Roles and Functions of ROS and RNS in Cellular Physiology and Pathology.
antioxidants
cell signaling
free radicals
growth
oxidative homeostasis
oxidative metabolism of the cells
pathophysiology of oxidative stress
redox balance
toxicity
Journal
Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052
Informations de publication
Date de publication:
21 03 2020
21 03 2020
Historique:
received:
13
03
2020
accepted:
17
03
2020
entrez:
5
4
2020
pubmed:
5
4
2020
medline:
11
2
2021
Statut:
epublish
Résumé
Our common knowledge on oxidative stress has evolved substantially over the years, being focused mostly on the fundamental chemical reactions and the most relevant chemical species involved in human pathophysiology of oxidative stress-associated diseases. Thus, reactive oxygen species and reactive nitrogen species (ROS and RNS) were identified as key players in initiating, mediating, and regulating the cellular and biochemical complexity of oxidative stress either as physiological (acting pro-hormetic) or as pathogenic (causing destructive vicious circles) processes. The papers published in this particular Special Issue of
Identifiants
pubmed: 32245147
pii: cells9030767
doi: 10.3390/cells9030767
pmc: PMC7140712
pii:
doi:
Substances chimiques
Reactive Nitrogen Species
0
Reactive Oxygen Species
0
Toll-Like Receptors
0
Types de publication
Editorial
Introductory Journal Article
Langues
eng
Sous-ensembles de citation
IM
Références
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