TCR
Acute Kidney Injury
/ chemically induced
Adoptive Transfer
Animals
Apoptosis
B7-H1 Antigen
/ immunology
Cell Proliferation
Cells, Cultured
Cisplatin
Coculture Techniques
Disease Models, Animal
Epithelial Cells
/ immunology
Kidney Tubules, Proximal
/ immunology
Male
Mice, Inbred C57BL
Phenotype
Receptors, Antigen, T-Cell
/ immunology
T-Lymphocyte Subsets
/ immunology
T cell receptor
acute kidney injury
cisplatin
double negative T cells
kidney epithelial cells
Journal
American journal of physiology. Renal physiology
ISSN: 1522-1466
Titre abrégé: Am J Physiol Renal Physiol
Pays: United States
ID NLM: 100901990
Informations de publication
Date de publication:
01 06 2020
01 06 2020
Historique:
pubmed:
14
4
2020
medline:
8
10
2020
entrez:
14
4
2020
Statut:
ppublish
Résumé
Acute kidney injury (AKI) due to cisplatin is a significant problem that limits its use as an effective chemotherapeutic agent. T cell receptor
Identifiants
pubmed: 32281417
doi: 10.1152/ajprenal.00033.2020
pmc: PMC7311707
doi:
Substances chimiques
B7-H1 Antigen
0
Cd274 protein, mouse
0
Receptors, Antigen, T-Cell
0
Cisplatin
Q20Q21Q62J
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
F1500-F1512Subventions
Organisme : NIDDK NIH HHS
ID : R01 DK104662
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK111209
Pays : United States
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