Role of Divalent Cations in HIV-1 Replication and Pathogenicity.
HIV-1 associated neurocognitive disorders
divalent cations
endolysosomes
human immunodeficiency virus type-1
transactivator of transcription
Journal
Viruses
ISSN: 1999-4915
Titre abrégé: Viruses
Pays: Switzerland
ID NLM: 101509722
Informations de publication
Date de publication:
21 04 2020
21 04 2020
Historique:
received:
31
03
2020
revised:
09
04
2020
accepted:
18
04
2020
entrez:
25
4
2020
pubmed:
25
4
2020
medline:
20
2
2021
Statut:
epublish
Résumé
Divalent cations are essential for life and are fundamentally important coordinators of cellular metabolism, cell growth, host-pathogen interactions, and cell death. Specifically, for human immunodeficiency virus type-1 (HIV-1), divalent cations are required for interactions between viral and host factors that govern HIV-1 replication and pathogenicity. Homeostatic regulation of divalent cations' levels and actions appear to change as HIV-1 infection progresses and as changes occur between HIV-1 and the host. In people living with HIV-1, dietary supplementation with divalent cations may increase HIV-1 replication, whereas cation chelation may suppress HIV-1 replication and decrease disease progression. Here, we review literature on the roles of zinc (Zn
Identifiants
pubmed: 32326317
pii: v12040471
doi: 10.3390/v12040471
pmc: PMC7232465
pii:
doi:
Substances chimiques
Cations, Divalent
0
tat Gene Products, Human Immunodeficiency Virus
0
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Review
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : NINDS NIH HHS
ID : R01 NS065957
Pays : United States
Organisme : NIGMS NIH HHS
ID : U54 GM115458
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH119000
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH100972
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH105329
Pays : United States
Organisme : NIDA NIH HHS
ID : R01 DA032444
Pays : United States
Déclaration de conflit d'intérêts
The authors disclose that this manuscript was written without any commercial or financial associations that could be construed as a conflict of interest.
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