The role of Toll-like receptor signaling pathways in cerebrovascular disorders: the impact of spreading depolarization.
Brain
Chemokines
Inflammatory mediators
Spreading depolarization
Stroke
Subarachnoid hemorrhage
Toll-like receptors
Journal
Journal of neuroinflammation
ISSN: 1742-2094
Titre abrégé: J Neuroinflammation
Pays: England
ID NLM: 101222974
Informations de publication
Date de publication:
07 Apr 2020
07 Apr 2020
Historique:
received:
10
01
2020
accepted:
24
03
2020
entrez:
9
4
2020
pubmed:
9
4
2020
medline:
11
2
2021
Statut:
epublish
Résumé
Cerebral vascular diseases (CVDs) are a group of disorders that affect the blood supply to the brain and lead to the reduction of oxygen and glucose supply to the neurons and the supporting cells. Spreading depolarization (SD), a propagating wave of neuroglial depolarization, occurs in different CVDs. A growing amount of evidence suggests that the inflammatory responses following hypoxic-ischemic insults and after SD plays a double-edged role in brain tissue injury and clinical outcome; a beneficial effect in the acute phase and a destructive role in the late phase. Toll-like receptors (TLRs) play a crucial role in the activation of inflammatory cascades and subsequent neuroprotective or harmful effects after CVDs and SD. Here, we review current data regarding the pathophysiological role of TLR signaling pathways in different CVDs and discuss the role of SD in the potentiation of the inflammatory cascade in CVDs through the modulation of TLRs.
Identifiants
pubmed: 32264928
doi: 10.1186/s12974-020-01785-6
pii: 10.1186/s12974-020-01785-6
pmc: PMC7140571
doi:
Substances chimiques
Toll-Like Receptors
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
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