Isavuconazole as Primary Antifungal Prophylaxis in Patients With Acute Myeloid Leukemia or Myelodysplastic Syndrome: An Open-label, Prospective, Phase 2 Study.


Journal

Clinical infectious diseases : an official publication of the Infectious Diseases Society of America
ISSN: 1537-6591
Titre abrégé: Clin Infect Dis
Pays: United States
ID NLM: 9203213

Informations de publication

Date de publication:
18 05 2021
Historique:
received: 05 02 2020
accepted: 30 03 2020
pubmed: 3 4 2020
medline: 3 7 2021
entrez: 3 4 2020
Statut: ppublish

Résumé

Mold-active primary antifungal prophylaxis (PAP) is routinely recommended in neutropenic patients with newly diagnosed acute myeloid leukemia (AML) or high-risk myelodysplastic syndrome (MDS) undergoing remission-induction chemotherapy (RIC). Isavuconazole (ISAV) is an extended spectrum mold-active triazole and has superior tolerability and fewer significant drug-drug interactions compared with other triazoles. In our investigator-initiated, phase 2 trial, treatment-naive adult patients with AML or MDS starting RIC received ISAV per the dosing recommendations in the US label until neutrophil recovery (absolute neutrophil count [ANC] ≥ 0.5 × 109/L) and attainment of complete remission, occurrence of invasive fungal infection (IFI), or for a maximum of 12 weeks. The primary endpoint was the incidence of proven/probable IFI during ISAV PAP and up to 30 days after the last dose. Sixty-five of 75 enrolled patients received ISAV PAP (median age, 67 years, median ANC at enrollment, 0.72 × 109/L). Thirty-two patients (49%) received oral targeted leukemia treatments (venetoclax, FTL3 inhibitors). Including the 30-day follow-up period, probable/proven and possible IFIs were encountered in 4 (6%) and 8 patients (12%), respectively. ISAV trough serum concentrations were consistently > 1 µg/mL, showed low intraindividual variation, and were not significantly influenced by chemotherapy regimen. Tolerability of ISAV was excellent, with only 3 cases (5%) of mild to moderate elevations of liver function tests and no QTc prolongations. ISAV is a safe and effective alternative for PAP in patients with newly diagnosed AML/MDS undergoing RIC in the era of recently approved or emerging small-molecule antileukemia therapies. NCT03019939.

Sections du résumé

BACKGROUND
Mold-active primary antifungal prophylaxis (PAP) is routinely recommended in neutropenic patients with newly diagnosed acute myeloid leukemia (AML) or high-risk myelodysplastic syndrome (MDS) undergoing remission-induction chemotherapy (RIC). Isavuconazole (ISAV) is an extended spectrum mold-active triazole and has superior tolerability and fewer significant drug-drug interactions compared with other triazoles.
METHODS
In our investigator-initiated, phase 2 trial, treatment-naive adult patients with AML or MDS starting RIC received ISAV per the dosing recommendations in the US label until neutrophil recovery (absolute neutrophil count [ANC] ≥ 0.5 × 109/L) and attainment of complete remission, occurrence of invasive fungal infection (IFI), or for a maximum of 12 weeks. The primary endpoint was the incidence of proven/probable IFI during ISAV PAP and up to 30 days after the last dose.
RESULTS
Sixty-five of 75 enrolled patients received ISAV PAP (median age, 67 years, median ANC at enrollment, 0.72 × 109/L). Thirty-two patients (49%) received oral targeted leukemia treatments (venetoclax, FTL3 inhibitors). Including the 30-day follow-up period, probable/proven and possible IFIs were encountered in 4 (6%) and 8 patients (12%), respectively. ISAV trough serum concentrations were consistently > 1 µg/mL, showed low intraindividual variation, and were not significantly influenced by chemotherapy regimen. Tolerability of ISAV was excellent, with only 3 cases (5%) of mild to moderate elevations of liver function tests and no QTc prolongations.
CONCLUSIONS
ISAV is a safe and effective alternative for PAP in patients with newly diagnosed AML/MDS undergoing RIC in the era of recently approved or emerging small-molecule antileukemia therapies.
CLINICAL TRIALS REGISTRATION
NCT03019939.

Identifiants

pubmed: 32236406
pii: 5814277
doi: 10.1093/cid/ciaa358
pmc: PMC8130026
doi:

Substances chimiques

Antifungal Agents 0
Nitriles 0
Pyridines 0
Triazoles 0
isavuconazole 60UTO373KE

Banques de données

ClinicalTrials.gov
['NCT03019939']

Types de publication

Clinical Trial, Phase II Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1755-1763

Commentaires et corrections

Type : CommentIn

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press for the Infectious Diseases Society of America. All rights reserved. For permissions, e-mail: journals.permissions@oup.com.

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Auteurs

Prithviraj Bose (P)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

David McCue (D)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Sebastian Wurster (S)

Department of Infectious Diseases, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Nathan P Wiederhold (NP)

Fungus Testing Laboratory, Department of Pathology and Laboratory Medicine, University of Texas Health Science Center at San Antonio, San Antonio, Texas, USA.

Marina Konopleva (M)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Tapan M Kadia (TM)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Gautam Borthakur (G)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Farhad Ravandi (F)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Lucia Masarova (L)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Koichi Takahashi (K)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Zeev Estrov (Z)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Musa Yilmaz (M)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Naval Daver (N)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Naveen Pemmaraju (N)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Kiran Naqvi (K)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Caitlin R Rausch (CR)

Division of Pharmacy, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Kayleigh R Marx (KR)

Division of Pharmacy, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Wei Qiao (W)

Department of Biostatistics, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Xuelin Huang (X)

Department of Biostatistics, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Carol A Bivins (CA)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Sherry A Pierce (SA)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Hagop M Kantarjian (HM)

Department of Leukemia, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Dimitrios P Kontoyiannis (DP)

Department of Infectious Diseases, University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

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Classifications MeSH