Ultrastructural characterization of mitochondrial damage in experimental autoimmune neuritis.


Journal

Journal of neuroimmunology
ISSN: 1872-8421
Titre abrégé: J Neuroimmunol
Pays: Netherlands
ID NLM: 8109498

Informations de publication

Date de publication:
15 06 2020
Historique:
received: 05 03 2020
revised: 16 03 2020
accepted: 18 03 2020
pubmed: 7 4 2020
medline: 11 11 2020
entrez: 7 4 2020
Statut: ppublish

Résumé

Data are sparse about mitochondrial damage in GBS and in its most frequently employed animal model, experimental autoimmune neuritis (EAN). We here characterized changes in mitochondrial content and morphology at different time points during EAN by use of ultrastructural imaging and immunofluorescent labelling. Histological examination revealed that demyelinated axons and their adjacent Schwann cells showed reduced mitochondrial content and remaining mitochondria appeared swollen with greater diameter in Schwann cells and unmyelinated axons. Our findings indicate that in EAN, particularly mitochondria in Schwann cells are damaged. Further studies are warranted to address whether these changes are amenable to novel, mitoprotective treatments.

Identifiants

pubmed: 32251941
pii: S0165-5728(20)30139-9
doi: 10.1016/j.jneuroim.2020.577218
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

577218

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors report that they do not have any financial or other conflicts of interest related to this study.

Auteurs

Ines Muke (I)

Department of Neurology, Faculty of Medicine and University Hospital Cologne, Germany; Center for Molecular Medicine Cologne (CMMC), University of Cologne, Germany.

Alina Sprenger (A)

Department of Neurology, Faculty of Medicine and University Hospital Cologne, Germany; Center for Molecular Medicine Cologne (CMMC), University of Cologne, Germany.

Ilja Bobylev (I)

Department of Neurology, Faculty of Medicine and University Hospital Cologne, Germany; Center for Molecular Medicine Cologne (CMMC), University of Cologne, Germany.

Valerie Wiemer (V)

Department of Neurology, Faculty of Medicine and University Hospital Cologne, Germany.

Mohammed Barham (M)

Department of Anatomy I, Faculty of Medicine, University of Cologne, Germany.

Wolfram Friedrich Neiss (WF)

Department of Anatomy I, Faculty of Medicine, University of Cologne, Germany.

Helmar Christoph Lehmann (HC)

Department of Neurology, Faculty of Medicine and University Hospital Cologne, Germany; Center for Molecular Medicine Cologne (CMMC), University of Cologne, Germany. Electronic address: helmar.lehmann@uk-koeln.de.

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Classifications MeSH