The AST/ALT (De Ritis) ratio predicts clinical outcome in patients with pancreatic cancer treated with first-line nab-paclitaxel and gemcitabine:

AST/ALT De Ritis ratio biomarker gemcitabine nab-paclitaxel pancreatic cancer treatment response

Journal

Therapeutic advances in medical oncology
ISSN: 1758-8340
Titre abrégé: Ther Adv Med Oncol
Pays: England
ID NLM: 101510808

Informations de publication

Date de publication:
2020
Historique:
received: 04 07 2019
accepted: 19 12 2019
entrez: 22 4 2020
pubmed: 22 4 2020
medline: 22 4 2020
Statut: epublish

Résumé

The pretreatment De Ritis ratio [aspartate transaminase (AST)/alanine transaminase (ALT)] has been shown to be an adverse prognostic marker in various cancer entities. However, its relevance to advanced pancreatic ductal adenocarcinoma (PDAC) has not yet been studied. In the present study we investigated the AST/ALT ratio as a possible predictor of treatment response and disease outcome in patients with advanced PDAC treated with first-line gemcitabine/nab-paclitaxel. A Median and 1-year progression-free survival estimates were 4.8 months and 5.1%, respectively in patients with an AST/ALT ratio above the 75th percentile of its distribution, and 6.0 months and 18.7%, respectively in patients with an AST/ALT ratio less than or equal to this cutoff, respectively (log-rank The pretreatment serum AST/ALT ratio predicts poor disease outcome and response rate in patients with advanced PDAC treated with gemcitabine/nab-paclitaxel and might represent a novel and inexpensive marker for individual risk assessment in the treatment of pancreatic cancer.

Sections du résumé

BACKGROUND BACKGROUND
The pretreatment De Ritis ratio [aspartate transaminase (AST)/alanine transaminase (ALT)] has been shown to be an adverse prognostic marker in various cancer entities. However, its relevance to advanced pancreatic ductal adenocarcinoma (PDAC) has not yet been studied. In the present study we investigated the AST/ALT ratio as a possible predictor of treatment response and disease outcome in patients with advanced PDAC treated with first-line gemcitabine/nab-paclitaxel.
METHODS METHODS
A
RESULTS RESULTS
Median and 1-year progression-free survival estimates were 4.8 months and 5.1%, respectively in patients with an AST/ALT ratio above the 75th percentile of its distribution, and 6.0 months and 18.7%, respectively in patients with an AST/ALT ratio less than or equal to this cutoff, respectively (log-rank
CONCLUSIONS CONCLUSIONS
The pretreatment serum AST/ALT ratio predicts poor disease outcome and response rate in patients with advanced PDAC treated with gemcitabine/nab-paclitaxel and might represent a novel and inexpensive marker for individual risk assessment in the treatment of pancreatic cancer.

Identifiants

pubmed: 32313566
doi: 10.1177/1758835919900872
pii: 10.1177_1758835919900872
pmc: PMC7153180
doi:

Types de publication

Journal Article

Langues

eng

Pagination

1758835919900872

Informations de copyright

© The Author(s), 2020.

Déclaration de conflit d'intérêts

Conflict of interest statement: All authors have completed the ICMJE uniform disclosure form at www.icmje.org/coi_disclosure.pdf and declare that they have no competing interests.

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Auteurs

Jakob Michael Riedl (JM)

Division of Oncology, Department of Internal Medicine, Medical University of Graz, Graz, Austria.

Florian Posch (F)

Division of Oncology, Department of Internal Medicine, Medical University of Graz, Graz, Austria.

Gerald Prager (G)

Department of Medicine I, Division of Oncology, Medical University of Vienna, Vienna, Austria.

Wolfgang Eisterer (W)

Department of Internal Medicine, Klinikum Klagenfurt am Wörthersee, Feschnigstraße Sarcoma Platform Austria, Austria.

Leopold Oehler (L)

Department of Medicine, St. Joseph Hospital, Vienna, Vienna, Austria.

Thamer Sliwa (T)

Third Medical Department, Hanusch Hospital, Vienna, Austria.

Klaus Wilthoner (K)

Landeskrankenhaus Vöcklabruck, Vöcklabruck, Austria.

Andreas Petzer (A)

Department of Internal Medicine I, Hematology with Stem Cell Transplantation, Hemostaseology and Medical Oncology, Ordensklinikum Linz-Elisabethinen, Linz, Austria.

Petra Pichler (P)

Universitätsklinikum St. Pölten, Sankt Pölten, Austria.

Eva Hubmann (E)

Department of Internal Medicine 1, Hospital of the Brothers of St. John of God, Graz, Austria.

Thomas Winder (T)

Division of Oncology, Department of Internal Medicine II, Academic Teaching Hospital Feldkirch, Feldkirch, Austria.

Sonja Burgstaller (S)

Abteilung für Innere Medizin IV, Klinikum Wels-Grieskirchen, Wels, Austria.

Markus Korger (M)

Krankenhaus der barmherzigen Brüder, Eisenstadt, Austria.

Johannes Andel (J)

Landeskrankenhaus Steyr, Steyr, Austria.

Richard Greil (R)

IIIrd Medical Department, Paracelsus Medical University Salzburg, Salzburg Cancer Research Institute, Cancer Cluster Salzburg, Salzburg, Austria.

Hans-Joerg Neumann (HJ)

Krankenhaus der Elisabethinen, Klagenfurt, Austria.

Martin Pecherstorfer (M)

Department of Internal Medicine 2, University Hospital Krems, Karl Landsteiner Private University of Health Sciences, Krems, Austria.

Kathrin Philipp-Abbrederis (K)

Department of Internal Medicine I, Gastroenterology, Hepatology, Metabolism & Endocrinology, Medical University Innsbruck, Innsbruck, Austria.

Angela Djanani (A)

Department of Internal Medicine I, Gastroenterology, Hepatology, Metabolism & Endocrinology, Medical University Innsbruck, Innsbruck, Austria.

Birgit Gruenberger (B)

Department of Surgery, Landesklinikum Wr. Neustadt, Wr. Neustadt, Austria.

Friedrich Laengle (F)

Department of Surgery, Landesklinikum Wr. Neustadt, Wr. Neustadt, Austria.

Ewald Wöll (E)

Department of Internal Medicine, St. Vinzenz Hospital Zams, Sanatoriumstrasse, Zams, Austria.

Armin Gerger (A)

Division of Oncology, Department of Internal Medicine, Medical University of Graz, Auenbruggerplatz 15, Graz, 8036, Austria.

Classifications MeSH