Leaking chemokines confuse neutrophils.


Journal

The Journal of clinical investigation
ISSN: 1558-8238
Titre abrégé: J Clin Invest
Pays: United States
ID NLM: 7802877

Informations de publication

Date de publication:
01 05 2020
Historique:
pubmed: 24 3 2020
medline: 22 1 2021
entrez: 24 3 2020
Statut: ppublish

Résumé

The physical integrity of endothelial cells (ECs) lining the blood vessels regulates the inflammatory response. Both innate immunity and inflammatory disorders hinge on the EC-neutrophil interaction. Neutrophil binding, rolling, and migrating along and between ECs is associated with vascular permeability. In this issue of the JCI, Owen-Woods et al. tracked neutrophils in vivo in venules of mouse striated muscle and revealed how endothelial permeability can affect neutrophil trafficking. Strikingly, many neutrophils that migrated between EC junctions were able to rejoin the blood circulation. Further, the chemokine and neutrophil chemoattractant, CXCL1, drove this reverse transendothelial migration (rTEM). This paradigm-shifting study provides a mechanism for distal organ damage as well as an explanation for sepsis-associated acute respiratory distress syndrome.

Identifiants

pubmed: 32202515
pii: 136259
doi: 10.1172/JCI136259
pmc: PMC7190970
doi:
pii:

Substances chimiques

Chemokines 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Comment

Langues

eng

Sous-ensembles de citation

IM

Pagination

2177-2179

Subventions

Organisme : NHLBI NIH HHS
ID : P01 HL078784
Pays : United States

Commentaires et corrections

Type : CommentOn

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Auteurs

Alex Marki (A)

La Jolla Institute for Immunology, La Jolla, California, USA.

Klaus Ley (K)

La Jolla Institute for Immunology, La Jolla, California, USA.
Department of Bioengineering and.
Institute of Engineering in Medicine, UCSD, La Jolla, California, USA.

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Classifications MeSH