DNA methylation disruption reshapes the hematopoietic differentiation landscape.


Journal

Nature genetics
ISSN: 1546-1718
Titre abrégé: Nat Genet
Pays: United States
ID NLM: 9216904

Informations de publication

Date de publication:
04 2020
Historique:
received: 16 04 2019
accepted: 19 02 2020
pubmed: 24 3 2020
medline: 27 6 2020
entrez: 24 3 2020
Statut: ppublish

Résumé

Mutations in genes involved in DNA methylation (DNAme; for example, TET2 and DNMT3A) are frequently observed in hematological malignancies

Identifiants

pubmed: 32203468
doi: 10.1038/s41588-020-0595-4
pii: 10.1038/s41588-020-0595-4
pmc: PMC7216752
mid: NIHMS1564247
doi:

Substances chimiques

DNA-Binding Proteins 0
DNA (Cytosine-5-)-Methyltransferases EC 2.1.1.37

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

378-387

Subventions

Organisme : NCI NIH HHS
ID : R00 CA218896
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL128239
Pays : United States
Organisme : NCI NIH HHS
ID : K08 CA215317
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NIGMS NIH HHS
ID : R35 GM122515
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL145283
Pays : United States
Organisme : NCI NIH HHS
ID : DP2 CA239065
Pays : United States

Commentaires et corrections

Type : CommentIn

Références

Ley, T. J. et al. DNMT3A mutations in acute myeloid leukemia. N. Engl. J. Med. 363, 2424–2433 (2010).
pubmed: 21067377 pmcid: 3201818 doi: 10.1056/NEJMoa1005143
Delhommeau, F. et al. Mutation in TET2 in myeloid cancers. N. Engl. J. Med. 360, 2289–2301 (2009).
pubmed: 19474426 doi: 10.1056/NEJMoa0810069
Gross, S. et al. Cancer-associated metabolite 2-hydroxyglutarate accumulates in acute myelogenous leukemia with isocitrate dehydrogenase 1 and 2 mutations. J. Exp. Med. 207, 339–344 (2010).
pubmed: 20142433 pmcid: 2822606 doi: 10.1084/jem.20092506
Busque, L. et al. Recurrent somatic TET2 mutations in normal elderly individuals with clonal hematopoiesis. Nat. Genet. 44, 1179–1181 (2012).
pubmed: 23001125 pmcid: 3483435 doi: 10.1038/ng.2413
Abelson, S. et al. Prediction of acute myeloid leukaemia risk in healthy individuals. Nature 559, 400–404 (2018).
pubmed: 29988082 pmcid: 6485381 doi: 10.1038/s41586-018-0317-6
Chang, H. H., Hemberg, M., Barahona, M., Ingber, D. E. & Huang, S. Transcriptome-wide noise controls lineage choice in mammalian progenitor cells. Nature 453, 544–547 (2008).
pubmed: 18497826 pmcid: 5546414 doi: 10.1038/nature06965
Velten, L. et al. Human haematopoietic stem cell lineage commitment is a continuous process. Nat. Cell Biol. 19, 271–281 (2017).
pubmed: 28319093 pmcid: 5496982 doi: 10.1038/ncb3493
Graf, T. & Stadtfeld, M. Heterogeneity of embryonic and adult stem cells. Cell Stem Cell 3, 480–483 (2008).
pubmed: 18983963 doi: 10.1016/j.stem.2008.10.007
Yu, V. W. C. et al. Epigenetic memory underlies cell-autonomous heterogeneous behavior of hematopoietic stem cells. Cell 168, 944–945 (2017).
pubmed: 28235203 pmcid: 5510238 doi: 10.1016/j.cell.2017.02.010
Bintu, L. et al. Dynamics of epigenetic regulation at the single-cell level. Science 351, 720–724 (2016).
pubmed: 26912859 pmcid: 5108652 doi: 10.1126/science.aab2956
Bird, A. DNA methylation patterns and epigenetic memory. Genes Dev. 16, 6–21 (2002).
pubmed: 11782440 doi: 10.1101/gad.947102
Domcke, S. et al. Competition between DNA methylation and transcription factors determines binding of NRF1. Nature 528, 575–579 (2015).
pubmed: 26675734 doi: 10.1038/nature16462
Stone, A. et al. DNA methylation of oestrogen-regulated enhancers defines endocrine sensitivity in breast cancer. Nat. Commun. 6, 7758 (2015).
pubmed: 26169690 doi: 10.1038/ncomms8758
Prendergast, G. C. & Ziff, E. B. Methylation-sensitive sequence-specific DNA binding by the c-Myc basic region. Science 251, 186–189 (1991).
pubmed: 1987636 doi: 10.1126/science.1987636
Yin, Y. et al. Impact of cytosine methylation on DNA binding specificities of human transcription factors. Science 356, eaaj2239 (2017).
pubmed: 28473536 doi: 10.1126/science.aaj2239 pmcid: 8009048
Kribelbauer, J. F. et al. Quantitative analysis of the DNA methylation sensitivity of transcription factor complexes. Cell Rep. 19, 2383–2395 (2017).
pubmed: 28614722 pmcid: 5533174 doi: 10.1016/j.celrep.2017.05.069
Yang, L. et al. DNMT3A loss drives enhancer hypomethylation in FLT3-ITD-associated leukemias. Cancer Cell 30, 363–365 (2016).
pubmed: 27505680 doi: 10.1016/j.ccell.2016.07.015
Bock, C. et al. DNA methylation dynamics during in vivo differentiation of blood and skin stem cells. Mol. Cell 47, 633–647 (2012).
pubmed: 22841485 pmcid: 3428428 doi: 10.1016/j.molcel.2012.06.019
Ji, H. et al. Comprehensive methylome map of lineage commitment from haematopoietic progenitors. Nature 467, 338–342 (2010).
pubmed: 20720541 pmcid: 2956609 doi: 10.1038/nature09367
Xu, W. et al. Oncometabolite 2-hydroxyglutarate is a competitive inhibitor of alpha-ketoglutarate-dependent dioxygenases. Cancer Cell 19, 17–30 (2011).
pubmed: 21251613 pmcid: 3229304 doi: 10.1016/j.ccr.2010.12.014
Abdel-Wahab, O. & Levine, R. L. Mutations in epigenetic modifiers in the pathogenesis and therapy of acute myeloid leukemia. Blood 121, 3563–3572 (2013).
pubmed: 23640996 pmcid: 3643757 doi: 10.1182/blood-2013-01-451781
Sperling, A. S., Gibson, C. J. & Ebert, B. L. The genetics of myelodysplastic syndrome: from clonal haematopoiesis to secondary leukaemia. Nat. Rev. Cancer 17, 5–19 (2017).
pubmed: 27834397 doi: 10.1038/nrc.2016.112
Steensma, D. P. et al. Clonal hematopoiesis of indeterminate potential and its distinction from myelodysplastic syndromes. Blood 126, 9–16 (2015).
pubmed: 25931582 pmcid: 4624443 doi: 10.1182/blood-2015-03-631747
Jaiswal, S. et al. Clonal hematopoiesis and risk of atherosclerotic cardiovascular disease. N. Engl. J. Med. 377, 111–121 (2017).
pubmed: 28636844 pmcid: 6717509 doi: 10.1056/NEJMoa1701719
Genovese, G., Jaiswal, S., Ebert, B. L. & McCarroll, S. A. Clonal hematopoiesis and blood-cancer risk. N. Engl. J. Med. 372, 1071–1072 (2015).
pubmed: 25760361 doi: 10.1056/NEJMc1500684
Jaiswal, S. et al. Age-related clonal hematopoiesis associated with adverse outcomes. N. Engl. J. Med. 371, 2488–2498 (2014).
pubmed: 25426837 pmcid: 4306669 doi: 10.1056/NEJMoa1408617
Couronne, L., Bastard, C. & Bernard, O. A. TET2 and DNMT3A mutations in human T-cell lymphoma. N. Engl. J. Med. 366, 95–96 (2012).
pubmed: 22216861 doi: 10.1056/NEJMc1111708
Li, W. et al. DNMT3A mutations and prognostic significance in childhood acute lymphoblastic leukemia. Leuk. Lymphoma 56, 1066–1071 (2015).
pubmed: 25242092 doi: 10.3109/10428194.2014.947607
Mayle, A. et al. Dnmt3a loss predisposes murine hematopoietic stem cells to malignant transformation. Blood 125, 629–638 (2015).
pubmed: 25416277 pmcid: 4304108 doi: 10.1182/blood-2014-08-594648
Kramer, A. C. et al. Dnmt3a regulates T-cell development and suppresses T-ALL transformation. Leukemia 31, 2479–2490 (2017).
pubmed: 28321121 pmcid: 5636646 doi: 10.1038/leu.2017.89
Pan, F. et al. Tet2 loss leads to hypermutagenicity in haematopoietic stem/progenitor cells. Nat. Commun. 8, 15102 (2017).
pubmed: 28440315 pmcid: 5414116 doi: 10.1038/ncomms15102
Paul, F. et al. Transcriptional heterogeneity and lineage commitment in myeloid progenitors. Cell 163, 1663–1677 (2015).
pubmed: 26627738 doi: 10.1016/j.cell.2015.11.013
Wilson, N. K. et al. Combined single-cell functional and gene expression analysis resolves heterogeneity within stem cell populations. Cell Stem Cell 16, 712–724 (2015).
pubmed: 26004780 pmcid: 4460190 doi: 10.1016/j.stem.2015.04.004
Mildner, A. et al. Genomic characterization of murine monocytes reveals C/EBPβ transcription factor dependence of Ly6C
pubmed: 28514690 doi: 10.1016/j.immuni.2017.04.018
Olsson, A. et al. Single-cell analysis of mixed-lineage states leading to a binary cell fate choice. Nature 537, 698–702 (2016).
pubmed: 27580035 pmcid: 5161694 doi: 10.1038/nature19348
Yanez, A. et al. Granulocyte-monocyte progenitors and monocyte-dendritic cell progenitors independently produce functionally distinct monocytes. Immunity 47, 890–902.e4 (2017).
pubmed: 29166589 pmcid: 5726802 doi: 10.1016/j.immuni.2017.10.021
Drissen, R. et al. Distinct myeloid progenitor-differentiation pathways identified through single-cell RNA sequencing. Nat. Immunol. 17, 666–676 (2016).
pubmed: 27043410 pmcid: 4972405 doi: 10.1038/ni.3412
Ward, P. S. et al. The common feature of leukemia-associated IDH1 and IDH2 mutations is a neomorphic enzyme activity converting alpha-ketoglutarate to 2-hydroxyglutarate. Cancer Cell 17, 225–234 (2010).
pubmed: 20171147 pmcid: 2849316 doi: 10.1016/j.ccr.2010.01.020
Shih, A. H., Abdel-Wahab, O., Patel, J. P. & Levine, R. L. The role of mutations in epigenetic regulators in myeloid malignancies. Nat. Rev. Cancer 12, 599–612 (2012).
pubmed: 22898539 doi: 10.1038/nrc3343
Sugiyama, T., Kohara, H., Noda, M. & Nagasawa, T. Maintenance of the hematopoietic stem cell pool by CXCL12-CXCR4 chemokine signaling in bone marrow stromal cell niches. Immunity 25, 977–988 (2006).
pubmed: 17174120 doi: 10.1016/j.immuni.2006.10.016
Tzeng, Y. S. et al. Loss of Cxcl12/Sdf-1 in adult mice decreases the quiescent state of hematopoietic stem/progenitor cells and alters the pattern of hematopoietic regeneration after myelosuppression. Blood 117, 429–439 (2011).
pubmed: 20833981 doi: 10.1182/blood-2010-01-266833
Hwang, H. S. et al. Enhanced anti-leukemic effects through induction of immunomodulating microenvironment by blocking CXCR4 and PD-L1 in an AML mouse model. Immunol. Invest. 48, 96–105 (2019).
pubmed: 30204524 doi: 10.1080/08820139.2018.1497057
Cho, B. S., Kim, H. J. & Konopleva, M. Targeting the CXCL12/CXCR4 axis in acute myeloid leukemia: from bench to bedside. Korean J. Intern. Med. 32, 248–257 (2017).
pubmed: 28219003 pmcid: 5339474 doi: 10.3904/kjim.2016.244
Pujato, M., Kieken, F., Skiles, A. A., Tapinos, N. & Fiser, A. Prediction of DNA binding motifs from 3D models of transcription factors; identifying TLX3 regulated genes. Nucleic Acids Res. 42, 13500–13512 (2014).
pubmed: 25428367 pmcid: 4267649 doi: 10.1093/nar/gku1228
Heinz, S. et al. Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities. Mol. Cell 38, 576–589 (2010).
pubmed: 20513432 pmcid: 2898526 doi: 10.1016/j.molcel.2010.05.004
Gaiti, F. et al. Epigenetic evolution and lineage histories of chronic lymphocytic leukaemia. Nature 569, 576–580 (2019).
pubmed: 31092926 pmcid: 6533116 doi: 10.1038/s41586-019-1198-z
Kulakovskiy, I. V. et al. HOCOMOCO: towards a complete collection of transcription factor binding models for human and mouse via large-scale ChIP-Seq analysis. Nucleic Acids Res. 46, D252–D259 (2018).
pubmed: 29140464 doi: 10.1093/nar/gkx1106
Nam, A. S. et al. Somatic mutations and cell identity linked by Genotyping of Transcriptomes. Nature 571, 355–360 (2019).
pubmed: 31270458 pmcid: 6782071 doi: 10.1038/s41586-019-1367-0
Kunimoto, H. et al. Tet2-mutated myeloid progenitors possess aberrant in vitro self-renewal capacity. Blood 123, 2897–2899 (2014).
pubmed: 24786459 doi: 10.1182/blood-2014-01-552471
Verbist, K. C. et al. Metabolic maintenance of cell asymmetry following division in activated T lymphocytes. Nature 532, 389–393 (2016).
pubmed: 27064903 pmcid: 4851250 doi: 10.1038/nature17442
Wilson, A. et al. c-Myc controls the balance between hematopoietic stem cell self-renewal and differentiation. Genes Dev. 18, 2747–2763 (2004).
pubmed: 15545632 pmcid: 528895 doi: 10.1101/gad.313104
Giladi, A. et al. Single-cell characterization of haematopoietic progenitors and their trajectories in homeostasis and perturbed haematopoiesis. Nat. Cell Biol. 20, 836–846 (2018).
pubmed: 29915358 doi: 10.1038/s41556-018-0121-4
Zhang, X. et al. DNMT3A and TET2 compete and cooperate to repress lineage-specific transcription factors in hematopoietic stem cells. Nat. Genet. 48, 1014–1023 (2016).
pubmed: 27428748 pmcid: 4957136 doi: 10.1038/ng.3610
Emperle, M. et al. Mutations of R882 change flanking sequence preferences of the DNA methyltransferase DNMT3A and cellular methylation patterns. Nucleic Acids Res. 47, 11355–11367 (2019).
pubmed: 31620784 pmcid: 6868496 doi: 10.1093/nar/gkz911
Viner, C. et al. Modeling methyl-sensitive transcription factor motifs with an expanded epigenetic alphabet. Preprint at bioRxiv https://doi.org/10.1101/043794 (2016).
Lawrence, M. S. et al. Mutational heterogeneity in cancer and the search for new cancer-associated genes. Nature 499, 214–218 (2013).
pubmed: 23770567 pmcid: 3919509 doi: 10.1038/nature12213
Tetteh, P. W. et al. Replacement of lost Lgr5-positive stem cells through plasticity of their enterocyte-lineage daughters. Cell Stem Cell 18, 203–213 (2016).
pubmed: 26831517 doi: 10.1016/j.stem.2016.01.001
Kuleshov, M. V. et al. Enrichr: a comprehensive gene set enrichment analysis web server 2016 update. Nucleic Acids Res. 44, W90–W97 (2016).
pubmed: 27141961 pmcid: 4987924 doi: 10.1093/nar/gkw377
Lhoumaud, P. et al. EpiMethylTag: simultaneous detection of ATAC-seq or ChIP-seq signals with DNA methylation. Genome Biol. 20, 248 (2019).
pubmed: 31752933 pmcid: 6868874 doi: 10.1186/s13059-019-1853-6
Liu, T. Use Model-based Analysis of ChIP-Seq (MACS) to analyze short reads generated by sequencing protein-DNA interactions in embryonic stem cells. Methods Mol. Biol. 1150, 81–95 (2014).
pubmed: 24743991 doi: 10.1007/978-1-4939-0512-6_4
Love, M. I., Huber, W. & Anders, S. Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2. Genome Biol. 15, 550 (2014).
pubmed: 25516281 pmcid: 4302049 doi: 10.1186/s13059-014-0550-8
Akalin, A. et al. methylKit: a comprehensive R package for the analysis of genome-wide DNA methylation profiles. Genome Biol. 13, R87 (2012).
pubmed: 23034086 pmcid: 3491415 doi: 10.1186/gb-2012-13-10-r87
Yoshida, H. et al. The cis-regulatory atlas of the mouse immune system. Cell 176, 897–912.e20 (2019).
pubmed: 30686579 pmcid: 6785993 doi: 10.1016/j.cell.2018.12.036
Thurman, R. E. et al. The accessible chromatin landscape of the human genome. Nature 489, 75–82 (2012).
pubmed: 22955617 pmcid: 3721348 doi: 10.1038/nature11232
Schep, A. N., Wu, B., Buenrostro, J. D. & Greenleaf, W. J. chromVAR: inferring transcription-factor-associated accessibility from single-cell epigenomic data. Nat. Methods 14, 975–978 (2017).
pubmed: 28825706 pmcid: 5623146 doi: 10.1038/nmeth.4401
Moran-Crusio, K. et al. Tet2 loss leads to increased hematopoietic stem cell self-renewal and myeloid transformation. Cancer Cell 20, 11–24 (2011).
pubmed: 21723200 pmcid: 3194039 doi: 10.1016/j.ccr.2011.06.001
Nguyen, S., Meletis, K., Fu, D., Jhaveri, S. & Jaenisch, R. Ablation of de novo DNA methyltransferase Dnmt3a in the nervous system leads to neuromuscular defects and shortened lifespan. Dev. Dyn. 236, 1663–1676 (2007).
pubmed: 17477386 doi: 10.1002/dvdy.21176
Shih, A. H. et al. Combination targeted therapy to disrupt aberrant oncogenic signaling and reverse epigenetic dysfunction in IDH2- and TET2-mutant acute myeloid lLeukemia. Cancer Discov. 7, 494–505 (2017).
pubmed: 28193779 pmcid: 5413413 doi: 10.1158/2159-8290.CD-16-1049
Kuhn, R., Schwenk, F., Aguet, M. & Rajewsky, K. Inducible gene targeting in mice. Science 269, 1427–1429 (1995).
pubmed: 7660125 doi: 10.1126/science.7660125
Macosko, E. Z. et al. Highly parallel genome-wide expression profiling of individual cells using nanoliter droplets. Cell 161, 1202–1214 (2015).
pubmed: 26000488 pmcid: 4481139 doi: 10.1016/j.cell.2015.05.002
Hafemeister, C. S. & Satija, R. Normalization and variance stabilization of single-cell RNA-seq data using regularized negative binomial regression. Genome Biol. 20, 296 (2019).
pubmed: 31870423 pmcid: 6927181 doi: 10.1186/s13059-019-1874-1
Dobin, A. et al. STAR: ultrafast universal RNA-seq aligner. Bioinformatics 29, 15–21 (2013).
pubmed: 23104886 doi: 10.1093/bioinformatics/bts635
Han, X. et al. Mapping the Mouse Cell Atlas by Microwell-Seq. Cell 172, 1091–1107.e17 (2018).
pubmed: 29474909 doi: 10.1016/j.cell.2018.02.001
Sun, H., Zhou, Y., Fei, L., Chen, H. & Guo, G. scMCA: a tool to define mouse cell types based on single-cell digital expression. Methods Mol. Biol. 1935, 91–96 (2019).
pubmed: 30758821 doi: 10.1007/978-1-4939-9057-3_6
Bolker, B. M. et al. Generalized linear mixed models: a practical guide for ecology and evolution. Trends Ecol. Evol. 24, 127–135 (2009).
pubmed: 19185386 doi: 10.1016/j.tree.2008.10.008
Martin, J. C. et al. Single-cell analysis of Crohn’s disease lesions identifies a pathogenic cellular module associated with resistance to anti-TNF therapy. Cell 178, 1493–1508.e20 (2019).
pubmed: 31474370 pmcid: 7060942 doi: 10.1016/j.cell.2019.08.008
Orlanski, S. et al. Tissue-specific DNA demethylation is required for proper B-cell differentiation and function. Proc. Natl Acad. Sci. USA 113, 5018–5023 (2016).
pubmed: 27091986 doi: 10.1073/pnas.1604365113 pmcid: 4983829
Aibar, S. et al. SCENIC: single-cell regulatory network inference and clustering. Nat. Methods 14, 1083–1086 (2017).
pubmed: 28991892 pmcid: 5937676 doi: 10.1038/nmeth.4463
Macaulay, I. C. et al. G&T–seq: parallel sequencing of single-cell genomes and transcriptomes. Nat. Methods 12, 519–522 (2015).
pubmed: 25915121 doi: 10.1038/nmeth.3370
Picelli, S. et al. Full-length RNA–seq from single cells using Smart–seq2. Nat. Protoc. 9, 171–181 (2014).
doi: 10.1038/nprot.2014.006 pubmed: 24385147
Krueger, F. & Andrews, S. R. Bismark: a flexible aligner and methylation caller for Bisulfite-Seq applications. Bioinformatics 27, 1571–1572 (2011).
pubmed: 21493656 pmcid: 3102221 doi: 10.1093/bioinformatics/btr167
Langmead, B. & Salzberg, S. L. Fast gapped-read alignment with Bowtie 2. Nat Methods 9, 357–359 (2012).
pubmed: 22388286 pmcid: 3322381 doi: 10.1038/nmeth.1923
Harrow, J. et al. GENCODE: the reference human genome annotation for The ENCODE Project. Genome Res 22, 1760–1774 (2012).
pubmed: 22955987 pmcid: 3431492 doi: 10.1101/gr.135350.111

Auteurs

Franco Izzo (F)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Stanley C Lee (SC)

Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Clinical Research Division, Fred Hutchinson Cancer Research Center, Seattle, WA, USA.

Asaf Poran (A)

Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Ronan Chaligne (R)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Federico Gaiti (F)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Baptiste Gross (B)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Rekha R Murali (RR)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Sunil D Deochand (SD)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Chelston Ang (C)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Philippa Wyndham Jones (PW)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Anna S Nam (AS)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Kyu-Tae Kim (KT)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Steven Kothen-Hill (S)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Rafael C Schulman (RC)

New York Genome Center, New York, NY, USA.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA.

Michelle Ki (M)

Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Priscillia Lhoumaud (P)

New York University Langone Health, New York, NY, USA.

Jane A Skok (JA)

New York University Langone Health, New York, NY, USA.

Aaron D Viny (AD)

Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Ross L Levine (RL)

Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Ephraim Kenigsberg (E)

Department of Genetics and Genomic Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Icahn Institute for Data Science and Genomic Technology, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Omar Abdel-Wahab (O)

Human Oncology and Pathogenesis Program, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Dan A Landau (DA)

New York Genome Center, New York, NY, USA. dlandau@nygenome.org.
Meyer Cancer Center, Weill Cornell Medicine, New York, NY, USA. dlandau@nygenome.org.
Institute of Computational Biomedicine, Weill Cornell Medicine, New York, NY, USA. dlandau@nygenome.org.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH