Immune phenotype and histopathological growth pattern in patients with colorectal liver metastases.


Journal

British journal of cancer
ISSN: 1532-1827
Titre abrégé: Br J Cancer
Pays: England
ID NLM: 0370635

Informations de publication

Date de publication:
05 2020
Historique:
received: 02 12 2019
accepted: 03 03 2020
revised: 18 02 2020
pubmed: 25 3 2020
medline: 23 12 2020
entrez: 25 3 2020
Statut: ppublish

Résumé

Patients with desmoplastic (angiogenic) histopathological growth pattern (HGP) colorectal liver metastases (CLM) might derive more benefit from bevacizumab-based chemotherapy than those with replacement (non-angiogenic) HGP. This study investigated the association of HGP with the immune phenotype (IP) and clinical outcome after liver resection. CLM of patients treated with perioperative bevacizumab-based chemotherapy and liver resection were investigated. Association of HGP and IP with response, recurrence-free survival (RFS) and overall survival (OS) was investigated. One hundred and eighteen patients (M/F 66/52, median age 62.3 (31.0-80.4) years, median follow-up 32.2 (5.0-92.7) months) were enrolled. The inflamed IP was associated with the desmoplastic HGP. The desmoplastic HGP was associated with better radiological and histological response compared to the replacement HGP, respectively. The replacement HGP was associated with shorter RFS (8.7 versus 16.3 months, HR 2.60, P = 0.001) and OS (36.6 months versus not reached, HR 2.32, P = 0.027), respectively. The non-inflamed IP was associated with shorter RFS (10.8 versus 16.5 months, HR 1.85, P = 0.029). The HGP but not the IP remained significant in multivariable analysis for RFS. The desmoplastic HGP is associated with the inflamed IP and HGP may be a potential biomarker for adjuvant treatment that includes targeting the immune contexture.

Sections du résumé

BACKGROUND
Patients with desmoplastic (angiogenic) histopathological growth pattern (HGP) colorectal liver metastases (CLM) might derive more benefit from bevacizumab-based chemotherapy than those with replacement (non-angiogenic) HGP. This study investigated the association of HGP with the immune phenotype (IP) and clinical outcome after liver resection.
METHODS
CLM of patients treated with perioperative bevacizumab-based chemotherapy and liver resection were investigated. Association of HGP and IP with response, recurrence-free survival (RFS) and overall survival (OS) was investigated.
RESULTS
One hundred and eighteen patients (M/F 66/52, median age 62.3 (31.0-80.4) years, median follow-up 32.2 (5.0-92.7) months) were enrolled. The inflamed IP was associated with the desmoplastic HGP. The desmoplastic HGP was associated with better radiological and histological response compared to the replacement HGP, respectively. The replacement HGP was associated with shorter RFS (8.7 versus 16.3 months, HR 2.60, P = 0.001) and OS (36.6 months versus not reached, HR 2.32, P = 0.027), respectively. The non-inflamed IP was associated with shorter RFS (10.8 versus 16.5 months, HR 1.85, P = 0.029). The HGP but not the IP remained significant in multivariable analysis for RFS.
CONCLUSIONS
The desmoplastic HGP is associated with the inflamed IP and HGP may be a potential biomarker for adjuvant treatment that includes targeting the immune contexture.

Identifiants

pubmed: 32205863
doi: 10.1038/s41416-020-0812-z
pii: 10.1038/s41416-020-0812-z
pmc: PMC7217855
doi:

Substances chimiques

Bevacizumab 2S9ZZM9Q9V

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1518-1524

Subventions

Organisme : NCI NIH HHS
ID : P30 CA014089
Pays : United States
Organisme : Austrian Science Fund (Fonds zur Förderung der Wissenschaftlichen Forschung)
ID : J3501-B13
Pays : International

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Auteurs

Stefan Stremitzer (S)

USC/Norris Comprehensive Cancer Center, 1441 Eastlake Avenue, Los Angeles, CA, 90033, USA. stefan.stremitzer@meduniwien.ac.at.
Department of Surgery, Medical University Vienna, Waehringer Guertel 18-20, 1090, Vienna, Austria. stefan.stremitzer@meduniwien.ac.at.

Peter Vermeulen (P)

Histogenex, Sint-Bavostraat 78, 2610, Antwerp, Belgium.

Shannon Graver (S)

University of Wuerzburg, Biocenter, Am Hubland, 97074, Wuerzburg, Germany.

Mark Kockx (M)

Histogenex, Sint-Bavostraat 78, 2610, Antwerp, Belgium.

Luc Dirix (L)

Sint-Augustinus Hospital Oncology Center, Medical Oncology, Oosterveldlaan 24, 2610, Antwerp, Belgium.

Dongyun Yang (D)

USC/Norris Comprehensive Cancer Center, 1441 Eastlake Avenue, Los Angeles, CA, 90033, USA.

Wu Zhang (W)

USC/Norris Comprehensive Cancer Center, 1441 Eastlake Avenue, Los Angeles, CA, 90033, USA.

Judith Stift (J)

Clinical Institute of Pathology, Medical University Vienna, Waehringer Guertel 18-20, 1090, Vienna, Austria.

Friedrich Wrba (F)

Clinical Institute of Pathology, Medical University Vienna, Waehringer Guertel 18-20, 1090, Vienna, Austria.

Thomas Gruenberger (T)

Department of Surgery, Social Medical Center South, Kundratstrasse 3, 1100, Vienna, Austria.

Heinz-Josef Lenz (HJ)

USC/Norris Comprehensive Cancer Center, 1441 Eastlake Avenue, Los Angeles, CA, 90033, USA.

Stefan J Scherer (SJ)

University of Wuerzburg, Biocenter, Am Hubland, 97074, Wuerzburg, Germany.

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