazawa University Graduate School of Medical Sciences, 13-1 Takara-machi, Kanazawa, Ishikawa, 920-8640, Japan.
Department of Health Science, Ishikawa Prefectural University, Kahoku, Ishikawa, 929-1210, Japan.

Yoh Takuwa (Y)

Department of Physiology, Kanazawa University Graduate School of Medical Sciences, 13-1 Takara-machi, Kanazawa, Ishikawa, 920-8640, Japan. ytakuwa@med.kanazawa-u.ac.jp.

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Classifications MeSH