Novel Prognostic Biomarkers of Mucosal Healing in Ulcerative Colitis Patients Treated With Anti-TNF: Neutrophil-to-Lymphocyte Ratio and Platelet-to-Lymphocyte Ratio.


Journal

Inflammatory bowel diseases
ISSN: 1536-4844
Titre abrégé: Inflamm Bowel Dis
Pays: England
ID NLM: 9508162

Informations de publication

Date de publication:
18 09 2020
Historique:
received: 18 01 2020
pubmed: 2 4 2020
medline: 21 10 2021
entrez: 2 4 2020
Statut: ppublish

Résumé

Anti-tumor necrosis factor drugs (anti-TNFs) are widely used for the treatment of ulcerative colitis (UC). However, many patients experience loss of response during the first year of therapy. An early predictor of clinical remission and mucosal healing is needed. The neutrophil-to-lymphocyte ratio (NLR) and platelet-to-lymphocyte ratio (PLR) are markers of subclinical inflammation poorly evaluated in UC patients treated with anti-TNFs. The aim of this multicenter study was to evaluate whether NLR and PLR could be used as prognostic markers of anti-TNF treatment response. Patients with UC who started anti-TNF treatment in monotherapy were evaluated. Patients with concomitant corticosteroid treatment ≥20 mg were excluded. We calculated NLR, PLR, and fecal calprotectin before treatment and after induction. The values of NLR and PLR were correlated with clinical remission and mucosal healing at the end of follow-up (54 weeks) using the Mann-Whitney U test and then multivariate analysis was conducted. Eighty-eight patients were included. Patients who reached mucosal healing after 54 weeks of therapy displayed lower levels of both baseline NLR and PLR (P = 0.0001 and P = 0.04, respectively); similar results were obtained at week 8 (P = 0.0001 and P = 0.001, respectively). Patients who presented with active ulcers at baseline endoscopic evaluation had higher baseline NLR and PLR values compared with those without detected ulcers (P = 0.002 and P = 0.0007, respectively). BothNLR and PLR showed a promising role as early predictors of therapeutic response to anti-TNF therapy in UC patients. If confirmed in larger studies, classification and regression trees proposed in this article could be useful to guide clinical decisions regarding anti-TNF treatment.

Sections du résumé

BACKGROUND
Anti-tumor necrosis factor drugs (anti-TNFs) are widely used for the treatment of ulcerative colitis (UC). However, many patients experience loss of response during the first year of therapy. An early predictor of clinical remission and mucosal healing is needed. The neutrophil-to-lymphocyte ratio (NLR) and platelet-to-lymphocyte ratio (PLR) are markers of subclinical inflammation poorly evaluated in UC patients treated with anti-TNFs. The aim of this multicenter study was to evaluate whether NLR and PLR could be used as prognostic markers of anti-TNF treatment response.
METHODS
Patients with UC who started anti-TNF treatment in monotherapy were evaluated. Patients with concomitant corticosteroid treatment ≥20 mg were excluded. We calculated NLR, PLR, and fecal calprotectin before treatment and after induction. The values of NLR and PLR were correlated with clinical remission and mucosal healing at the end of follow-up (54 weeks) using the Mann-Whitney U test and then multivariate analysis was conducted.
RESULTS
Eighty-eight patients were included. Patients who reached mucosal healing after 54 weeks of therapy displayed lower levels of both baseline NLR and PLR (P = 0.0001 and P = 0.04, respectively); similar results were obtained at week 8 (P = 0.0001 and P = 0.001, respectively). Patients who presented with active ulcers at baseline endoscopic evaluation had higher baseline NLR and PLR values compared with those without detected ulcers (P = 0.002 and P = 0.0007, respectively).
CONCLUSIONS
BothNLR and PLR showed a promising role as early predictors of therapeutic response to anti-TNF therapy in UC patients. If confirmed in larger studies, classification and regression trees proposed in this article could be useful to guide clinical decisions regarding anti-TNF treatment.

Identifiants

pubmed: 32232392
pii: 5813970
doi: 10.1093/ibd/izaa062
doi:

Substances chimiques

Biomarkers 0
Tumor Necrosis Factor Inhibitors 0

Types de publication

Evaluation Study Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1579-1587

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

© 2020 Crohn’s & Colitis Foundation. Published by Oxford University Press. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Auteurs

Lorenzo Bertani (L)

Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Federico Rossari (F)

Institute of Life Sciences, Sant'Anna School of Advanced Studies, Pisa, Italy.

Brigida Barberio (B)

Department of Surgery, Oncology and Gastroenterology, University of Padua, Padua, Italy.

Maria Giulia Demarzo (MG)

Department of Internal Medicine, Gastroenterology Unit, University of Genoa, Genoa, Italy.

Gherardo Tapete (G)

Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Eleonora Albano (E)

Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Giovanni Baiano Svizzero (G)

Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Linda Ceccarelli (L)

Department of General Surgery and Gastroenterology, IBD Unit, Pisa University Hospital, Pisa, Italy.

Maria Gloria Mumolo (MG)

Department of General Surgery and Gastroenterology, IBD Unit, Pisa University Hospital, Pisa, Italy.

Chiara Brombin (C)

University Centre for Statistics in the Biomedical Sciences, Vita-Salute San Raffaele University, Milan, Italy.

Nicola de Bortoli (N)

Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Massimo Bellini (M)

Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Santino Marchi (S)

Department of Translational Research and New Technologies in Medicine and Surgery, University of Pisa, Pisa, Italy.

Giorgia Bodini (G)

Department of Internal Medicine, Gastroenterology Unit, University of Genoa, Genoa, Italy.

Edoardo Savarino (E)

Department of Surgery, Oncology and Gastroenterology, University of Padua, Padua, Italy.

Francesco Costa (F)

Department of General Surgery and Gastroenterology, IBD Unit, Pisa University Hospital, Pisa, Italy.

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