PACT/PRKRA and p53 regulate transcriptional activity of DMRT1.


Journal

Genetics and molecular biology
ISSN: 1415-4757
Titre abrégé: Genet Mol Biol
Pays: Brazil
ID NLM: 100883590

Informations de publication

Date de publication:
2020
Historique:
received: 23 01 2019
accepted: 08 05 2019
entrez: 7 4 2020
pubmed: 7 4 2020
medline: 7 4 2020
Statut: epublish

Résumé

The transcription factor DMRT1 (doublesex and mab-3 related transcription factor) has two distinct functions, somatic-cell masculinization and germ-cell development in some vertebrate species, including mouse and the African clawed frog Xenopus laevis. However, its transcriptional regulation remains unclear. We tried to identify DMRT1-interacting proteins from X. laevis testes by immunoprecipitation with an anti-DMRT1 antibody and MS/MS analysis, and selected three proteins, including PACT/PRKRA (Interferon-inducible double-stranded RNA dependent protein kinase activator A) derived from testes. Next, we examined the effects of PACT/PRKRA and/or p53 on the transcriptional activity of DMRT1. In transfected 293T cells, PACT/PRKRA and p53 significantly enhanced and repressed DMRT1-driven luciferase activity, respectively. We also observed that the enhanced activity by PACT/PRKRA was strongly attenuated by p53. Moreover, in situ hybridization analysis of Pact/Prkra mRNA in tadpole gonads indicated high expression in female and male germline stem cells. Taken together, these findings suggest that PACT/PRKRA and p53 might positively and negatively regulate the activity of DMRT1, respectively, for germline stem cell fate.

Identifiants

pubmed: 32251494
pii: S1415-47572020000400702
doi: 10.1590/1678-4685-GMB-2019-0017
pmc: PMC7198010
pii:
doi:

Types de publication

Journal Article

Langues

eng

Pagination

e20190017

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Auteurs

Kazuko Fujitani (K)

Kitasato University, Gene Analysis Center, School of Medicine, Sagamihara, Japan.

Asako Otomo (A)

Tokai University School of Medicine, Department of Molecular Life Sciences, Isehara, Japan.

Yuto Nagayama (Y)

Osaka City University, Department of Bioengineering, Graduate School of Engineering, Osaka, Japan.

Taro Tachibana (T)

Osaka City University, Department of Bioengineering, Graduate School of Engineering, Osaka, Japan.
Cell Engineering Corporation, Osaka, Japan.

Rika Kato (R)

Kitasato University, Department of Physics, School of Science, Sagamihara, Japan.

Yusuke Kawashima (Y)

Kitasato University, Department of Physics, School of Science, Sagamihara, Japan.

Yoshio Kodera (Y)

Kitasato University, Department of Physics, School of Science, Sagamihara, Japan.

Tomoko Kato (T)

National Research Institute for Child Health and Development, Department of Systems BioMedicine, Tokyo, Japan.

Shuji Takada (S)

National Research Institute for Child Health and Development, Department of Systems BioMedicine, Tokyo, Japan.

Kei Tamura (K)

Kitasato University, Department of Bioscience, School of Science, Sagamihara, Japan.

Nobuhiko Takamatsu (N)

Kitasato University, Department of Bioscience, School of Science, Sagamihara, Japan.

Michihiko Ito (M)

Kitasato University, Department of Bioscience, School of Science, Sagamihara, Japan.

Classifications MeSH