RXRs control serous macrophage neonatal expansion and identity and contribute to ovarian cancer progression.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
03 04 2020
Historique:
received: 26 09 2019
accepted: 28 02 2020
entrez: 5 4 2020
pubmed: 5 4 2020
medline: 24 7 2020
Statut: epublish

Résumé

Tissue-resident macrophages (TRMs) populate all tissues and play key roles in homeostasis, immunity and repair. TRMs express a molecular program that is mostly shaped by tissue cues. However, TRM identity and the mechanisms that maintain TRMs in tissues remain poorly understood. We recently found that serous-cavity TRMs (LPMs) are highly enriched in RXR transcripts and RXR-response elements. Here, we show that RXRs control mouse serous-macrophage identity by regulating chromatin accessibility and the transcriptional regulation of canonical macrophage genes. RXR deficiency impairs neonatal expansion of the LPM pool and reduces the survival of adult LPMs through excess lipid accumulation. We also find that peritoneal LPMs infiltrate early ovarian tumours and that RXR deletion diminishes LPM accumulation in tumours and strongly reduces ovarian tumour progression in mice. Our study reveals that RXR signalling controls the maintenance of the serous macrophage pool and that targeting peritoneal LPMs may improve ovarian cancer outcomes.

Identifiants

pubmed: 32246014
doi: 10.1038/s41467-020-15371-0
pii: 10.1038/s41467-020-15371-0
pmc: PMC7125161
doi:

Substances chimiques

Retinoid X Receptors 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1655

Subventions

Organisme : NCI NIH HHS
ID : R01 CA190400
Pays : United States

Références

Bonnardel, J. & Guilliams, M. Developmental control of macrophage function. Curr. Opin. Immunol. 50, 64–74 (2018).
doi: 10.1016/j.coi.2017.12.001
Lavin, Y., Mortha, A., Rahman, A. & Merad, M. Regulation of macrophage development and function in peripheral tissues. Nat. Rev. Immunol. 15, 731–744 (2015).
doi: 10.1038/nri3920 pubmed: 4706379 pmcid: 4706379
Davies, L. C. et al. Distinct bone marrow-derived and tissue-resident macrophage lineages proliferate at key stages during inflammation. Nat. Commun. 4, 1886 (2013).
doi: 10.1038/ncomms2877
Mass E., et al. Specification of tissue-resident macrophages during organogenesis. Science 353, 1–10 (2016).
doi: 10.1126/science.aaf4238
AG, N. et al. The nuclear receptor LXRalpha controls the functional specialization of splenic macrophages. Nat. Immunol. 14, 831–839 (2013).
doi: 10.1038/ni.2622
Gautier, E. L. et al. Systemic analysis of PPARgamma in mouse macrophage populations reveals marked diversity in expression with critical roles in resolution of inflammation and airway immunity. J. Immunol. 189, 2614–2624 (2012).
doi: 10.4049/jimmunol.1200495 pubmed: 3537497 pmcid: 3537497
Kohyama, M. et al. Role for Spi-C in the development of red pulp macrophages and splenic iron homeostasis. Nature 457, 318–321 (2009).
doi: 10.1038/nature07472
Bain, C. C. & Jenkins, S. J. The biology of serous cavity macrophages. Cell Immunol. 330, 126–135 (2018).
doi: 10.1016/j.cellimm.2018.01.003
Ghosn, E. E. et al. Two physically, functionally, and developmentally distinct peritoneal macrophage subsets. Proc. Natl Acad. Sci. USA 107, 2568–2573 (2010).
doi: 10.1073/pnas.0915000107
Roberts, A. W. et al. Tissue-resident macrophages are locally programmed for silent clearance of apoptotic cells. Immunity 47, 913–27 e916 (2017).
doi: 10.1016/j.immuni.2017.10.006 pubmed: 5728676 pmcid: 5728676
Okabe, Y. & Medzhitov, R. Tissue-specific signals control reversible program of localization and functional polarization of macrophages. Cell 157, 832–844 (2014).
doi: 10.1016/j.cell.2014.04.016 pubmed: 4137874 pmcid: 4137874
Takenaka, E., Van, Vo. A., Yamashita-Kanemaru, Y., Shibuya, A. & Shibuya, K. Selective DNAM-1 expression on small peritoneal macrophages contributes to CD4(+) T cell costimulation. Sci. Rep. 8, 15180 (2018).
doi: 10.1038/s41598-018-33437-4 pubmed: 6185969 pmcid: 6185969
Hashimoto, D. et al. Tissue-resident macrophages self-maintain locally throughout adult life with minimal contribution from circulating monocytes. Immunity 38, 792–804 (2013).
doi: 10.1016/j.immuni.2013.04.004
Yona, S. et al. Fate mapping reveals origins and dynamics of monocytes and tissue macrophages under homeostasis. Immunity 38, 79–91 (2013).
doi: 10.1016/j.immuni.2012.12.001
Sheng, J., Ruedl, C. & Karjalainen, K. Most tissue-resident macrophages except microglia are derived from fetal hematopoietic stem cells. Immunity 43, 382–393 (2015).
doi: 10.1016/j.immuni.2015.07.016
Bain, C. C. et al. Long-lived self-renewing bone marrow-derived macrophages displace embryo-derived cells to inhabit adult serous cavities. Nat. Commun. 7, ncomms11852 (2016).
doi: 10.1038/ncomms11852 pubmed: 4910019 pmcid: 4910019
Liu, Z. et al. Fate mapping via Ms4a3-expression history traces monocyte-derived cells. Cell 178, 1509–25 e1519 (2019).
doi: 10.1016/j.cell.2019.08.009
Rosas, M. et al. The transcription factor Gata6 links tissue macrophage phenotype and proliferative renewal. Science 344, 645–648 (2014).
doi: 10.1126/science.1251414 pubmed: 4185421 pmcid: 4185421
Gautier, E. L. et al. Gata6 regulates aspartoacylase expression in resident peritoneal macrophages and controls their survival. J. Exp. Med. 211, 1525–1531 (2014).
doi: 10.1084/jem.20140570 pubmed: 4113942 pmcid: 4113942
Kim, K. W. et al. MHC II+ resident peritoneal and pleural macrophages rely on IRF4 for development from circulating monocytes. J. Exp. Med. 213, 1951–1959 (2016).
doi: 10.1084/jem.20160486 pubmed: 5030807 pmcid: 5030807
Dawson, M. I. & Xia, Z. The retinoid X receptors and their ligands. Biochim Biophysic Acta 1821, 21–56 (2012).
doi: 10.1016/j.bbalip.2011.09.014
Rőszer, T., Menendez-Gutierrez, M. P., Cedenilla, M., Ricote, M. & Retinoid, X receptors in macrophage biology. Trends Endocrinol. Metab. 460–468 (2013).
Lefebvre, P., Benomar, Y. & Staels, B. Retinoid X receptors: common heterodimerization partners with distinct functions. Trends Endocrinol. Metab. 21, 676–683 (2010).
doi: 10.1016/j.tem.2010.06.009
Nunez, V. et al. Retinoid X receptor alpha controls innate inflammatory responses through the up-regulation of chemokine expression. Proc. Natl Acad. Sci. USA 107, 10626–10631 (2010).
doi: 10.1073/pnas.0913545107
Menendez-Gutierrez, M. P. et al. Retinoid X receptors orchestrate osteoclast differentiation and postnatal bone remodeling. J. Clin. Invest. 125, 809–823 (2015).
doi: 10.1172/JCI77186 pubmed: 4319420 pmcid: 4319420
Rőszer, T. et al. Autoimmune kidney disease and impaired engulfment of apoptotic cells in mice with macrophage peroxisome proliferator-activated receptor gamma or retinoid X receptor alpha deficiency. J. Immunol. 186, 621–631 (2011).
doi: 10.4049/jimmunol.1002230
Lavin, Y. et al. Tissue-resident macrophage enhancer landscapes are shaped by the local microenvironment. Cell 159, 1312–1326 (2014).
doi: 10.1016/j.cell.2014.11.018 pubmed: 4437213 pmcid: 4437213
Ricote, M. et al. Normal hematopoiesis after conditional targeting of RXRalpha in murine hematopoietic stem/progenitor cells. J. Leukoc. Biol. 80, 850–861 (2006).
doi: 10.1189/jlb.0206097
Gosselin, D. et al. Environment drives selection and function of enhancers controlling tissue-specific macrophage identities. Cell 159, 1327–1340 (2014).
doi: 10.1016/j.cell.2014.11.023 pubmed: 4364385 pmcid: 4364385
Buechler, M. B. et al. A stromal niche defined by expression of the transcription factor WT1 mediates programming and homeostasis of cavity-resident macrophages. Immunity 51, 119–30 e115 (2019).
doi: 10.1016/j.immuni.2019.05.010
Cassado Ados, A. et al. Cellular renewal and improvement of local cell effector activity in peritoneal cavity in response to infectious stimuli. PLoS One 6, e22141 (2011).
doi: 10.1371/journal.pone.0022141
Toyonaga, K. et al. C-Type lectin receptor DCAR recognizes mycobacterial phosphatidyl-inositol mannosides to promote a Th1 response during infection. Immunity 45, 1245–1257 (2016).
doi: 10.1016/j.immuni.2016.10.012
Cain, D. W. et al. Identification of a tissue-specific, C/EBPbeta-dependent pathway of differentiation for murine peritoneal macrophages. J. Immunol. 191, 4665–4675 (2013).
doi: 10.4049/jimmunol.1300581
Evans, R. M. & Mangelsdorf, D. J. Nuclear receptors, RXR, and the big bang. Cell 157, 255–266 (2014).
doi: 10.1016/j.cell.2014.03.012 pubmed: 4029515 pmcid: 4029515
Liu F., Wu D., Wang X. Roles of CTCF in conformation and functions of chromosome. Semin. Cell Dev. Biol. 168–173 (2018).
Kim, S., Yu, N. K. & Kaang, B. K. CTCF as a multifunctional protein in genome regulation and gene expression. Exp. Mol. Med. 47, e166 (2015).
doi: 10.1038/emm.2015.33 pubmed: 4491725 pmcid: 4491725
Hoeffel, G. et al. C-Myb(+) erythro-myeloid progenitor-derived fetal monocytes give rise to adult tissue-resident macrophages. Immunity 42, 665–678 (2015).
doi: 10.1016/j.immuni.2015.03.011 pubmed: 4545768 pmcid: 4545768
de Boer, J. et al. Transgenic mice with hematopoietic and lymphoid specific expression of Cre. Eur. J. Immunol. 33, 314–325 (2003).
doi: 10.1002/immu.200310005 pubmed: 12548562 pmcid: 12548562
Davies, L. C. et al. A quantifiable proliferative burst of tissue macrophages restores homeostatic macrophage populations after acute inflammation. Eur. J. Immunol. 41, 2155–2164 (2011).
doi: 10.1002/eji.201141817 pubmed: 21710478 pmcid: 21710478
Lu, N., Shen, Q., Mahoney, T. R., Liu, X. & Zhou, Z. Three sorting nexins drive the degradation of apoptotic cells in response to PtdIns(3)P signaling. Mol. Biol. Cell 22, 354–374 (2011).
doi: 10.1091/mbc.e10-09-0756 pubmed: 3031466 pmcid: 3031466
Longatti, A. et al. TBC1D14 regulates autophagosome formation via Rab11- and ULK1-positive recycling endosomes. J. Cell Biol. 197, 659–675 (2012).
doi: 10.1083/jcb.201111079 pubmed: 3365497 pmcid: 3365497
Xu, X. et al. Obesity activates a program of lysosomal-dependent lipid metabolism in adipose tissue macrophages independently of classic activation. Cell Metab. 18, 816–830 (2013).
doi: 10.1016/j.cmet.2013.11.001 pubmed: 3939841 pmcid: 3939841
Saraswathi, V. & Hasty, A. H. Inhibition of long-chain acyl coenzyme A synthetases during fatty acid loading induces lipotoxicity in macrophages. Arterioscler. Thromb. Vasc. Biol. 29, 1937–1943 (2009).
doi: 10.1161/ATVBAHA.109.195362 pubmed: 2766024 pmcid: 2766024
Tabas, I. & Bornfeldt, K. E. Macrophage phenotype and function in different stages of atherosclerosis. Circ. Res. 118, 653–667 (2016).
doi: 10.1161/CIRCRESAHA.115.306256 pubmed: 4762068 pmcid: 4762068
Torre, L. A. et al. Ovarian cancer statistics, 2018. CA Cancer J. Clin. 68, 284–296 (2018).
doi: 10.3322/caac.21456 pubmed: 6621554 pmcid: 6621554
Dizon, D. S. et al. Clinical cancer advances 2016: annual report on progress against cancer from the american society of clinical oncology. J. Clin. Oncol. 34, 987–1011 (2016).
doi: 10.1200/JCO.2015.65.8427 pubmed: 5075244 pmcid: 5075244
Wang, J. & Kubes, P. A reservoir of mature cavity macrophages that can rapidly invade visceral organs to affect tissue repair. Cell 165, 668–678 (2016).
doi: 10.1016/j.cell.2016.03.009
Scarlett, U. K. et al. Ovarian cancer progression is controlled by phenotypic changes in dendritic cells. J. Exp. Med. 209, 495–506 (2012).
doi: 10.1084/jem.20111413 pubmed: 3302234 pmcid: 3302234
Heng, T. S. & Painter, M. W. Immunological Genome Project C. The Immunological Genome Project: networks of gene expression in immune cells. Nat. Immunol. 9, 1091–1094 (2008).
doi: 10.1038/ni1008-1091
Louis, C. et al. Specific contributions of CSF-1 and GM-CSF to the dynamics of the mononuclear phagocyte system. J. Immunol. 195, 134–144 (2015).
doi: 10.4049/jimmunol.1500369
MacDonald, K. P. et al. An antibody against the colony-stimulating factor 1 receptor depletes the resident subset of monocytes and tissue- and tumor-associated macrophages but does not inhibit inflammation. Blood 116, 3955–3963 (2010).
doi: 10.1182/blood-2010-02-266296
Cecchini, M. G. et al. Role of colony stimulating factor-1 in the establishment and regulation of tissue macrophages during postnatal development of the mouse. Development 120, 1357–1372 (1994).
Ryan, G. R. et al. Rescue of the colony-stimulating factor 1 (CSF-1)-nullizygous mouse (Csf1(op)/Csf1(op)) phenotype with a CSF-1 transgene and identification of sites of local CSF-1 synthesis. Blood 98, 74–84 (2001).
doi: 10.1182/blood.V98.1.74
Dai, X. M., Zong, X. H., Sylvestre, V. & Stanley, E. R. Incomplete restoration of colony-stimulating factor 1 (CSF-1) function in CSF-1-deficient Csf1op/Csf1op mice by transgenic expression of cell surface CSF-1. Blood 103, 1114–1123 (2004).
doi: 10.1182/blood-2003-08-2739
Wong, K. et al. Phosphatidylserine receptor Tim-4 is essential for the maintenance of the homeostatic state of resident peritoneal macrophages. Proc. Natl Acad. Sci. USA 107, 8712–8717 (2010).
doi: 10.1073/pnas.0910929107
Settembre, C. & Ballabio, A. Lysosome: regulator of lipid degradation pathways. Trends Cell Biol. 24, 743–750 (2014).
doi: 10.1016/j.tcb.2014.06.006 pubmed: 4247383 pmcid: 4247383
Zhou, Z. & Yu, X. Phagosome maturation during the removal of apoptotic cells: receptors lead the way. Trends Cell Biol. 18, 474–485 (2008).
doi: 10.1016/j.tcb.2008.08.002 pubmed: 3125982 pmcid: 3125982
Markman, M. Poly (ADP-ribose) polymerase inhibitors in the management of ovarian cancer. Women’s Health 14, 1–6 (2018).
Weiss, J. M. et al. Itaconic acid mediates crosstalk between macrophage metabolism and peritoneal tumors. J. Clin. Investig. 128, 3794–3805 (2018).
doi: 10.1172/JCI99169
Finkernagel, F. et al. The transcriptional signature of human ovarian carcinoma macrophages is associated with extracellular matrix reorganization. Oncotarget 7, 75339–75352 (2016).
doi: 10.18632/oncotarget.12180 pubmed: 5342745 pmcid: 5342745
Li, M. et al. Retinoid X receptor ablation in adult mouse keratinocytes generates an atopic dermatitis triggered by thymic stromal lymphopoietin. Proc. Natl Acad. Sci. USA 102, 14795–14800 (2005).
doi: 10.1073/pnas.0507385102
Srinivas, S. et al. Cre reporter strains produced by targeted insertion of EYFP and ECFP into the ROSA26 locus. BMC Dev. Biol. 1, 4 (2001).
doi: 10.1186/1471-213X-1-4 pubmed: 31338 pmcid: 31338
Jimenez-Carretero, D., Ligos, J. M., Martinez-Lopez, M., Sancho, D. & Montoya, M. C. Flow cytometry data preparation guidelines for improved automated phenotypic analysis. J. Immunol. 200, 3319–3331 (2018).
doi: 10.4049/jimmunol.1800446
Buenrostro, J. D., Giresi, P. G., Zaba, L. C., Chang, H. Y. & Greenleaf, W. J. Transposition of native chromatin for fast and sensitive epigenomic profiling of open chromatin, DNA-binding proteins and nucleosome position. Nat. Methods 10, 1213–1218 (2013).
doi: 10.1038/nmeth.2688 pubmed: 3959825 pmcid: 3959825
Langmead, B. & Salzberg, S. L. Fast gapped-read alignment with Bowtie 2. Nat. Methods 9, 357–359 (2012).
doi: 10.1038/nmeth.1923 pubmed: 3322381 pmcid: 3322381
Zhang, Y. et al. Model-based analysis of ChIP-Seq (MACS). Genome Biol. 9, R137 (2008).
doi: 10.1186/gb-2008-9-9-r137 pubmed: 2592715 pmcid: 2592715
Robinson, M. D., McCarthy, D. J. & Smyth, G. K. edgeR: A bioconductor package for differential expression analysis of digital gene expression data. Bioinformatics 26, 139–140 (2010).
doi: 10.1093/bioinformatics/btp616
Heinz, S. et al. Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities. Mol. Cell 38, 576–589 (2010).
doi: 10.1016/j.molcel.2010.05.004 pubmed: 2898526 pmcid: 2898526

Auteurs

María Casanova-Acebes (M)

Department of Oncological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

María Piedad Menéndez-Gutiérrez (MP)

Area of Myocardial Pathophysiology, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.

Jesús Porcuna (J)

Area of Myocardial Pathophysiology, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.

Damiana Álvarez-Errico (D)

Area of Myocardial Pathophysiology, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.

Yonit Lavin (Y)

Department of Oncological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Ana García (A)

Area of Myocardial Pathophysiology, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.

Soma Kobayashi (S)

Department of Oncological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Jessica Le Berichel (J)

Department of Oncological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Vanessa Núñez (V)

Area of Myocardial Pathophysiology, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.

Felipe Were (F)

Bioinformatics Unit, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.

Daniel Jiménez-Carretero (D)

Cellomics Unit, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.

Fátima Sánchez-Cabo (F)

Bioinformatics Unit, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain.

Miriam Merad (M)

Department of Oncological Sciences, Icahn School of Medicine at Mount Sinai, New York, NY, USA. miriam.merad@mssm.edu.
Precision Immunology Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA. miriam.merad@mssm.edu.
Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA. miriam.merad@mssm.edu.

Mercedes Ricote (M)

Area of Myocardial Pathophysiology, Centro Nacional de Investigaciones Cardiovasculares (CNIC), Madrid, Spain. mricote@cnic.es.

Articles similaires

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male
Humans Meals Time Factors Female Adult

Classifications MeSH