Premenopausal Plasma Osteoprotegerin and Breast Cancer Risk: A Case-Control Analysis Nested within the Nurses' Health Study II.


Journal

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology
ISSN: 1538-7755
Titre abrégé: Cancer Epidemiol Biomarkers Prev
Pays: United States
ID NLM: 9200608

Informations de publication

Date de publication:
06 2020
Historique:
received: 15 10 2019
revised: 13 12 2019
accepted: 06 04 2020
pubmed: 12 4 2020
medline: 16 9 2021
entrez: 12 4 2020
Statut: ppublish

Résumé

Emerging evidence supports a role of the receptor activator of NF-κB (RANK) pathway in normal mammary gland development and breast carcinogenesis. Osteoprotegerin (OPG) is the endogenous decoy receptor for RANK-ligand (RANKL), which inhibits RANK-signaling. Whether OPG may be a biomarker of breast cancer risk remains unclear. We evaluated the association between plasma OPG and breast cancer risk in a case ( Overall, there was no substantial evidence for an association between plasma OPG levels and breast cancer risk, although the point estimate for the highest (vs. lowest) quartile was below 1 (OR = 0.78; 95% CI, 0.46-1.33; Findings from this prospective study do not provide substantial evidence for an association between circulating OPG and breast cancer risk among premenopausal women; however, we were underpowered in stratified analyses. Results do not provide strong evidence for OPG as a potential biomarker of breast cancer risk among premenopausal women.

Sections du résumé

BACKGROUND
Emerging evidence supports a role of the receptor activator of NF-κB (RANK) pathway in normal mammary gland development and breast carcinogenesis. Osteoprotegerin (OPG) is the endogenous decoy receptor for RANK-ligand (RANKL), which inhibits RANK-signaling. Whether OPG may be a biomarker of breast cancer risk remains unclear.
METHODS
We evaluated the association between plasma OPG and breast cancer risk in a case (
RESULTS
Overall, there was no substantial evidence for an association between plasma OPG levels and breast cancer risk, although the point estimate for the highest (vs. lowest) quartile was below 1 (OR = 0.78; 95% CI, 0.46-1.33;
CONCLUSIONS
Findings from this prospective study do not provide substantial evidence for an association between circulating OPG and breast cancer risk among premenopausal women; however, we were underpowered in stratified analyses.
IMPACT
Results do not provide strong evidence for OPG as a potential biomarker of breast cancer risk among premenopausal women.

Identifiants

pubmed: 32277005
pii: 1055-9965.EPI-19-1154
doi: 10.1158/1055-9965.EPI-19-1154
pmc: PMC7269832
mid: NIHMS1584106
doi:

Substances chimiques

Osteoprotegerin 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1264-1270

Subventions

Organisme : NCI NIH HHS
ID : R01 CA050385
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA067262
Pays : United States
Organisme : NCI NIH HHS
ID : UM1 CA176726
Pays : United States
Organisme : NCI NIH HHS
ID : U01 CA176726
Pays : United States
Organisme : NCI NIH HHS
ID : P50 CA168504
Pays : United States

Informations de copyright

©2020 American Association for Cancer Research.

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Auteurs

Joanne Kotsopoulos (J)

Women's College Research Institute, Women's College Hospital, Toronto, Ontario, Canada. joanne.kotsopoulos@wchospital.ca.
Dalla Lana School of Public Health, University of Toronto, Toronto, Ontario, Canada.

Emma E McGee (EE)

Channing Division of Network Medicine, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts.
Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, Massachusetts.

Susana Lozano-Esparza (S)

Channing Division of Network Medicine, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts.

Judy E Garber (JE)

Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts.

Jennifer Ligibel (J)

Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts.

Laura C Collins (LC)

Department of Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts.

Kornelia Polyak (K)

Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts.

Myles Brown (M)

Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts.

Steven Narod (S)

Women's College Research Institute, Women's College Hospital, Toronto, Ontario, Canada.
Dalla Lana School of Public Health, University of Toronto, Toronto, Ontario, Canada.

Rulla M Tamimi (RM)

Channing Division of Network Medicine, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts.
Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, Massachusetts.

A Heather Eliassen (AH)

Channing Division of Network Medicine, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, Massachusetts.
Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, Massachusetts.

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